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by Kajimoto, Katsuya and Minami, Yuichiro and Otsubo, Shigeru and Sato, Yukihito and Sato, Takatoshi and Sato, Takamasa and Sato, Naoki and Asai, Kuniya and Munakata, Ryo and Aokage, Toshiyuki and Yoshida, Asuka and Yoshida, Daisuke and Yoshida, Kentaro and Yumino, Dai and Mizuno, Kiyo-o and Mizuno, Masayuki and Kawada, Erisa and Ozaki, Yuri and Kogure, Tomohito and Haruki, Shintaro and Nakao, Koichi and Sawamura, Tadashi and Sawamura, Akinori and Nuki, Toshiaki and Ishiki, Ryoji and Yokota, Yuya and Yokota, Shigeki and Fujinaga, Hiroyuki and Yamamoto, Takashi and Harada, Kenji and Saito, Taro and Saito, Akihiro and Kageyama, Norihito and Okumura, Takanobu and Hata, Noritake and Murai, Koji and Nozaki, Ayaka and Kawanaka, Hidekazu and Tanabe, Jun and Ishii, Katsuhisa and Oiwa, Hitoshi and Matsumoto, Tomoaki and Kato, Hiromasa and Kato, Nobuo and Kato, Masahiko and Suzuki, Hiroshi and Shimizu, Nobuyuki and Shimizu, Hayato and Keida, Takehiko and Fujita, Takashi and Fujita, Masaki and Nakamura, Keita and Nakamura, Makiko and Nakamura, Kentaro and Chinen, Toshiya and Meguro, Kentaro and Kikuchi, Tatsuro and Nishikido, Toshiyuki and Nakata, Masaya and Nakata, Marohito and Yamashita, Shizuya and Yamashita, Eiji and Yamashita, Tatsuya and Hirono, Akitoshi and Mitsudo, Kazuaki and Kadota, Kazushige and Makita, Noriko and Watanabe, Nagisa and Kawabata, Masaaki and Fujii, Kenichi and Fujii, Shinya and Okuda, Shinichi and Kobayashi, Hiroshi and Kobayashi, Shigeki and Moriuchi, Ikuo and Osato, Kazuo and Murakami, Tatsuaki and Shimada, Yoshifumi and Shimada, Katsunori and Misawa, Katsushi and Kokado, Hiromasa and Fukuoka, Yoshitomo and Takabatake, Syu and Takata, Yoshifumi and Miyagi, Manabu and Tanaka, Nobuhiro and Yamashina, Akira and Sudo, Shinji and Shimamura, Koichi and Nagashima, Michitaka and Kaneda, Tomoya and Ueda, Yasunori and Ueda, Kosei and Higashikata, Toshinori and Fujimori, Kanichi and Yagi, Masahiro and Takaki, Jyunko and Toyama, Takuji and Hirata, Tetsuo and Kamisihima, Kazuho and ... and Acute Decompensated Heart Failure and investigators of the Acute Decompensated Heart Failure Syndromes (ATTEND) registry
The American Journal of Cardiology, ISSN 0002-9149, 09/2017, Volume 120, Issue 5, pp. 809 - 816
The aim of this study was to evaluate the association of functional mitral regurgitation (FMR), preserved or reduced ejection fraction (EF), and ischemic or... 
VENTRICULAR SYSTOLIC DYSFUNCTION | PROGNOSTIC IMPLICATIONS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | DETERMINANTS | DISEASE | RISK | DILATED CARDIOMYOPATHY | ECHOCARDIOGRAPHY | QUANTITATIVE DOPPLER ASSESSMENT | SEVERITY | Japan - epidemiology | Prospective Studies | Humans | Heart Failure - physiopathology | Male | Mitral Valve Insufficiency - epidemiology | Heart Ventricles - diagnostic imaging | Incidence | Echocardiography, Doppler | Myocardial Ischemia - physiopathology | Female | Registries | Ventricular Function, Left - physiology | Myocardial Ischemia - diagnosis | Heart Failure - mortality | Severity of Illness Index | Heart Failure - complications | Acute Disease | Survival Rate - trends | Stroke Volume - physiology | Mitral Valve Insufficiency - diagnosis | Heart Ventricles - physiopathology | Myocardial Ischemia - complications | Cause of Death - trends | Mitral Valve Insufficiency - etiology | Heart failure | Care and treatment | Mitral valve insufficiency | Patient outcomes | Analysis | Cardiac patients | Hypertension | Heart attacks | Cardiomyopathy | Mortality | Color | Disorders | Cardiovascular disease | Hospitalization | Patients | Discharge | Confidence intervals | Ultrasonic imaging | Ischemia | Regurgitation | Acute coronary syndromes | Health risk assessment | Heart diseases | Ejection
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 4842 - 11
Hepatocellular carcinoma (HCC) is a common malignant tumor with poor prognosis. We previously showed that expression of Delta-like 3 (DLL3), a member of the... 
CELLS | HDACS | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | PATTERNS | HUMAN HEPATOCELLULAR-CARCINOMA | CANCER | DLL3 | Histone deacetylase | Chromatin | Hepatocellular carcinoma | Infections | Genomes | siRNA | Hepatitis | DNA methylation | Notch protein | Acetylation | X protein | Viral infections | Hepatitis B | Apoptosis
Journal Article
Journal of Gastroenterology, ISSN 0944-1174, 5/2015, Volume 50, Issue 5, pp. 541 - 554
Journal Article
Journal of Molecular Histology, ISSN 1567-2379, 6/2013, Volume 44, Issue 3, pp. 311 - 326
Ethanol-induced hepatic steatosis may induce the progression of alcoholic liver disease. The involvement of autophagic clearance of damaged mitochondria... 
Life Sciences | Ethanol | Lipophagy | Rat | Liver | Developmental Biology | Mitophagy | Autophagy | Biomedicine general | Cell Biology | CANCER-CELLS | APOPTOSIS | PARKIN-MEDIATED MITOPHAGY | ALCOHOLIC LIVER-DISEASE | INJURY | CELL BIOLOGY | MONITORING AUTOPHAGY | FATTY LIVER | ASSAYS | STRESS | CATHEPSIN-D | Protein Kinases - metabolism | Rats, Wistar | Fatty Liver - pathology | Microscopy, Electron, Transmission - methods | Microtubule-Associated Proteins - metabolism | Hepatocytes - pathology | Lipids | Male | Autophagy - physiology | Hepatocytes - metabolism | Fatty Liver - chemically induced | Mitochondria - ultrastructure | Lysosomes - metabolism | Ethanol - adverse effects | Ultrasonography | Lysosomes - pathology | Phagosomes - ultrastructure | Liver Diseases, Alcoholic - pathology | Fatty Liver - metabolism | Liver Diseases, Alcoholic - diagnostic imaging | Liver - ultrastructure | Cytochromes c - metabolism | Liver - metabolism | Phagosomes - metabolism | Rats | Mitochondria - metabolism | Mitochondria - pathology | Mitochondrial Degradation - physiology | Phagosomes - pathology | Liver Diseases, Alcoholic - metabolism | Immunohistochemistry - methods | Lysosomes - ultrastructure | Fatty Liver - diagnostic imaging | Animals | Cathepsins - metabolism | Hepatocytes - ultrastructure | Lysosomal-Associated Membrane Protein 2 - metabolism | Immunohistochemistry | Alcohol, Denatured | Alcohol | Mitochondrial DNA | Electron microscopy | Liver cirrhosis | Cells | Index Medicus
Journal Article
Journal of Reproductive Immunology, ISSN 0165-0378, 11/2015, Volume 112, p. 137
Journal Article
Journal of Reproductive Immunology, ISSN 0165-0378, 2015, Volume 112, pp. 137 - 138
Journal Article
Journal Article
Medical Molecular Morphology, ISSN 1860-1480, 9/2017, Volume 50, Issue 3, pp. 130 - 144
The establishment of consistent and reliable methods for the analysis of atherosclerosis molecular pathways and for testing the efficiency of new therapeutics... 
Pathology | Medicine & Public Health | Real-time PCR | Atherosclerosis | Molecular Medicine | ApoE -knockout mouse | Anatomy | Antibody array | ApoE-knockout mouse | FAMILIAL HYPERCHOLESTEROLEMIA | HEPATOCYTE GROWTH-FACTOR | LOW-DENSITY-LIPOPROTEIN | LECTIN-LIKE | SCAVENGER RECEPTOR | E-NULL MICE | CARDIOVASCULAR-DISEASE | TISSUE INHIBITOR | EXTRACELLULAR-MATRIX | E-DEFICIENT MICE | Immunohistochemistry | Atherosclerosis - pathology | Apolipoproteins E - deficiency | Carotid Intima-Media Thickness | Atherosclerosis - genetics | Mice, Inbred C57BL | Scavenger Receptors, Class E | Male | Apolipoproteins E - metabolism | Mice, Knockout | Atherosclerosis - blood | Animals | Atherosclerosis - diagnostic imaging | Lipids - blood | Matrix Metalloproteinases - metabolism | Real-Time Polymerase Chain Reaction | Viral antibodies | Analysis | Therapeutics | Antibodies | Molecular biology | Apolipoproteins | Cells | Formaldehyde | Homeopathy | Materia medica and therapeutics | Tenascin | Drugs | Paraffin | Tissues | Tissue inhibitor of metalloproteinase 1 | Macrophages | High fat diet | Proteins | Apolipoprotein E | Rodents | CXCL16 protein | Lesions | Tenascin C | Categories | Inflammation | LOX-1 protein | Gelatinase B | Polymerase chain reaction | Molecular modelling | Liquid oxygen | Arteriosclerosis | Neutrophil collagenase | Mice | Chemokines
Journal Article
Cancer Science, ISSN 1347-9032, 05/2018, Volume 109, Issue 5, pp. 1660 - 1671
We previously reported that, in a mouse model of mammary cancer, α‐mangostin alone exhibits anti‐metastatic properties. To enhance this anti‐metastatic effect,... 
mouse | mammary cancer metastasis | therapeutics | α‐mangostin | lauric acid | α-mangostin | Original
Journal Article
Acute Medicine & Surgery, ISSN 2052-8817, 10/2018, Volume 5, Issue 4, pp. 350 - 357
Aim To examine whether prolonged hemorrhagic shock (HS) at a mean arterial pressure (MAP) of 40 mmHg leads to brain damage. Methods Rats were anesthetized with... 
hemorrhagic shock | resuscitation | brain function | Apoptosis | ischemia | Brain damage | Rodents
Journal Article
PLoS Medicine, ISSN 1549-1277, 06/2016, Volume 13, Issue 6, p. e1002051
Journal Article