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Laboratory Investigation, ISSN 0023-6837, 2013, Volume 93, Issue 11, pp. 1232 - 1240
The molecular marker of well-differentiated/de-differentiated liposarcomas is MDM2 gene amplification coupled with protein overexpression and wild-type TP53.... 
MDM2 | Nutlin-3A | molecular modeling | TP53 | MDMX | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | DOMAIN | ACTIVATION | RNA | HDMX | P53 PATHWAY | PATHOLOGY | GENE AMPLIFICATION | INHIBITORS | EXPRESSION | Proto-Oncogene Proteins c-mdm2 - genetics | Alternative Splicing | Humans | Middle Aged | Male | Piperazines - metabolism | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - metabolism | Liposarcoma - metabolism | Protein Isoforms - metabolism | Computer Simulation | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Antineoplastic Agents - pharmacology | Cell Differentiation | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-mdm2 - antagonists & inhibitors | Proto-Oncogene Proteins c-mdm2 - metabolism | Imidazoles - metabolism | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Liposarcoma - drug therapy | Liposarcoma - pathology | Tumor Suppressor Protein p53 - metabolism | Models, Molecular | In Situ Hybridization, Fluorescence | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Gene Dosage | Imidazoles - pharmacology | Piperazines - pharmacology | Molecular Dynamics Simulation | Drug Resistance, Neoplasm - genetics | Nuclear Proteins - antagonists & inhibitors | Protein Conformation | Aged | Biomarkers, Tumor - genetics | Polymorphism, Single Nucleotide | Drug Resistance, Neoplasm - physiology | Protein Isoforms - antagonists & inhibitors | Protein Isoforms - genetics | Index Medicus
Journal Article
Genes, Chromosomes and Cancer, ISSN 1045-2257, 02/2017, Volume 56, Issue 2, pp. 147 - 158
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 04/2018, Volume 25, Issue 12, pp. 1 - 2180
Inactivation of p53 contributes significantly to the dismal prognosis of breast tumors, most notably triple-negative breast cancers (TNBCs). How the relief... 
EPITHELIAL-MESENCHYMAL TRANSITION | MICRORNA-30A | MOLECULAR PORTRAITS | BIOCHEMISTRY & MOLECULAR BIOLOGY | E-CADHERIN | EXPRESSION | TUMOR-SUPPRESSOR P53 | CELLULAR PLASTICITY | FEEDBACK LOOP | FAMILY | SNAIL | CELL BIOLOGY | Prognosis | Mesenchyme | p53 Protein | MiRNA | Breast cancer | Drug resistance | Cell migration | Lymph nodes | Tumors | Cell adhesion & migration
Journal Article
Journal Article
Oncotarget, ISSN 1949-2553, 2011, Volume 2, Issue 12, pp. 1165 - 1175
Head and neck squamous cell carcinomas (HNSCC) are a heterogeneous group of tumors with variable presentation and clinical behavior. Despite improvements in... 
SNAI1 | TWIST2 | SNAI2 | TWIST1 | EMT | HNSCC | Index Medicus
Journal Article
PLoS Genetics, ISSN 1553-7390, 04/2017, Volume 13, Issue 4, pp. e1006752 - e1006752
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 01/2017, Volume 23, Issue 1, pp. 273 - 282
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 11/2013, Volume 33, Issue 22, pp. 4473 - 4491
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2003, Volume 278, Issue 14, pp. 12579 - 12589
Histone deacetylase activity is potently inhibited by hydroaximc acid derivatives such as suberoylanilide hydroxamic acid (SAHA) and trichostatin-A (TSA)....