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American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 2014, Volume 307, Issue 3, pp. F326 - F336
Development of higher rates of nondiabetic glomerulosclerosis (GS) in African Americans has been attributed to two coding sequence variants (G1 and G2) in the... 
Kidney disease | Podocyte | Lysosomal membrane permeability | APOL1 risk variants | Necrosis | TRYPANOSOME LYTIC FACTOR | PHYSIOLOGY | LIPID-BINDING PROTEIN | kidney disease | AUTOPHAGIC CELL-DEATH | HUMAN SERUM | TRYPANOLYTIC FACTOR | necrosis | APOLIPOPROTEIN-L | podocyte | HIV-ASSOCIATED NEPHROPATHY | lysosomal membrane permeability | UROLOGY & NEPHROLOGY | AFRICAN-AMERICANS | CHRONIC KIDNEY-DISEASE | STAGE RENAL-DISEASE | Lipoproteins, HDL - genetics | Chloride Channels - drug effects | Glomerulosclerosis, Focal Segmental - genetics | Humans | Actins - metabolism | Lipoproteins, HDL - metabolism | Lysosomes - physiology | Chloroquine - pharmacology | Necrosis - physiopathology | Apolipoproteins - genetics | Glomerulosclerosis, Focal Segmental - ethnology | Genetic Predisposition to Disease - genetics | Chloride Channels - antagonists & inhibitors | Podocytes - metabolism | 4,4'-Diisothiocyanostilbene-2,2'-Disulfonic Acid - pharmacology | Apolipoproteins - metabolism | Cells, Cultured | African Americans - genetics | Permeability | Podocytes - pathology | African Americans - ethnology | Podocytes - drug effects | Apolipoprotein L1 | Genetic Predisposition to Disease - ethnology | Genetic Variation - genetics | Lysosomes | Physiological aspects | Development and progression | Cell membranes | Apolipoproteins | Health aspects | Cells | African Americans | Hypoxia | Hydrogen peroxide | Kidney diseases | Human immunodeficiency virus--HIV | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0167071 - e0167071
Background Cigarette smoking plays an important role in the progression of chronic kidney disease (CKD). Nicotine, one of the major components of cigarette... 
ACETYLCHOLINE-RECEPTORS | CIGARETTE-SMOKING | NMDA RECEPTORS | RISK-FACTOR | MULTIDISCIPLINARY SCIENCES | RENAL-FUNCTION | INJURY | DIABETIC-NEPHROPATHY | CHRONIC KIDNEY-DISEASE | CARDIOVASCULAR MORBIDITY | ROS PRODUCTION | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Apoptosis - genetics | Male | Nicotinic Antagonists - pharmacology | Spin Labels | Reactive Oxygen Species - agonists | Nicotine - pharmacology | MAP Kinase Kinase 4 - metabolism | Cyclic N-Oxides - pharmacology | Mitogen-Activated Protein Kinase 1 - genetics | Female | MAP Kinase Kinase 4 - antagonists & inhibitors | Membrane Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Receptors, Nicotinic - metabolism | Podocytes - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Membrane Proteins - genetics | Tissue Culture Techniques | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | Nicotine - antagonists & inhibitors | Podocytes - cytology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Podocytes - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Acetylcysteine - pharmacology | MAP Kinase Kinase 4 - genetics | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Oxidative Stress - drug effects | Receptors, Nicotinic - genetics | Cell Line, Transformed | Mitogen-Activated Protein Kinase 1 - metabolism | Oxidative stress | Physiological aspects | Research | Health aspects | Health/Alcohol and other Drugs/Information/Nicotine and Tobacco | Nicotine | Smoking | Apoptosis | Cell proliferation | Reactive oxygen species | Phosphorylation | Laboratories | Syngeneic grafts | Downstream effects | Tempol | Acetylcysteine | Superoxide dismutase | Antagonists | mRNA | Medical schools | Antioxidants | Proteins | Receptors | Rodents | Mesangial cells | Localization | Medical research | Extracellular signal-regulated kinase | JNK protein | Superoxide | Permeability | Gene expression | Cigarette smoking | Signaling | Molecular modelling | Inhibitors | Acetylcholine receptors (nicotinic) | In vivo methods and tests | Acetylcholine | Kidney diseases | Diabetes | Kidney transplantation | Index Medicus
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 3582 - 16
APOL1-miR193a axis participates in the preservation of molecular phenotype of differentiated podocytes (DPDs). We examined the hypothesis that APOL1 (G0)... 
KIDNEY-DISEASE | MICRORNA-193A | PROTEIN | NEPHRIN | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | APOL1 RISK VARIANTS | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | CYTOTOXICITY | EXPRESSION | PREDICTION | Creatinine | Cathepsin L | Phenotypes | Preservation | Actin | Transgenic mice | Dynamin | Cytoskeleton | Nuclear transport
Journal Article
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 2013, Volume 304, Issue 11, pp. 1347 - 1357
Journal Article
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 04/2013, Volume 93, Issue 4, pp. 623 - 631
HIV‐induced T cell loss was mediated through ROS generation in response to HIV‐induced VDR methylation and associated activation of the RAS. Epigenetics... 
DNA methyltransferase | renin angiotensin system | double‐strand break | reactive oxygen species | Reactive oxygen species | Renin angiotensin system | Double-strand break | APOPTOSIS | OXIDATIVE DAMAGE | NEGATIVE ENDOCRINE REGULATOR | VITAMIN-D-RECEPTOR | AIDS | IMMUNOLOGY | CELL BIOLOGY | RENIN-ANGIOTENSIN SYSTEM | DNA | double-strand break | IMMUNODEFICIENCY VIRUS-INFECTION | HEMATOLOGY | EXPRESSION | LYMPHOCYTES | ras Proteins - genetics | Reactive Oxygen Species - metabolism | Catalase - antagonists & inhibitors | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | ras Proteins - metabolism | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Angiotensin II Type 1 Receptor Blockers - pharmacology | Receptors, Calcitriol - genetics | T-Lymphocytes - virology | DNA (Cytosine-5-)-Methyltransferases - metabolism | Calcitriol - analogs & derivatives | DNA Methylation | HIV-1 - physiology | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Antimetabolites, Antineoplastic - pharmacology | T-Lymphocytes - pathology | Receptors, Calcitriol - antagonists & inhibitors | Catalase - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Losartan - pharmacology | ras Proteins - antagonists & inhibitors | Receptors, Calcitriol - metabolism | Catalase - metabolism | Azacitidine - pharmacology | DNA (Cytosine-5-)-Methyltransferases - genetics | Calcitriol - pharmacology | DNA Repair | CpG Islands | Lymphocyte Count | Apoptosis | Index Medicus | Host Defense & Pathophysiology
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 10/2013, Volume 94, Issue 4, pp. 769 - 777
Journal Article