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Journal of Dermatological Science, ISSN 0923-1811, 02/2019, Volume 93, Issue 2, pp. 133 - 135
Journal Article
New Journal of Chemistry, ISSN 1144-0546, 2016, Volume 40, Issue 11, pp. 9507 - 9519
Ethylene diamine tetra acetate (EDTA) capped Fe3O4 nanoparticles (NPs) have been synthesized and encapsulated using polymeric micelles. The synthesized... 
Journal Article
Journal of Dermatological Science, ISSN 0923-1811, 02/2019, Volume 93, Issue 2, pp. 133 - 135
Journal Article
BMC Genomics, ISSN 1471-2164, 01/2014, Volume 15, Issue Suppl 2, pp. P39 - P39
Journal Article
ISSN 1144-0546, 10/2016, Volume 4, Issue 11, pp. 957 - 9519
Ethylene diamine tetra acetate (EDTA) capped Fe 3 O 4 nanoparticles (NPs) have been synthesized and encapsulated using polymeric micelles. The synthesized Fe 3... 
Journal Article
New Journal of Chemistry, ISSN 1144-0546, 2016, Volume 40, Issue 11, pp. 9507 - 9519
Ethylene diamine tetra acetate (EDTA) capped Fe3O4 nanoparticles (NPs) have been synthesized and encapsulated using polymeric micelles. The synthesized... 
Journal Article
PLoS ONE, 03/2016, Volume 11, Issue 3
Autoimmune hypothyroidism is known to be caused by immune responses related to the thyroid gland and its immunological feature includes presence of autoimmune... 
Journal Article
Autoimmunity reviews, 01/2015, Volume 14, Issue 1, p. 49
Vitiligo is a hypomelanotic autoimmune skin disease arising from a breakdown in immunological self-tolerance, which leads to aberrant immune responses against... 
Vitiligo - therapy | Animals | Vitiligo - pathology | Melanocytes - cytology | Humans | Vitiligo - immunology | Self Tolerance | T-Lymphocytes, Regulatory - immunology
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2017, Volume 12, Issue 7, p. e0180958
Background Autoimmunity has been implicated in the destruction of melanocytes from vitiligo skin. Major histocompatibility complex (MHC) class-II linked genes... 
GENERALIZED VITILIGO | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | CLASS-I | SUSCEPTIBILITY LOCI | GENETIC-VARIANTS | MAJOR HISTOCOMPATIBILITY COMPLEX | MUTATIONS | PROGRESSION | PEPTIDE | GENOME-WIDE ASSOCIATION | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Humans | Middle Aged | Computational Biology | Genotype | Male | ATP-Binding Cassette Sub-Family B Member 2 - genetics | Vitiligo - metabolism | Blotting, Western | Case-Control Studies | Proteasome Endopeptidase Complex - genetics | Haplotypes - genetics | Polymorphism, Restriction Fragment Length - genetics | Adolescent | Polymorphism, Single Nucleotide - genetics | Adult | Female | Proteasome Endopeptidase Complex - metabolism | Vitiligo - genetics | ATP-Binding Cassette Sub-Family B Member 2 - metabolism | Autoimmunity | Antigens | Vitiligo | Care and treatment | Genome-wide association studies | Analysis | Genetic aspects | Single nucleotide polymorphisms | Research | Disease | Transcription | Peptides | Destruction | Control methods | Antigen processing | Science | Cytotoxicity | Melanocytes | Biochemistry | Genomes | Single-nucleotide polymorphism | Gene polymorphism | Fragmentation | Consent | Lymphocytes | Population | Tumor necrosis factor-TNF | Genotypes | Antigen presentation | Baldness | Melanoma | TAP protein | Disease control | Patients | Polymerase chain reaction | Amplification | Restriction fragment length polymorphism | Major histocompatibility complex | Skin | Mutation | Autoimmune diseases | Histocompatibility | Transporter | Polymorphism
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, p. e0149441
Autoimmune hypothyroidism is known to be caused by immune responses related to the thyroid gland and its immunological feature includes presence of autoimmune... 
GLOBAL IODINE STATUS | SOLUBLE CTLA-4 | GENOTYPE | MARKER | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | GRAVES-DISEASE | THYROID-DISEASE | POLYMORPHISM | PREVALENCE | EXON-1 | Iodide Peroxidase - immunology | Genetic Predisposition to Disease | Hashimoto Disease - genetics | Genetic Association Studies | Thyroglobulin - metabolism | Humans | RNA, Messenger - genetics | Gene Expression Regulation | 3' Untranslated Regions - genetics | CTLA-4 Antigen - metabolism | Linkage Disequilibrium - genetics | CTLA-4 Antigen - genetics | Exons - genetics | Logistic Models | Male | RNA, Messenger - metabolism | Thyroiditis, Autoimmune - genetics | Case-Control Studies | Haplotypes - genetics | Thyroglobulin - genetics | Polymorphism, Single Nucleotide - genetics | Female | Haplotypes | Pathogenesis | Linkage disequilibrium | Genes | Antibodies | Cytotoxicity | Lymphocytes T | Thyroid gland | Biochemistry | Single-nucleotide polymorphism | Hormones | Gene polymorphism | Risk factors | CTLA-4 protein | Cell growth | Immunology | Lymphocytes | Enzyme-linked immunosorbent assay | Thyroid | Iodine | Linkage analysis | Antigens | Immunoglobulins | Immune response | Olefinic thermoplastic elastomers | Thyroid diseases | Regression analysis | Hypothyroidism | Risk analysis | Gene expression | Patients | Polymerase chain reaction | Genetic variance | Restriction fragment length polymorphism | Genotyping | Gene frequency | Thyroglobulin | 3' Untranslated regions | Pharmacy | Alleles | Diabetes | Autoimmune diseases | Polymorphism
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0164437
Background Neuropeptide Y (NPY) is known to play a role in the regulation of satiety, energy balance, body weight, and insulin release. Interleukin-1beta... 
LEUCINE-7 | SERUM-LIPIDS | PROLINE 7 POLYMORPHISM | GENETIC ASSOCIATION | PREPRONEUROPEPTIDE-Y | MULTIDISCIPLINARY SCIENCES | LEU7PRO POLYMORPHISM | SUSCEPTIBILITY | RETINOPATHY | HAPLOTYPE | SIGNAL PEPTIDE | Haplotypes | Exons | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Male | Risk | Neuropeptide Y - genetics | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Case-Control Studies | Neuropeptide Y - metabolism | Interleukin-1beta - metabolism | Lipids - blood | Adult | Female | Real-Time Polymerase Chain Reaction | Body Mass Index | Promoter Regions, Genetic | Blood Glucose - analysis | Genetic Association Studies | Gene Frequency | Genotype | Linkage Disequilibrium | Alleles | Triglycerides - blood | Polymorphism, Single Nucleotide | Diabetes Mellitus, Type 2 - pathology | Lipoproteins, LDL - blood | Neuropeptide Y | Medical research | Interleukins | Low density lipoproteins | Analysis | Genes | Body weight | Physiological aspects | Medicine, Experimental | Genetic aspects | Blood lipids | Genetic polymorphisms | Lipoproteins (low density) | Diabetic retinopathy | Transcription | Zoology | Linkage disequilibrium | Interleukin | Science | Lipids | Cardiovascular disease | Neuropeptides | Biochemistry | Males | Gene polymorphism | Rodents | Interleukin 1 | Satiety | Population | Lipid metabolism | Deoxyribonucleic acid--DNA | Linkage analysis | Phenotypes | Cytokines | Diabetes mellitus | Health risks | Metabolism | Insulin | Patients | Cholesterol | Polymerase chain reaction | Beta cells | Energy balance | Nutrition research | Lipoproteins | Restriction fragment length polymorphism | Genotyping | Body mass | Insulin resistance | Diabetes | Risk management | Polymorphism | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, p. e107020
Journal Article
Autoimmunity Reviews, ISSN 1568-9972, 2014, Volume 14, Issue 1, pp. 49 - 56
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e81736
Journal Article