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The Journal of Pathology, ISSN 0022-3417, 02/2017, Volume 241, Issue 3, pp. 405 - 419
Journal Article
Virchows Archiv, ISSN 0945-6317, 12/2017, Volume 471, Issue 6, pp. 713 - 720
Typical (TCs) and atypical carcinoids (ACs) are defined based on morphological criteria, and no grading system is currently accepted to further stratify these... 
Pathology | Ki-67 | Heterogeneity | Prognosis | Medicine & Public Health | Lung | Carcinoid | WHO classification | SURVIVAL | MANAGEMENT | PROGNOSTIC-FACTORS | NEOPLASMS | CELL NEUROENDOCRINE CARCINOMA | CLASSIFICATION | PATHOLOGY | TUMORS | SPECIMENS | ATYPICAL PULMONARY CARCINOIDS | Evaluation | Typing | Statistical analysis | Biopsy | Medical prognosis | Lung cancer | Cytology | Statistical methods | Multivariate analysis | Survival | Index Medicus | Original
Journal Article
Histopathology, ISSN 0309-0167, 10/2012, Volume 61, Issue 4, pp. 629 - 643
Di Palma S, Simpson R H W, Marchiò C, Skálová A, Ungari M, Sandison A, Whitaker S, Parry S & Reis‐Filho J S 
(2012) Histopathology   61, 629–643 Salivary duct... 
basal‐like phenotype | molecular subtypes | androgen receptor | basal cytokeratins | HER2 | salivary duct carcinoma | Basal cytokeratins | Molecular subtypes | Salivary ductcarcinoma | Basal-like phenotype | Androgen receptor | PROGNOSTIC FACTORS | INTRADUCTAL GROWTH | PROGESTERONE-RECEPTORS | PAROTID-GLAND | IN-SITU HYBRIDIZATION | PATHOLOGY | TUMORS | basal-like phenotype | NEGATIVE BREAST-CANCER | CELL BIOLOGY | MOLECULAR PORTRAITS | GRADE | EXPRESSION | Immunohistochemistry | Carcinoma in Situ - classification | Carcinoma, Ductal - classification | Oligonucleotide Array Sequence Analysis | Receptor, ErbB-2 - genetics | Carcinoma, Ductal - genetics | Salivary Gland Neoplasms - genetics | Tissue Array Analysis | Carcinoma, Ductal - pathology | Humans | Middle Aged | Transcriptome | Male | Carcinoma in Situ - genetics | In Situ Hybridization | Receptors, Androgen - analysis | Receptors, Androgen - biosynthesis | Aged, 80 and over | Adult | Female | Carcinoma - pathology | Receptor, ErbB-2 - biosynthesis | Biomarkers, Tumor - analysis | Carcinoma in Situ - pathology | Phenotype | Receptors, Androgen - genetics | Carcinoma - genetics | Salivary Ducts - pathology | Aged | Receptor, ErbB-2 - analysis | Salivary Gland Neoplasms - classification | Carcinoma - classification | Salivary Gland Neoplasms - pathology | Androgens | Epidermal growth factor | Analysis | Genetic aspects | Progesterone | Gene expression | Histochemistry | Cancer | Index Medicus
Journal Article
The Journal of Pathology, ISSN 0022-3417, 03/2015, Volume 235, Issue 4, pp. 571 - 580
Mutations in genes encoding proteins involved in RNA splicing have been found to occur at relatively high frequencies in several tumour types including... 
breast cancer | spliceostatin A | alternative splicing | drivers | SF3B1 | next‐generation sequencing | Alternative splicing | Breast cancer | Next-generation sequencing | Drivers | Spliceostatin A | DUCTAL CARCINOMAS | MICROPAPILLARY CARCINOMAS | next-generation sequencing | MYELODYSPLASIA | PATHOLOGY | TUMORS | PRE-MESSENGER-RNA | UVEAL MELANOMA | ONCOLOGY | PATHWAY | GENES | CHRONIC LYMPHOCYTIC-LEUKEMIA | SPLICING FACTOR | Carcinoma, Papillary - genetics | Receptors, Estrogen - metabolism | Adenocarcinoma, Mucinous - pathology | Humans | Gene Expression Regulation, Neoplastic | Phosphoproteins - antagonists & inhibitors | Molecular Targeted Therapy | Phosphoproteins - metabolism | Carcinoma, Papillary - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Ribonucleoprotein, U2 Small Nuclear - metabolism | Transfection | RNA Interference | Ribonucleoprotein, U2 Small Nuclear - genetics | Female | Antineoplastic Agents - pharmacology | Adenocarcinoma, Mucinous - drug therapy | Pyrans - pharmacology | Cell Survival - drug effects | Genetic Predisposition to Disease | Alternative Splicing - genetics | RNA Splicing Factors | Carcinoma, Papillary - drug therapy | Phosphoproteins - genetics | Breast Neoplasms - drug therapy | Carcinoma, Papillary - pathology | Phenotype | Alternative Splicing - drug effects | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Adenocarcinoma, Mucinous - metabolism | Cell Line, Tumor | Ribonucleoprotein, U2 Small Nuclear - antagonists & inhibitors | Adenocarcinoma, Mucinous - genetics | Mutation | Spiro Compounds - pharmacology | RNA | Leukemia | Genomics | Melanoma | Proteins | Analysis | Pancreatic cancer | Genetic research | Genetic aspects | Nucleotide sequencing | Health aspects | DNA sequencing | Index Medicus | Original Papers
Journal Article
The Journal of Pathology, ISSN 0022-3417, 10/2015, Volume 237, Issue 2, pp. 166 - 178
Journal Article
Cancers, ISSN 2072-6694, 04/2019, Volume 11, Issue 4, p. 454
Hotspot codon 132 mutations (R132xIDH1m) are frequent in intrahepatic cholangiocarcinoma (ICC), are druggable by anti-IDH1m agents, and could represent a... 
IDH1 mutation | Liquid biopsy | Biomarker | qPCR | Intrahepatic cholangiocarcinoma | ISOCITRATE DEHYDROGENASE 1 | PATHOGENESIS | ONCOLOGY | biomarker | intrahepatic cholangiocarcinoma | liquid biopsy
Journal Article
Journal Article
Frontiers in Oncology, ISSN 2234-943X, 2013, Volume 3, pp. 129 - 129
HER2 overexpression and anti-HER2 agents represent probably the best story of success of individualized therapy in breast cancer. Due to the important... 
Therapy | CEP17 amplification | Test | Diagnosis | HER2 | HER2 mutations | Truncated HER2 | diagnosis | test | therapy | truncated HER2
Journal Article
Nature Genetics, ISSN 1061-4036, 11/2014, Volume 46, Issue 11, pp. 1166 - 1169
Journal Article
Journal Article