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Cold Spring Harbor Perspectives in Biology, 10/2018, Volume 10, Issue 10
Endothelial cell–cell adherens junctions (AJs) supervise fundamental vascular functions, such as the control of permeability and transmigration of circulating... 
CCM2 protein | Leukocyte migration | Blood vessels | Adherens junctions | Permeability | Leukocytes | Cadherin | Endothelial cells | White blood cells | Vascular diseases | Human pathology | Regulatory mechanisms (biology) | Mutation | Cell migration
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 07/2015, Volume 112, Issue 27, p. 8421
  Cerebral cavernous malformation (CCM) is a disease of the central nervous system causing hemorrhage-prone multiple lumen vascular malformations and very... 
Rodents | Medical treatment | Nervous system | Pharmacology | Cells
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 8/2006, Volume 174, Issue 4, pp. 593 - 604
Receptor endocytosis is a fundamental step in controlling the magnitude, duration, and nature of cell signaling events. Confluent endothelial cells are contact... 
Receptors | Endocytosis | Phosphorylation | Cell growth | Internalization | Cell lines | Antibodies | Small interfering RNA | Cadherins | Endothelial cells | GROWTH-FACTOR RECEPTOR | TYROSINE PHOSPHORYLATION | MEDIATED ENDOCYTOSIS | VE-CADHERIN | CELL-PROLIFERATION | TGF-BETA RECEPTOR | ADHERENS JUNCTIONS | DNA-SYNTHESIS | PLASMA-MEMBRANE | NUCLEAR TRANSLOCATION | CELL BIOLOGY | Cadherins - metabolism | Humans | Protein Tyrosine Phosphatases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 | Antigens, CD - metabolism | Intracellular Membranes - ultrastructure | Endothelial Cells - ultrastructure | Vascular Endothelial Growth Factor Receptor-2 - agonists | Cell Membrane - metabolism | Protein Phosphatase 1 | Vascular Endothelial Growth Factor A - pharmacology | Phospholipase C gamma - metabolism | Endothelial Cells - metabolism | Endocytosis - drug effects | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Enzyme Activation - drug effects | Endocytosis - physiology | Cell Compartmentation - physiology | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Cell Compartmentation - drug effects | Signal Transduction - physiology | Cell Proliferation - drug effects | Enzyme Activation - physiology | Intracellular Membranes - metabolism | Vascular endothelial growth factor | Protein kinases | Research
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 498, Issue 7455, pp. 492 - 496
Journal Article
Nature Communications, ISSN 2041-1723, 2012, Volume 3, Issue 1, p. 1208
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 3/2014, Volume 355, Issue 3, pp. 515 - 522
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 07/2015, Volume 112, Issue 27, pp. 8421 - 8426
Cerebral cavernous malformation (CCM) is a disease of the central nervous system causing hemorrhage-prone multiple lumen vascular malformations and very severe... 
Cerebral cavernous malformation | Sulindac metabolites | catenin | Endothelial cells | Vascular pathology | cerebral cavernous malformation | beta-catenin | endothelial cells | SIGNALING CONTROLS | MULTIDISCIPLINARY SCIENCES | BLOOD-BRAIN-BARRIER | EFFECTOR | CEREBRAL CAVERNOUS MALFORMATIONS | vascular pathology | PATHWAY | RAP1 | DISEASE | JUNCTIONS | GROWTH | sulindac metabolites | Immunohistochemistry | Central Nervous System Neoplasms - metabolism | Endothelial Cells - metabolism | Central Nervous System Neoplasms - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Hemangioma, Cavernous, Central Nervous System - drug therapy | beta Catenin - metabolism | Hemangioma, Cavernous, Central Nervous System - genetics | Hemangioma, Cavernous, Central Nervous System - metabolism | beta Catenin - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Mice, Knockout | Intracellular Signaling Peptides and Proteins - deficiency | Sulindac - analogs & derivatives | Animals | Signal Transduction - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Central Nervous System Neoplasms - drug therapy | Intracellular Signaling Peptides and Proteins - genetics | Sulindac - pharmacology | Transforming Growth Factor beta - metabolism | Disease Models, Animal | Usage | Sulindac | Genetic aspects | Research | Drug therapy | Health aspects | Cerebrovascular disease | Biological Sciences | β-catenin | Other Medical Sciences not elsewhere specified | Other Medical Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Annan medicin och hälsovetenskap | Övrig annan medicin och hälsovetenskap
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 5/2003, Volume 161, Issue 4, pp. 793 - 804
Confluent endothelial cells respond poorly to the proliferative signals of VEGF. Comparing isogenic endothelial cells differing for vascular endothelial... 
Phosphorylation | Cell growth | Human umbilical vein endothelial cells | Receptors | Phosphatases | Cell lines | Antibodies | Cadherins | Endothelial cells | Cells | Proliferation | Vascular endothelial growth fator | Catenins | Endothelium | endothelium | ADHERENS JUNCTION | ACTIVATION | proliferation | catenins | cadherins | CELL-GROWTH | DNA-SYNTHESIS | PLC-GAMMA | CELL BIOLOGY | SIGNAL-TRANSDUCTION | vascular endothelial growth factor | IN-VIVO | GROWTH-FACTOR | PROTEIN-TYROSINE-PHOSPHATASE | N-CADHERIN | Endothelium, Vascular - cytology | Vascular Endothelial Growth Factor A | Cadherins - metabolism | Vascular Endothelial Growth Factors | Humans | Endothelium, Vascular - drug effects | Protein Tyrosine Phosphatases - metabolism | Endothelial Growth Factors - pharmacology | Receptor-Like Protein Tyrosine Phosphatases, Class 3 | Dose-Response Relationship, Drug | Cytoskeletal Proteins - metabolism | Lymphokines - pharmacology | Cadherins - genetics | Cell Line | beta Catenin | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Cell Division - drug effects | Contact Inhibition | Animals | Intercellular Signaling Peptides and Proteins - pharmacology | Protein Binding | Trans-Activators - metabolism | Mice | Antigens, CD | Mitogen-Activated Protein Kinases - metabolism | endothelium; cadherins; catenins; vascular endothelial growth factor; proliferation
Journal Article
Circulation Research, ISSN 0009-7330, 09/2017, Volume 121, Issue 8, p. 981
Correct organization of the vascular tree requires the balanced activities of several signaling pathways that regulate tubulogenesis and vascular branching,... 
Journal Article
Journal Article
Journal of Cell Science, ISSN 0021-9533, 1998, Volume 111, Issue 13, pp. 1853 - 1865
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 08/2006, Volume 174, Issue 4, p. 593
  Receptor endocytosis is a fundamental step in controlling the magnitude, duration, and nature of cell signaling events. Confluent endothelial cells are... 
Proteins | Cellular biology | Protease inhibitors
Journal Article
Cold Spring Harbor perspectives in biology, ISSN 1943-0264, 10/2017, Volume 9, Issue 10, p. a033720
Journal Article