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Cytokine and Growth Factor Reviews, ISSN 1359-6101, 08/2017, Volume 36, pp. 107 - 116
Journal Article
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 03/2018, Volume 103, Issue 3, pp. 493 - 500
Heart failure (HF), the final stage of pathological cardiac hypertrophy, is a major cause of hospitalization and mortality. The role of inflammation in the... 
regulatory T cells | cardiovascular disease | heart failure | abatacept | immunotherapy | PRESSURE-OVERLOAD | MYOCARDIAL-INFARCTION | HEART-FAILURE | CD4(+) T-CELLS | IMMUNOLOGY | CELL BIOLOGY | FAILING HEART | CARDIAC-HYPERTROPHY | IN-VIVO | MICE | INFLAMMATORY CYTOKINES | HEMATOLOGY | LYMPHOCYTES
Journal Article
Stroke, ISSN 0039-2499, 08/2019, Volume 50, Issue 8, pp. 2207 - 2215
BACKGROUND AND PURPOSE—After ischemic injury, microglia and infiltrated macrophages may acquire different polarization phenotypes promoting inflammation and... 
FUNCTIONAL RECOVERY | INTERLEUKIN-4 | ACTIVATION | brain ischemia | MACROPHAGES | mice | CLINICAL NEUROLOGY | STROKE | RESPONSES | MONOCYTE | inflammation | PERIPHERAL VASCULAR DISEASE | cytokines | microglia | POLARIZATION | T-CELLS
Journal Article
Microscopy Research and Technique, ISSN 1059-910X, 06/2017, Volume 80, Issue 6, pp. 563 - 569
Journal Article
Journal Article
Rheumatology International, ISSN 0172-8172, 2/2019, Volume 39, Issue 2, pp. 239 - 243
To estimate biologic influence on heart failure (HF) risk in rheumatoid arthritis. Retrospective cohort (RECORD Study of Italian Society for Rheumatology)... 
Medicine & Public Health | DMARDs | Cardiovascular | Epidemiology | Biologic | Rheumatology | TUMOR-NECROSIS-FACTOR | SAFETY | DISEASE | RHEUMATOLOGY
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2015, Volume 135, Issue 1, pp. 109 - 124
Changes in the homeostasis of tumor necrosis factor α (TNFα) have been demonstrated in patients and experimental models of amyotrophic lateral sclerosis (ALS).... 
tumor necrosis factor receptor 2 | tumor necrosis factor α | amyotrophic lateral sclerosis | neuromuscular junctions | motor neuron | CYTOKINES | BIOCHEMISTRY & MOLECULAR BIOLOGY | SPINAL-CORD | ALS PATIENTS | ASTROCYTES | NEUROSCIENCES | TUMOR-NECROSIS-FACTOR | IN-VITRO | tumor necrosis factor alpha | REGULATORY T-CELLS | EXTEND SURVIVAL | TRANSGENIC MOUSE MODEL | EXPRESSION | Amyotrophic Lateral Sclerosis - genetics | Coculture Techniques | Cells, Cultured | Axons - metabolism | Disease Progression | Receptors, Tumor Necrosis Factor, Type II - deficiency | DNA-Binding Proteins - metabolism | Motor Neurons - metabolism | Animals | Cell Death - physiology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Amyotrophic Lateral Sclerosis - metabolism | Neuroglia - metabolism | Mice | Astrocytes - metabolism | Disease Models, Animal | Amyotrophic lateral sclerosis | Tumor necrosis factor | Neurons | Analysis | Protein binding | Neurochemistry | Mutation | Tumor necrosis factor receptors | Animal models | Homeostasis | Superoxide dismutase | Neuronal-glial interactions | Proteins | Receptors | Tubulin | Clonal deletion | Neurodegeneration | Nerves | Deletion | Tumor necrosis factor-TNF | Degeneration | Deoxyribonucleic acid--DNA | Motor neurons | Astrocytes | Mortality | Muscles | Cultures | Tumor necrosis factor-α | Survival | Cell death | Death
Journal Article