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Journal Article
Angewandte Chemie International Edition, ISSN 1433-7851, 06/2019, Volume 58, Issue 24, pp. 7997 - 8001
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e109842
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 03/2019, Volume 139, Issue 11, pp. 1407 - 1421
Journal Article
Scientific Reports, ISSN 2045-2322, 06/2016, Volume 6, Issue 1, p. 27517
Hydrogen sulfide (H2S) is known to act protectively during renal ischemia/reperfusion injury (IRI). However, the role of the endogenous H2S in acute kidney... 
SYNTHASE | ISCHEMIA/REPERFUSION INJURY | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | MICE | GENERATION | DYSFUNCTION | HYPERTENSION | FAILURE | ENDOGENOUS HYDROGEN-SULFIDE | PROTECTS
Journal Article
International Journal of Rehabilitation Research, ISSN 0342-5282, 03/2018, Volume 41, Issue 1, pp. 81 - 83
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 11/2019, Volume 10, Issue 1, pp. 5155 - 14
Gain-of-function mutations in the chloride channel ClC-2 were recently described as a cause of familial hyperaldosteronism type II (FH-II). Here, we report the... 
CARDIOVASCULAR EVENTS | MULTIDISCIPLINARY SCIENCES | PLASMA-RENIN | DIETARY SALT | INVALIDATION | CHANNEL MUTATIONS | MICE | PREVALENCE | HYPERTENSION | SOMATIC MUTATIONS | ADENOMAS | Hypertension | Efflux | Calcium | Adrenal glands | Aldosterone synthase | Homology | Aldosterone | Depolarization | Chloride ions | Missense mutation | Renin | Sodium | Chlorides | Morphology | Ion channels | Blood pressure | Mutation
Journal Article
Journal Article
Nature Reviews Nephrology, ISSN 1759-5061, 2019, Volume 15, Issue 9, pp. 546 - 558
Sodium intake is undoubtedly indispensable for normal body functions but can be detrimental when taken in excess of dietary requirements. The consequences of... 
II-INDUCED HYPERTENSION | DIETARY SALT INTAKE | ARYL-HYDROCARBON RECEPTOR | OSMOTICALLY INACTIVE NA+ | GUT MICROBIOME | UROLOGY & NEPHROLOGY | SENSITIVE HYPERTENSION | INNATE LYMPHOID-CELLS | URINARY SODIUM | CD8(+) T-CELLS | BLOOD-PRESSURE
Journal Article