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by de Vette, Ivar and Lombardo, Angelo and Berkhout, Ben and Vervoordeldonk, Margriet and Dekkers, Johanna F and Kruisselbrink, Evelien and Vonk, Annelotte M and de Jonge, Hugo R and de Winter, Karin M and de Groot, – and Janssens, Hettie M and Bronsveld, Inez and Nieuwenhuis, Edward E.S and Houwen, Roderick H.J and Vleggaar, Frank P and Escher, Hankje C and Clevers, Hans and van der Ent, Cornelis K and Buckland, Karen and Rivat, Christine and Himoudi, Nourredine and Gilmour, Kimberly and Booth, Claire and Cornetta, Kenneth and Kohn, Don B and Carbonaro, Denise and Paruzynski, Anna and Schmidt, Manfred and Thrasher, Adrian J and Schaffer, David and Hwang, Tae-Ho and Zinn, E and Khaychuk, V and Sarkar, D and Carvalho, L and Pacouret, S and Morris, H and Plovie, E and van Gent, Michiel and Gram, Anna and Boer, Ingrid and Horst, Danielle and Zaldumbide, Arnaud and Hoeben, Rob and Wiertz, Emmanuel and Maring, J and Smits, AM and Ouyang, Hong and Lin, Ying and Wang, Yujuan and Cai, Humim and Li, Gen and Patel, Sherrina and Zhu, Jie and Lin, Danni and Wen, Cindy and Zhu, Jin and Tsaalbi-Shtylik, Anastasia and Martín-Pardillos, Ana and Ferras, Cristina and Verspuy, Johan and Speksnijder, Ewoud and Jansen, Jacob and Peng, Kah Whye and Federspiel, Mark and Naik, Shruthi and Ruiz, Autumn and Lacy, Martha and Dispenzieri, Angela and O'Donovan, Liz and Arzumanov, Andrey A and Rahim, A.A and Burke, D.G and Buckley, S.M and Cullen, E and Mukherji, S and Mills, K and Sirka, E and Herbert, B and Karlsson, S and Hughes, D.A and Mehta, A.B and Howe, S.J and Heales, S and Cooper, J.D and Cheng, S.H and van Til, Niek P and Liang, Q and Stok, M and Wagemaker, G and Reiss, Ulrike M and Tuddenham, Edward G.D and Nienhuis, Arthur W and Davidoff, Andrew M and Meneghini, V and Lattanzi, A and Tiradani, L and Bravo, G and Morena, F and Martino, S and ...
Human Gene Therapy, ISSN 1043-0342, 11/2014, Volume 25, Issue 11, pp. A1 - A121
Journal Article
BMC CANCER, ISSN 1471-2407, 07/2019, Volume 19, Issue 1, pp. 666 - 26
BackgroundCancer is a rapidly evolving, multifactorial disease that accumulates numerous genetic and epigenetic alterations. This results in molecular and... 
CELLS | TGF-BETA | INTRATUMOR HETEROGENEITY | VALIDATION | Metastasis | DNA METHYLATION MICROARRAY | TUMOR HETEROGENEITY | GENOME | Heterogeneity | EVOLUTION | ONCOLOGY | Cooperation | CPG SITES | Breast | Tumor | MDA-MB-231 | Cancer | Clone | Communication | Clonal selection theory | Care and treatment | Usage | Phenotype | Breast cancer | Diagnosis | Research | Gene expression
Journal Article
Physiological Reports, ISSN 2051-817X, 11/2015, Volume 3, Issue 11, pp. e12626 - n/a
Age‐related effects of the vascular wall have been associated with several hemodynamic dysfunctions, including medial vascular calcification. Vascular aging... 
senescence | Aging | proliferation | vascular smooth muscle cells | vascular calcification | Calcification (ectopic) | Senescence | Transcription factors | Calcium | Smooth muscle | Osteoblasts | Genotype & phenotype | Msx2 protein | Actin | Atherosclerosis | Aorta | Oxidation | Telomerase | Age | Cbfa-1 protein | Phenotypes | Cell division | Gene expression | Calcium phosphates | Animals | Life span | Cell lines | Calcification | Apoptosis
Journal Article
ONCOLOGY LETTERS, ISSN 1792-1074, 09/2019, Volume 18, Issue 3, pp. 2853 - 2860
Adenocarcinomas exhibit great heterogeneity, with many genetic and epigenetic alterations. The Kelch domain-containing protein 7B (KLHDC7B) has recently been... 
Kelch domain-containing protein 7B | BYPASS | DNA METHYLATION | epigenetic | MARKER | PATTERNS | PROLIFERATION | biomarker | RSK4 | ONCOLOGY | methylation | cancer | SENESCENCE | breast
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 4245 - 1
A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has been fixed in the paper. 
NAD | Poly(ADP-ribose) polymerase | Mitochondria | SIRT1 protein
Journal Article
Blood, ISSN 0006-4971, 09/2017, Volume 130, Issue 13, pp. 1523 - 1534
Endogenous DNA damage is causally associated with the functional decline and transformation of stem cells that characterize aging. DNA lesions that have... 
DAMAGE RESPONSE | REPAIR | STEM-CELL FUNCTION | CLONAL HEMATOPOIESIS | BONE-MARROW | TRANSLESION SYNTHESIS | MICE | REPLICATION STRESS | CANCER | POLYMERASE-ZETA
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 03/2018, Volume 8, Issue 1, p. 12480
Nucleic acids, which constitute the genetic material of all organisms, are continuously exposed to endogenous and exogenous damaging agents, representing a... 
MULTIDISCIPLINARY SCIENCES
Journal Article
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