American Journal of Epidemiology, ISSN 0002-9262, 09/2018, Volume 187, Issue 9, pp. 2069 - 2069
Journal Article
The New England journal of medicine, ISSN 0028-4793, 03/2015, Volume 372, Issue 13, pp. 1270 - 1271
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 09/2013, Volume 105, Issue 18, p. 1342
Here, Terry and Tehranifar say Hou and colleagues provide further evidence that the increased breast cancer risk from hormone replacement therapy (HRT) may...
Body mass index | Womens health | Risk assessment | Oncology | Hormone replacement therapy | Clinical outcomes
Body mass index | Womens health | Risk assessment | Oncology | Hormone replacement therapy | Clinical outcomes
Journal Article
Breast Cancer Research, ISSN 1465-5411, 07/2017, Volume 19, Issue 1, pp. 85 - 3
Adolescence | Body size | Breast cancer | BODY-SIZE | RISK | CHILDHOOD | BREAST-CANCER | FATNESS | ONCOLOGY | PREMENOPAUSAL | WEIGHT | YOUNG-WOMEN | LIFE | Body Mass Index | Risk | Breast Neoplasms | Body Size | Humans | Risk Factors | Research | Epidemiology | Oncology, Experimental | Cancer
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 07/2015, Volume 107, Issue 7, p. 1
Clinical guidelines for breast cancer chemoprevention and MRI screening involve estimates of remaining lifetime risk (RLR); in the United States, women with...
Disease prevention | Womens health | Breast cancer | Medical screening
Disease prevention | Womens health | Breast cancer | Medical screening
Journal Article
Occupational and Environmental Medicine, ISSN 1351-0711, 08/2019, Volume 76, Issue 8, pp. 592 - 592
Journal Article
Nature Genetics, ISSN 1061-4036, 2015, Volume 47, Issue 2, pp. 164 - 171
Genome-wide association studies (GWAS) have identified 12 epithelial ovarian cancer (EOC) susceptibility alleles. The pattern of association at these loci is...
BRCA2 MUTATION CARRIERS | BREAST-CANCER | COMMON VARIANTS | METAANALYSIS | HIGH-RISK | MYOPODIN | GENETICS & HEREDITY | HUMAN ELG1 | ABO BLOOD-GROUP | SINGLE NUCLEOTIDE POLYMORPHISMS | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease | Humans | Genotype | Risk | Ovarian Neoplasms - genetics | Young Adult | BRCA1 Protein - genetics | Neoplasms, Glandular and Epithelial - genetics | Adolescent | Alleles | Carcinoma, Ovarian Epithelial | Adult | Female | Heterozygote | Polymorphism, Single Nucleotide | Mutation | Genome-Wide Association Study - methods | Quantitative Trait Loci | BRCA2 Protein - genetics | Genes, Reporter | Quantitative trait loci | Genetic aspects | Identification and classification | Health aspects | Ovarian cancer | Haplotypes | Accuracy | Population | Hormone replacement therapy | Genomes | Meta-analysis | Cèl·lules epitelials | Càncer d'ovari | Developmental genetics | Human genome | Epithelial cells | Genètica del desenvolupament | Nucleotides | Genoma humà | Nucleòtids | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
BRCA2 MUTATION CARRIERS | BREAST-CANCER | COMMON VARIANTS | METAANALYSIS | HIGH-RISK | MYOPODIN | GENETICS & HEREDITY | HUMAN ELG1 | ABO BLOOD-GROUP | SINGLE NUCLEOTIDE POLYMORPHISMS | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease | Humans | Genotype | Risk | Ovarian Neoplasms - genetics | Young Adult | BRCA1 Protein - genetics | Neoplasms, Glandular and Epithelial - genetics | Adolescent | Alleles | Carcinoma, Ovarian Epithelial | Adult | Female | Heterozygote | Polymorphism, Single Nucleotide | Mutation | Genome-Wide Association Study - methods | Quantitative Trait Loci | BRCA2 Protein - genetics | Genes, Reporter | Quantitative trait loci | Genetic aspects | Identification and classification | Health aspects | Ovarian cancer | Haplotypes | Accuracy | Population | Hormone replacement therapy | Genomes | Meta-analysis | Cèl·lules epitelials | Càncer d'ovari | Developmental genetics | Human genome | Epithelial cells | Genètica del desenvolupament | Nucleotides | Genoma humà | Nucleòtids | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
Journal Article
Nature, ISSN 0028-0836, 2017, Volume 551, Issue 7678, pp. 92 - 94
Breast cancer risk is influenced by rare coding variants in susceptibility genes, such as BRCA1, and many common, mostly non-coding variants. However, much of...
COMPREHENSIVE MOLECULAR PORTRAITS | PROTEIN | ANNOTATION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | ARCHITECTURE | PATHWAY ANALYSIS | MUTATIONS | ENHANCERS | EXPRESSION | GENOME-WIDE ASSOCIATION | Multifactorial Inheritance - genetics | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Risk Assessment | Humans | Regulatory Sequences, Nucleic Acid | Asia - ethnology | Asian Continental Ancestry Group - genetics | Binding Sites - genetics | Genetic Loci | Europe - ethnology | Transcription Factors - metabolism | Breast Neoplasms - genetics | Computer Simulation | Polymorphism, Single Nucleotide - genetics | Female | Breast Neoplasms - diagnosis | Quantitative trait loci | Breast cancer | Genetic aspects | Health aspects | Risk factors | Medical research | Transcription factors | BRCA1 protein | Genes | Health risks | Association analysis | Risk | Genomes | Biology | Single-nucleotide polymorphism | Regulatory sequences | Epidemiology | Loci | Medicine | Womens health | Breast | Genetics | Heritability | Health risk assessment | Binding sites | Tumors | Cancer | Medical and Health Sciences | Medicin och hälsovetenskap
COMPREHENSIVE MOLECULAR PORTRAITS | PROTEIN | ANNOTATION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | ARCHITECTURE | PATHWAY ANALYSIS | MUTATIONS | ENHANCERS | EXPRESSION | GENOME-WIDE ASSOCIATION | Multifactorial Inheritance - genetics | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Risk Assessment | Humans | Regulatory Sequences, Nucleic Acid | Asia - ethnology | Asian Continental Ancestry Group - genetics | Binding Sites - genetics | Genetic Loci | Europe - ethnology | Transcription Factors - metabolism | Breast Neoplasms - genetics | Computer Simulation | Polymorphism, Single Nucleotide - genetics | Female | Breast Neoplasms - diagnosis | Quantitative trait loci | Breast cancer | Genetic aspects | Health aspects | Risk factors | Medical research | Transcription factors | BRCA1 protein | Genes | Health risks | Association analysis | Risk | Genomes | Biology | Single-nucleotide polymorphism | Regulatory sequences | Epidemiology | Loci | Medicine | Womens health | Breast | Genetics | Heritability | Health risk assessment | Binding sites | Tumors | Cancer | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
JAMA, ISSN 0098-7484, 06/2017, Volume 317, Issue 23, pp. 2402 - 2416
IMPORTANCE: The clinical management of BRCA1 and BRCA2 mutation carriers requires accurate, prospective cancer risk estimates. OBJECTIVES: To estimate...
PENETRANCE | POSITION | WOMEN | MEDICINE, GENERAL & INTERNAL | SERIES | FAMILIES | SUSCEPTIBILITY | MODEL | Age Distribution | Prospective Studies | Age Factors | Risk Assessment | Humans | Middle Aged | Incidence | Neoplasms, Second Primary - epidemiology | Ovarian Neoplasms - epidemiology | Ovarian Neoplasms - genetics | Breast Neoplasms - genetics | Time Factors | Aged, 80 and over | Genes, BRCA2 | Adult | Family | Female | Aged | Genes, BRCA1 | Mutation | Neoplasms, Second Primary - genetics | Breast Neoplasms - epidemiology | BRCA2 protein | Ovarian carcinoma | BRCA1 protein | Health risks | Oncology | Population studies | Breast cancer | Estimates | Ovarian cancer | Carriers | Consortia | Design modifications | Risk assessment | Data collection | Breast | Genetics | Diagnosis | Genetic testing | Position (location) | Age | Cancer | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
PENETRANCE | POSITION | WOMEN | MEDICINE, GENERAL & INTERNAL | SERIES | FAMILIES | SUSCEPTIBILITY | MODEL | Age Distribution | Prospective Studies | Age Factors | Risk Assessment | Humans | Middle Aged | Incidence | Neoplasms, Second Primary - epidemiology | Ovarian Neoplasms - epidemiology | Ovarian Neoplasms - genetics | Breast Neoplasms - genetics | Time Factors | Aged, 80 and over | Genes, BRCA2 | Adult | Family | Female | Aged | Genes, BRCA1 | Mutation | Neoplasms, Second Primary - genetics | Breast Neoplasms - epidemiology | BRCA2 protein | Ovarian carcinoma | BRCA1 protein | Health risks | Oncology | Population studies | Breast cancer | Estimates | Ovarian cancer | Carriers | Consortia | Design modifications | Risk assessment | Data collection | Breast | Genetics | Diagnosis | Genetic testing | Position (location) | Age | Cancer | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Cancer and Oncology | Medicin och hälsovetenskap | Cancer och onkologi
Journal Article
Pediatrics, ISSN 0031-4005, 11/2016, Volume 138, Issue Supplement, pp. S78 - S80
Family history is 1 of the strongest predictors of most cancers. 1 Yet primary cancer prevention efforts are often not targeted toward individuals at greatest...
MOTHERS | RISK | PEDIATRICS | IMPACT | Disease prevention | Intervention | Families & family life | Risk factors | Cancer
MOTHERS | RISK | PEDIATRICS | IMPACT | Disease prevention | Intervention | Families & family life | Risk factors | Cancer
Journal Article
Epigenetics, ISSN 1559-2294, 07/2011, Volume 6, Issue 7, pp. 828 - 837
Alterations in DNA methylation patterns, both at specific loci and overall in the genome, have been associated with many different health outcomes. In cancer...
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | White blood cells | DNA methylation | Gene-specific | Epidemiology | Global | Risk factors | BLADDER-CANCER | risk factors | HEALTHY-SUBJECTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | global | LYMPHOCYTE DNA | BREAST-CANCER | epidemiology | WOMEN | 5-METHYLCYTOSINE CONTENT | white blood cells | GENETICS & HEREDITY | gene-specific | COLORECTAL ADENOMA | TOBACCO-SMOKE | LEUKOCYTE DNA | MODERATE FOLATE-DEPLETION | Epidemiologic Studies | Smoke - adverse effects | Humans | Risk Factors | Air Pollution - adverse effects | Alcohol Drinking - adverse effects | Environmental Exposure - adverse effects | DNA Methylation | Neoplasms - chemically induced | Neoplasms - genetics | Tobacco - adverse effects | Leukocytes - metabolism | Neoplasms - epidemiology | Review
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | White blood cells | DNA methylation | Gene-specific | Epidemiology | Global | Risk factors | BLADDER-CANCER | risk factors | HEALTHY-SUBJECTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | global | LYMPHOCYTE DNA | BREAST-CANCER | epidemiology | WOMEN | 5-METHYLCYTOSINE CONTENT | white blood cells | GENETICS & HEREDITY | gene-specific | COLORECTAL ADENOMA | TOBACCO-SMOKE | LEUKOCYTE DNA | MODERATE FOLATE-DEPLETION | Epidemiologic Studies | Smoke - adverse effects | Humans | Risk Factors | Air Pollution - adverse effects | Alcohol Drinking - adverse effects | Environmental Exposure - adverse effects | DNA Methylation | Neoplasms - chemically induced | Neoplasms - genetics | Tobacco - adverse effects | Leukocytes - metabolism | Neoplasms - epidemiology | Review
Journal Article