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index medicus (118) 118
gastroenterology & hepatology (89) 89
animals (82) 82
humans (73) 73
male (58) 58
rats (46) 46
gastroenterology and hepatology (37) 37
liver (36) 36
apoptosis (29) 29
article (28) 28
proliferation (27) 27
liver diseases (26) 26
rats, inbred f344 (26) 26
cell proliferation (25) 25
mice (25) 25
expression (24) 24
cholestasis (23) 23
bile (20) 20
female (20) 20
intrahepatic biliary epithelium (19) 19
research (19) 19
abridged index medicus (18) 18
primary sclerosing cholangitis (18) 18
bile ducts - cytology (17) 17
bile ducts - metabolism (17) 17
bile ducts - pathology (17) 17
epithelial-cells (17) 17
liver - pathology (17) 17
physiology (17) 17
cirrhosis (16) 16
growth (16) 16
ligation (16) 16
rodents (16) 16
cholangiocarcinoma (15) 15
cholangiocytes (15) 15
cyclic amp - metabolism (15) 15
disease models, animal (15) 15
duct-ligated rats (15) 15
functional-heterogeneity (14) 14
risk factors (14) 14
cholangiocarcinoma - pathology (13) 13
liver - metabolism (13) 13
middle aged (13) 13
protein-kinase-c (13) 13
signal transduction (13) 13
apoptosis - drug effects (12) 12
cancer (12) 12
cell proliferation - drug effects (12) 12
cells (12) 12
fibrosis (12) 12
physiological aspects (12) 12
aged (11) 11
bile ducts, intrahepatic - pathology (11) 11
biliary epithelium (11) 11
hepatic stellate cells (11) 11
hepatocytes (11) 11
ligated rats (11) 11
primary biliary-cirrhosis (11) 11
bile duct neoplasms - pathology (10) 10
bile ducts - drug effects (10) 10
cell division - drug effects (10) 10
cells, cultured (10) 10
cholestasis - metabolism (10) 10
digestive system (10) 10
phosphorylation (10) 10
activation (9) 9
bile acids and salts - metabolism (9) 9
cholangiocyte proliferation (9) 9
cholangitis (9) 9
digestive system diseases (9) 9
disease progression (9) 9
gene expression (9) 9
genetic aspects (9) 9
growth factors (9) 9
liver cirrhosis (9) 9
normal rat-liver (9) 9
secretin (9) 9
analysis (8) 8
care and treatment (8) 8
cholangiopathies (8) 8
cholestasis - pathology (8) 8
diagnosis (8) 8
pathology (8) 8
protein kinase c - metabolism (8) 8
protein kinases (8) 8
secretin - metabolism (8) 8
tumors (8) 8
ursodeoxycholic acid (8) 8
bicarbonate secretion (7) 7
bile ducts (7) 7
bile ducts, intrahepatic - metabolism (7) 7
bile-acid transporter (7) 7
biliary tree (7) 7
calcium - metabolism (7) 7
cell line, tumor (7) 7
cell-proliferation (7) 7
cholangiocarcinoma - metabolism (7) 7
cholangiocyte (7) 7
cholestasis - physiopathology (7) 7
down-regulation (7) 7
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1. Full Text Silvia Martini, Cecilia Carubbi, Elena Masselli, Maria Gildone, Giulia Pozzi, Daniela Grandi, Marco Vitale
by Chiara Avellini and Caterina Licini and Raffaella Lazzarini and Antonio D Procopio and Giovanni Muzzonigro and Giovanni Tossetta and Roberta Mazzucchelli and Rosaria Gesuita and Mario Castellucci and Fabiola Olivieri and Daniela Marzioni
Italian Journal of Anatomy and Embryology, ISSN 1122-6714, 01/2015, Volume 120, Issue 1, p. 46
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2. Full Text Long-term albumin administration in decompensated cirrhosis (ANSWER): an open-label randomised trial
by Caraceni, Paolo and Riggio, Oliviero and Angeli, Paolo and Alessandria, Carlo and Neri, Sergio and Neri, Elga and Foschi, Francesco G and Levantesi, Fabio and Airoldi, Aldo and Boccia, Sergio and Svegliati-Baroni, Gianluca and Fagiuoli, Stefano and Romanelli, Roberto G and Cozzolongo, Raffaele and Di Marco, Vito and Sangiovanni, Vincenzo and Morisco, Filomena and Toniutto, Pierluigi and Tortora, Annalisa and De Marco, Rosanna and Angelico, Mario and Cacciola, Irene and Elia, Chiara and Elia, Gianfranco and Federico, Alessandro and Massironi, Sara and Guarisco, Riccardo and Galioto, Alessandra and Ballardini, Giorgio and Rendina, Maria and Nardelli, Silvia and Piano, Salvatore and Prestianni, Loredana and Cappa, Federica Mirici and Cesarini, Lucia and Simone, Loredana and Pasquale, Chiara and Cavallin, Marta and Andrealli, Alida and Fidone, Federica and Ruggeri, Matteo and Roncadori, Andrea and Baldassarre, Maurizio and Tufoni, Manuel and Zaccherini, Giacomo and Bernardi, Mauro and Domenicali, Marco and Giannone, Ferdinando A and Merli, Manuela and Gioia, Stefania and Fasolato, Silvano and Sticca, Antonietta and Campion, Daniela and Risso, Alessandro and Saracco, Giorgio M and Maiorca, Daniela and Rizzotto, Agostino and Lanzi, Arianna and Visani, Anna and Mastroianni, Antonio and Alberti, Alberto B and Mazzarelli, Chiara and Vangeli, Marcello and Marzioni, Marco and Capretti, Francesca and Kostandini, Alba and Magini, Giulia and Colpani, Maria and Laffi, Giacomo and Gabbani, Tommaso and Marsico, Maria and Zappimbulso, Marianna and Petruzzi, Josè and Calvaruso, Vincenza and Parrella, Giovanni and Caporaso, Nicola and Auriemma, Francesco and Guarino, Maria and Pugliese, Fabio and Gasbarrini, Antonio and Leo, Pietro and De Leonardis, Francesco and Pecchioli, Alessandra and Rossi, Piera and Raimondo, Giovanni and Negri, Elisa and Dallio, Marcello and Loguercio, Carmelina and Conte, Dario and Celli, Natascia and Bringiotti, Roberto and Castellaneta, Nicola M and Salerno, Francesco and ANSWER Study Investigators
The Lancet, ISSN 0140-6736, 06/2018, Volume 391, Issue 10138, pp. 2417 - 2429
Evidence is scarce on the efficacy of long-term human albumin (HA) administration in patients with decompensated cirrhosis. The human Albumin for the treatmeNt... 
SURVIVAL | MEDICINE, GENERAL & INTERNAL | INFECTIONS | ASCITES | MANAGEMENT | IMPROVES | PLASMA EXPANDERS | NATURAL-HISTORY | LIVER-DISEASE | PERIPHERAL ARTERIAL VASODILATION | FAILURE | Hyponatremia - chemically induced | Furosemide - administration & dosage | Humans | Middle Aged | Male | Albumins - therapeutic use | Furosemide - adverse effects | Time Factors | Ascites - therapy | Female | Drug Therapy, Combination | Quality-Adjusted Life Years | Liver Cirrhosis - drug therapy | Ascites - etiology | Diuretics - administration & dosage | Liver Cirrhosis - complications | Kaplan-Meier Estimate | Survival Rate | Mineralocorticoid Receptor Antagonists - therapeutic use | Hyperkalemia - chemically induced | Quality of Life | Aged | Liver Cirrhosis - physiopathology | Diuretics - adverse effects | Paracentesis | Albumin | Analysis | Bacterial infections | Liver | Mortality | Medical treatment | Medical services | Clinical trials | Population studies | Survival | Patients | Furosemide | Quality of life | Cirrhosis | Randomization | Hospitals | Motivation | Medical prognosis | Cost analysis | Ascites | Index Medicus | Abridged Index Medicus
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3. Full Text Safety and efficacy of ombitasvir/paritaprevir/ritonavir/dasabuvir plus ribavirin in patients over 65 years with HCV genotype 1 cirrhosis
by Ascione, Antonio and De Luca, Massimo and Melazzini, Mario and Montilla, Simona and Trotta, Maria Paola and Petta, Salvatore and Puoti, Massimo and Sangiovanni, Vincenzo and Messina, Vincenzo and Bruno, Savino and Izzi, Antonio and Villa, Erica and Aghemo, Alessio and Zignego, Anna Linda and Orlandini, Alessandra and Fontanella, Luca and Gasbarrini, Antonio and Marzioni, Marco and Giannini, Edoardo G and Craxì, Antonio and ABACUS Study Group and For the ABACUS Study Group
Infection, ISSN 0300-8126, 10/2018, Volume 46, Issue 5, pp. 607 - 615
To analyse safety and efficacy of treatment based on ombitasvir/paritaprevir/ritonavir/dasabuvir plus ribavirin in the sub-group of GT1 patients older than... 
Cirrhosis | Ombitasvir | Medicine & Public Health | General Practice / Family Medicine | Infectious Diseases | Internal Medicine | Paritaprevir | Elderly | Dasabuvir | Humans | Hepacivirus - genetics | Hepatitis C, Chronic - virology | Male | Carbamates - administration & dosage | Hepatitis C, Chronic - complications | Uracil - administration & dosage | Ribavirin - administration & dosage | Aged, 80 and over | Female | Drug Therapy, Combination | Liver Cirrhosis - etiology | Liver Cirrhosis - diagnosis | Ritonavir - administration & dosage | Antiviral Agents - therapeutic use | Genotype | Treatment Outcome | Hepatitis C, Chronic - diagnosis | Hepatitis C, Chronic - drug therapy | Antiviral Agents - administration & dosage | Anilides - administration & dosage | Antiviral Agents - adverse effects | Biomarkers | Aged | Macrocyclic Compounds - administration & dosage | Sulfonamides - administration & dosage | Uracil - analogs & derivatives | Complications and side effects | Research | Hepatitis C | Drug therapy | Liver cirrhosis | Risk factors | Hypertension | Effectiveness | Liver diseases | Ritonavir | Health risks | Viruses | Pharmacology | Bilirubin | Multivariate analysis | Ribonucleic acid--RNA | Patients | Ribavirin | Hepatitis | Patient safety | Safety | Hepatitis C virus | Genotypes | Index Medicus
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4. Full Text Manipulation of the gut–liver axis by interruption of bile acid recirculation: an option for the treatment of sclerosing cholangitis?
by Jahn, Daniel and Marzioni, Marco and Geier, Andreas
Gut, ISSN 0017-5749, 09/2018, Volume 67, Issue 9, pp. 1565 - 1567
Fuchs et al present two possible explanations which are not mutually exclusive: on the one hand, LCA and DCA are the two most potent endogenous agonists for... 
cholestatic liver diseases | bile acid | glucagen-like peptides | primary sclerosing cholangitis | GLUCAGON-LIKE PEPTIDE-1 | RECEPTOR AGONIST | GASTROENTEROLOGY & HEPATOLOGY | EXENDIN-4 | Fibroblast growth factor | Active transport | Peptides | Glucagon | Liver | Homeostasis | Intracellular signalling | Proteins | Signal transduction | Microbiota | Hepatology | Rodents | Fibroblasts | Fibroblast growth factor receptor 4 | Colon | Resins | Growth factors | Glucagon-like peptide 1 | Enzymes | Liver diseases | Secretion | Multidrug resistance | Hydroxylase | Cholangitis | Ileum | Cholesterol | Studies | Acids | Sodium | Large intestine | Intracellular | Bile | Index Medicus | Abridged Index Medicus
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5. Full Text Genetic association analysis identifies variants associated with disease progression in primary sclerosing cholangitis
by Alberts, Rudi and de Vries, Elisabeth M G and Goode, Elizabeth C and Jiang, Xiaojun and Sampaziotis, Fotis and Rombouts, Krista and Böttcher, Katrin and Folseraas, Trine and Weismüller, Tobias J and Mason, Andrew L and Wang, Weiwei and Alexander, Graeme and Alvaro, Domenico and Bergquist, Annika and Björkström, Niklas K and Beuers, Ulrich and Björnsson, Einar and Boberg, Kirsten Muri and Bowlus, Christopher L and Bragazzi, Maria C and Carbone, Marco and Chazouillères, Olivier and Cheung, Angela and Dalekos, Georgios and Eaton, John and Eksteen, Bertus and Ellinghaus, David and Färkkilä, Martti and Festen, Eleonora A M and Floreani, Annarosa and Franceschet, Irene and Gotthardt, Daniel Nils and Hirschfield, Gideon M and Hoek, Bart van and Holm, Kristian and Hohenester, Simon and Hov, Johannes Roksund and Imhann, Floris and Invernizzi, Pietro and Juran, Brian D and Lenzen, Henrike and Lieb, Wolfgang and Liu, Jimmy Z and Marschall, Hanns-Ulrich and Marzioni, Marco and Melum, Espen and Milkiewicz, Piotr and Müller, Tobias and Pares, Albert and Rupp, Christian and Rust, Christian and Sandford, Richard N and Schramm, Christoph and Schreiber, Stefan and Schrumpf, Erik and Silverberg, Mark S and Srivastava, Brijesh and Sterneck, Martina and Teufel, Andreas and Vallier, Ludovic and Verheij, Joanne and Vila, Arnau Vich and Vries, Boudewijn de and Zachou, Kalliopi and Chapman, Roger W and Manns, Michael P and Pinzani, Massimo and Rushbrook, Simon M and Lazaridis, Konstantinos N and Franke, Andre and Anderson, Carl A and Karlsen, Tom H and Ponsioen, Cyriel Y and Weersma, Rinse K and PSC Study Grp and UK PSC Consortium and International PSC Study Group, The UK PSC Consortium and Sahlgrenska akademin and Institute of Medicine, Department of Molecular and Clinical Medicine and Institutionen för medicin, avdelningen för molekylär och klinisk medicin and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Gut, ISSN 0017-5749, 08/2018, Volume 67, Issue 8, pp. 1517 - 1524
ObjectivePrimary sclerosing cholangitis (PSC) is a genetically complex, inflammatory bile duct disease of largely unknown aetiology often leading to liver... 
Primary sclerosing cholangitis | liver transplantation | genetics | RISK LOCI | ACTIVATION | MALIGNANCY | HEPATIC STELLATE CELLS | CIRRHOSIS | ULCERATIVE-COLITIS | LIVER FIBROSIS | GASTROENTEROLOGY & HEPATOLOGY | MULTIPLE | EPIDEMIOLOGY | GENOME-WIDE ASSOCIATION | Animal models | Transplants & implants | Disease | Syngeneic grafts | Association analysis | Effector cells | Genomes | Single-nucleotide polymorphism | Allografts | Etiology | Gastroenterology | Genetic analysis | Gallbladder | Antigens | Stellate cells | Liver diseases | Cholangitis | Regression analysis | Gene expression | Bile duct | Inflammatory bowel disease | Genetic variance | Hepatocytes | Quality control | Liver cirrhosis | Bile | Liver transplantation | Index Medicus | Abridged Index Medicus | Genetics | Genètica | Trasplantament hepàtic | Bilious diseases and biliousness | Hepatic transplantation | Malalties del tracte biliar | Gastroenterology and Hepatology | Gastroenterologi
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6. Full Text Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLD
by Irene, Pierantonelli and Chiara, Rychlicki and Laura, Agostinelli and Maria, Giordano Debora and Melania, Gaggini and Cristina, Fraumene and Chiara, Saponaro and Valeria, Manghina and Loris, Sartini and Eleonora, Mingarelli and Claudio, Pinto and Emma, Buzzigoli and Luciano, Trozzi and Antonio, Giordano and Marco, Marzioni and Samuele, De Minicis and Sergio, Uzzau and Saverio, Cinti and Gastalderi, Amalia and Gianluca, Svegliati-Baroni
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 12200 - 15
Non-Alcoholic Fatty Liver Disease (NAFLD) represents the most common form of chronic liver injury and can progress to cirrhosis and hepatocellular carcinoma. A... 
OXIDATIVE STRESS | HIGH-FAT DIET | INSULIN-RESISTANCE | INFLAMMASOME ACTIVATION | MULTIDISCIPLINARY SCIENCES | AKKERMANSIA-MUCINIPHILA | PPAR-GAMMA | NONALCOHOLIC STEATOHEPATITIS | HEPATIC STEATOSIS | INTESTINAL PERMEABILITY | ADIPOSE-TISSUE | Non-alcoholic Fatty Liver Disease - pathology | Intestinal Mucosa - metabolism | Dysbiosis - drug therapy | Humans | Non-alcoholic Fatty Liver Disease - etiology | Male | Bacterial Translocation - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Toll-Like Receptor 9 - immunology | Anti-Bacterial Agents - therapeutic use | Dysbiosis - immunology | Intestinal Mucosa - immunology | Disease Models, Animal | Fructose - adverse effects | Mice, Inbred C57BL | Bacterial Translocation - immunology | Toll-Like Receptor 4 - immunology | Intestinal Mucosa - microbiology | Permeability | Immunity, Innate | Diet, Western - adverse effects | Toll-Like Receptor 4 - metabolism | Disease Progression | Mice, Knockout | Phenotype | Animals | Gastrointestinal Microbiome - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Inflammasomes - immunology | Anti-Bacterial Agents - pharmacology | Mice | Gastrointestinal Microbiome - immunology | Toll-Like Receptor 9 - metabolism | Drinking water | Dysbacteriosis | Adipose tissue | Liver | Hepatocellular carcinoma | High fat diet | Lipopolysaccharides | Fatty liver | Intestine | Rodents | Antimicrobial peptides | Toll-like receptors | Bacteria | Digestive tract | Deoxyribonucleic acid--DNA | Liver diseases | Immune response | Drinking behavior | Digestive system | TLR4 protein | Fructose | TLR9 protein | Cirrhosis | Peroxisome proliferator-activated receptors | Index Medicus
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7. Full Text Expert consensus document: Cholangiocarcinoma: current knowledge and future perspectives consensus statement from the European Network for the Study of Cholangiocarcinoma (ENS-CCA)
by Banales, Jesus M and Cardinale, Vincenzo and Carpino, Guido and Marzioni, Marco and Andersen, Jesper B and Invernizzi, Pietro and Lind, Guro E and Folseraas, Trine and Forbes, Stuart J and Fouassier, Laura and Geier, Andreas and Calvisi, Diego F and Mertens, Joachim C and Trauner, Michael and Benedetti, Antonio and Maroni, Luca and Vaquero, Javier and Macias, Rocio I. R and Raggi, Chiara and Perugorria, Maria J and Gaudio, Eugenio and Boberg, Kirsten M and Marin, Jose J. G and Alvaro, Domenico
Nature Reviews Gastroenterology and Hepatology, ISSN 1759-5045, 05/2016, Volume 13, Issue 5, pp. 261 - 280
Cholangiocarcinoma (CCA) is a heterogeneous group of malignancies with features of biliary tract differentiation. CCA is the second most common primary liver... 
Liver Transplantation | Humans | Neoplasm Proteins - physiology | Intercellular Signaling Peptides and Proteins - metabolism | Tumor Microenvironment - genetics | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Bile Duct Neoplasms - genetics | Stents | Molecular Targeted Therapy - methods | Cancer-Associated Fibroblasts - physiology | Macrophages - physiology | Cholangiocarcinoma - therapy | Cytokines - metabolism | Early Detection of Cancer | Risk Factors | Cell Proliferation - physiology | Signal Transduction - genetics | Forecasting | Genetic Heterogeneity | Gene Fusion - genetics | Bile Duct Neoplasms - therapy | Cholangiocarcinoma - pathology | Drug Resistance, Neoplasm - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Epigenesis, Genetic - genetics | Cholangiocarcinoma - genetics | Bile Duct Neoplasms - pathology | Neoplasm Staging | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Index Medicus
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8. Full Text Dysbiosis contributes to fibrogenesis in the course of chronic liver injury in mice
by Minicis, Samuele and Rychlicki, Chiara and Agostinelli, Laura and Saccomanno, Stefania and Candelaresi, Cinzia and Trozzi, Luciano and Mingarelli, Eleonora and Facinelli, Bruna and Magi, Gloria and Palmieri, Claudio and Marzioni, Marco and Benedetti, Antonio and Svegliati‐Baroni, Gianluca
Hepatology, ISSN 0270-9139, 05/2014, Volume 59, Issue 5, pp. 1738 - 1749
Nonalcoholic fatty liver disease (NAFLD) may lead to hepatic fibrosis. Dietary habits affect gut microbiota composition, whereas endotoxins produced by... 
METABOLIC SYNDROME | OBESITY | ACTIVATION | CORE GUT MICROBIOME | INSULIN-RESISTANCE | DISEASE | FATTY LIVER | STEATOHEPATITIS | GASTROENTEROLOGY & HEPATOLOGY | BACTERIAL TRANSLOCATION | INNATE IMMUNITY | Inflammasomes - physiology | Microbiota - physiology | Animals | Diet, High-Fat | Mice, Inbred C57BL | Liver Cirrhosis, Experimental - etiology | Gastrointestinal Tract - microbiology | Male | Mice | Carbon Tetrachloride - toxicity | Dysbiosis - complications | Bacterial Translocation | Bacteria | Gram-negative bacteria | Liver | Rodents | Hepatology | Bile | Index Medicus
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9. Full Text EASL Clinical Practice Guidelines: The diagnosis and management of patients with primary biliary cholangitis
by Hirschfield, Gideon M and Beuers, Ulrich and Corpechot, Christophe and Invernizzi, Pietro and Jones, David and Marzioni, Marco and Schramm, Christoph and European Assoc Study Liver and European Association for the Study of the Liver and European Association for the Study of the Liver. Electronic address: easloffice@easloffice.eu
Journal of Hepatology, ISSN 0168-8278, 2017, Volume 67, Issue 1, pp. 145 - 172
Summary Primary biliary cholangitis (PBC) is a chronic inflammatory autoimmune cholestatic liver disease, which when untreated will culminate in end-stage... 
Gastroenterology and Hepatology | Care pathway | Liver | Cholestasis | Guidelines | LONG-TERM PROGNOSIS | RANDOMIZED CONTROLLED-TRIALS | ACID-TREATED PATIENTS | LIVER FIBROSIS PROGRESSION | URSODEOXYCHOLIC-ACID | ORAL NALTREXONE TREATMENT | QUALITY-OF-LIFE | PLACEBO-CONTROLLED TRIAL | GASTROENTEROLOGY & HEPATOLOGY | EARLY BIOCHEMICAL RESPONSE | PRIMARY SCLEROSING CHOLANGITIS | Liver Transplantation | Prognosis | Humans | Cholangitis - complications | Female | Male | Cholangitis - diagnosis | Cholangitis - therapy | Practice Guidelines as Topic | Ursodeoxycholic Acid - therapeutic use | Liver cirrhosis | Care and treatment | Diagnosis | Studies | Training | Cirrhosis | Budesonide | Hepatitis | Liver diseases | Cholangitis | Gallbladder diseases | Derivatives | Patients | Ursodeoxycholic acid | Index Medicus
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