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Nature Medicine, ISSN 1078-8956, 10/2018, Volume 24, Issue 10, pp. 1559 - 1567
Visual inspection of histopathology slides is one of the main methods used by pathologists to assess the stage, type and subtype of lung tumors. Adenocarcinoma... 
TARGETED THERAPY | INACTIVATION | MEDICINE, RESEARCH & EXPERIMENTAL | NEURAL-NETWORKS | PROMISE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADENOCARCINOMAS | EGFR MUTATION | KRAS | LKB1 | SOCIETY | CELL BIOLOGY | Carcinoma, Non-Small-Cell Lung - pathology | Adenocarcinoma - pathology | Neoplasm Proteins - classification | Carcinoma, Non-Small-Cell Lung - genetics | Adenocarcinoma - classification | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Non-Small-Cell Lung - diagnosis | Mutation - genetics | Deep Learning | Carcinoma, Squamous Cell - diagnosis | Adenocarcinoma - diagnosis | Carcinoma, Non-Small-Cell Lung - classification | Carcinoma, Squamous Cell - classification | Adenocarcinoma - genetics | Neoplasm Proteins - genetics | Neural Networks (Computer) | Usage | Gene mutations | Machine learning | Development and progression | Genetic aspects | Lung cancer, Non-small cell | Histology, Pathological | Identification and classification | Methods | Adenocarcinoma | Paraffins | Visual discrimination learning | Squamous cell carcinoma | Medical imaging | Epidermal growth factor receptors | p53 Protein | Lung | Lung cancer | Artificial neural networks | Inspection | Genomes | Paraffin | Tissues | Histopathology | Neural networks | Predictions | Mathematical models | Mutation | Cancer | Tumors | Image classification | Index Medicus
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2018, Volume 1741, pp. 31 - 51
In this chapter, we describe the use of Illumina Infinium HD Assay in conjunction with Illumina's EPIC Methylation 8-sample array platform to obtain... 
Array | GBM | Profiling | FFPE | DNA | Multiplex | DNA methylation | Tumor | Illumina | Subgroup classification | DNA Methylation | Glioblastoma - genetics | Oligonucleotide Array Sequence Analysis | Humans | Gene Dosage | High-Throughput Nucleotide Sequencing | Genome, Human | Genomics - methods | Nucleic Acid Amplification Techniques | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2015, Volume 520, Issue 7547, pp. 363 - 367
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 16, pp. 4476 - 4481
Inhibition of the vascular endothelial growth factor (VEGF) pathway has failed to improve overall survival of patients with glioblastoma (GBM). We previously... 
Anti-angiogenic therapy | Macrophage polarization | Tumor microenvironment | Microglia reprogramming | Anti-tumor immunity | anti-angiogenic therapy | BEVACIZUMAB | MULTIDISCIPLINARY SCIENCES | macrophage polarization | RANDOMIZED-TRIAL | ANGIOPOIETIN-2 | CANCER | THERAPY | microglia reprogramming | TIE2-EXPRESSING MONOCYTES | BLOOD-VESSELS | TUMOR-ASSOCIATED MACROPHAGES | tumor microenvironment | NORMALIZATION | anti-tumor immunity | ENDOTHELIAL GROWTH-FACTOR | Macrophages - pathology | Antibodies, Bispecific - pharmacology | Vesicular Transport Proteins - metabolism | Humans | Neoplasm Proteins - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Neoplasm Proteins - metabolism | Neoplasms, Experimental - pathology | Xenograft Model Antitumor Assays | Macrophages - metabolism | Vesicular Transport Proteins - antagonists & inhibitors | Animals | Antibodies, Neoplasm - pharmacology | Ribonuclease, Pancreatic - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Mice | Neoplasms, Experimental - metabolism | Glioblastoma - drug therapy | Ribonuclease, Pancreatic - antagonists & inhibitors | Neoplasms, Experimental - drug therapy | Viral antibodies | Care and treatment | Antibodies | Physiological aspects | Models | Macrophages | Vascular endothelial growth factor | Health aspects | Glioblastoma multiforme | Genotype & phenotype | Survival analysis | Protein expression | Cells | Tumors | Cancer | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2013, Volume 110, Issue 47, pp. 19059 - 19064
Antiangiogenic therapy has shown clear activity and improved survival benefit for certain tumor types. However, an incomplete understanding of the mechanisms... 
Oxygen | Perfusion | Glioblastoma | Biological markers | Radiotherapy | Blood | Tissue oxygenation | Tumors | Cancer | Blood plasma | VEGFR2 | VASCULAR NORMALIZATION | BEVACIZUMAB | MULTIDISCIPLINARY SCIENCES | brain tumor | RADIATION | personalized treatment | TEMOZOLOMIDE | GROWTH-FACTOR RECEPTOR | ANTIANGIOGENIC THERAPY | AMPLIFICATION | MICROENVIRONMENT | TYROSINE KINASE INHIBITOR | Prospective Studies | Humans | Brain Neoplasms - pathology | Quinazolines | Glioblastoma - radiotherapy | Brain Neoplasms - metabolism | Oxygen - metabolism | DNA Repair Enzymes - metabolism | Dacarbazine - analogs & derivatives | Polymerase Chain Reaction | Statistics, Nonparametric | Glioblastoma - metabolism | Brain Neoplasms - radiotherapy | Tumor Suppressor Proteins - metabolism | DNA Modification Methylases - metabolism | Enzyme-Linked Immunosorbent Assay | Angiogenesis Inhibitors - pharmacology | Treatment Outcome | Brain Neoplasms - drug therapy | Receptor Protein-Tyrosine Kinases - metabolism | Biomarkers, Tumor - blood | Magnetic Resonance Imaging | Glioblastoma - pathology | Protein Kinase Inhibitors - pharmacology | Glioblastoma - drug therapy | Cancer patients | Chemotherapy | Patient outcomes | Physiological aspects | Dosage and administration | Drug therapy | Glioblastoma multiforme | Methods | Cediranib | Survival analysis | Cell growth | Biomarkers | Clinical trials | Kinases | Patients | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 16, pp. 4470 - 4475
Glioblastomas (GBMs) rapidly become refractory to anti-VEGF therapies. We previously demonstrated that ectopic overexpression of angiopoietin-2 (Ang-2)... 
Anti-angiogenic therapy | Colony-stimulating factor 1 | Tumor immunity | Tumor microenvironment | Macrophage | anti-angiogenic therapy | ANGIOPOIETIN-2 FUNCTIONS | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | macrophage | BEVACIZUMAB PLUS IRINOTECAN | RANDOMIZED-TRIAL | ANTIBODY | tumor immunity | KINASE INHIBITOR | ANTIANGIOGENIC THERAPY | PHASE-II TRIAL | colony-stimulating factor 1 | tumor microenvironment | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Macrophages - pathology | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Neovascularization, Pathologic - pathology | Neoplasms, Experimental - pathology | Macrophages - metabolism | Animals | Antibodies, Neoplasm - pharmacology | Receptors, Vascular Endothelial Growth Factor - metabolism | Ribonuclease, Pancreatic - metabolism | Neovascularization, Pathologic - drug therapy | Glioblastoma - pathology | Cell Line, Tumor | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Glioblastoma - metabolism | Mice | Neovascularization, Pathologic - metabolism | Neoplasms, Experimental - metabolism | Glioblastoma - drug therapy | Quinazolines - pharmacology | Ribonuclease, Pancreatic - antagonists & inhibitors | Neoplasms, Experimental - drug therapy | Drug Screening Assays, Antitumor | Survival analysis | Gene expression | Kinases | Cells | Tumors | Cancer | Index Medicus | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2015, Volume 112, Issue 46, pp. 14325 - 14330
Journal Article
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 04/2015, Volume 107, Issue 4, p. 1
Background: Matrix metalloproteinase (MMP) 14 may mediate tumor progression through vascular and immune-modulatory effects. Methods: Orthotopic murine breast... 
VASCULAR NORMALIZATION | INVASION | CANCER CELLS | ANGIOGENESIS | ONCOLOGY | MATRIX-METALLOPROTEINASE INHIBITORS | DRUG-DELIVERY | NITRIC-OXIDE SYNTHASE | TUMOR-GROWTH | EXPRESSION | BETA | Up-Regulation | Immunoglobulin G - blood | Breast Neoplasms - immunology | Humans | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | Smad3 Protein - metabolism | Breast Neoplasms - metabolism | Nitric Oxide Synthase Type II - antagonists & inhibitors | Dose Fractionation | Female | Antineoplastic Agents - pharmacology | Matrix Metalloproteinase 14 - drug effects | Antibodies, Monoclonal - pharmacology | Enzyme Inhibitors - pharmacology | Smad2 Protein - metabolism | Nitric Oxide Synthase Type II - drug effects | Breast Neoplasms - blood supply | Matrix Metalloproteinase 14 - metabolism | Breast Neoplasms - drug therapy | Enzyme Inhibitors - therapeutic use | Macrophages - enzymology | Gene Expression Regulation, Enzymologic | Breast Neoplasms - radiotherapy | Phenotype | Animals | Signal Transduction - drug effects | Mammary Neoplasms, Experimental | Cell Line, Tumor | Macrophages - drug effects | Amidines - pharmacology | Mice | Benzylamines - pharmacology | Transforming Growth Factor beta - metabolism | Nitric Oxide Synthase Type II - metabolism | Enzymes | Genotype & phenotype | Immunoglobulins | Immunology | Radiation therapy | Gene expression | Tumors | Index Medicus
Journal Article
Journal Article