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1. Full Text Genetics of Inflammatory Bowel Diseases
by McGovern, Dermot P.B and Kugathasan, Subra and Cho, Judy H
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2015, Volume 149, Issue 5, pp. 1163 - 1176.e2
Gastroenterology and Hepatology | Epigenetics | Crohn's Disease | Autophagy | Ulcerative Colitis | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Pharmacogenetics - methods | Gene Expression - drug effects | Humans | Genetic Testing - methods | Gene Expression Regulation - drug effects | Exome - drug effects | Epigenomics - methods | Genetic Variation - drug effects | Alleles | Inflammatory Bowel Diseases - genetics | Genome-Wide Association Study - methods | Direct-To-Consumer Screening and Testing - methods | Genetic Therapy - methods | RNA, Untranslated - drug effects | Quantitative genetics | Epigenetic inheritance | Medical colleges | Pharmacogenetics | Gastrointestinal diseases | Medical genetics | African Americans | Genetic aspects | Gene expression | Index Medicus | Abridged Index Medicus | Crohn's disease
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2. Full Text Interactions Between Commensal Fungi and the C-Type Lectin Receptor Dectin-1 Influence Colitis
by Iliyan D. Iliev and Vincent A. Funari and Kent D. Taylor and Quoclinh Nguyen and Christopher N. Reyes and Samuel P. Strom and Jordan Brown and Courtney A. Becker and Phillip R. Fleshner and Marla Dubinsky and Jerome I. Rotter and Hanlin L. Wang and Dermot P. B. McGovern and Gordon D. Brown and David M. Underhill
Science (American Association for the Advancement of Science), ISSN 0036-8075, 6/2012, Volume 336, Issue 6086, pp. 1314 - 1317
Fungi | Haplotypes | Microorganisms | Inflammatory bowel diseases | Commensals | REPORTS | Bacteria | Mice | Colitis | Intestinal diseases | Ulcerative colitis | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Dextran Sulfate | Fungi - classification | Candida tropicalis - isolation & purification | Humans | Immunity, Mucosal | Fungi - isolation & purification | Colon - immunology | Colony Count, Microbial | Candida tropicalis - physiology | Lectins, C-Type - genetics | Colitis, Ulcerative - immunology | Intestines - immunology | Fungi - immunology | Lectins, C-Type - metabolism | Intestinal Mucosa - immunology | Fungi - physiology | Female | Lectins, C-Type - deficiency | Disease Susceptibility | Mice, Inbred C57BL | Candida tropicalis - pathogenicity | Antibodies, Fungal - blood | Intestinal Mucosa - microbiology | Immunity, Innate | Animals | Intestines - microbiology | Colitis, Ulcerative - microbiology | Colitis, Ulcerative - chemically induced | Candida tropicalis - immunology | Colon - microbiology | Polymorphism, Single Nucleotide | Metagenome | Microbiota (Symbiotic organisms) | Causes of | Physiological aspects | Lectins | Development and progression | Genetic aspects | Inflammatory bowel disease | Digestive system | Immune system | Index Medicus | Pathology | Health | Deletion | Mammals | Inhabitants
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3. Full Text Gene-microbiota interactions contribute to the pathogenesis of inflammatory bowel disease
by Chu, Hiutung and Khosravi, Arya and Kusumawardhani, Indah P and Kwon, Alice H. K and Vasconcelos, Anilton C and Cunha, Larissa D and Mayer, Anne E and Shen, Yue and Wu, Wei-Li and Kambal, Amal and Targan, Stephan R and Xavier, Ramnik J and Ernst, Peter B and Green, Douglas R and McGovern, Dermot P.B and Virgin, Herbert W and Mazmanian, Sarkis K
Science (American Association for the Advancement of Science), ISSN 0036-8075, 05/2016, Volume 352, Issue 6289, pp. 1116 - 1120
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Autophagy-Related Proteins | Dendritic Cells - immunology | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Male | Nod2 Signaling Adaptor Protein - genetics | Gene-Environment Interaction | T-Lymphocytes, Regulatory - immunology | Autophagy - immunology | Extracellular Vesicles - immunology | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - genetics | Adult | Female | Bacteroides fragilis - immunology | Genetic Predisposition to Disease | Mice, Inbred C57BL | Intestinal Mucosa - microbiology | Polymorphism, Genetic | Carrier Proteins - genetics | Animals | Inflammatory Bowel Diseases - microbiology | Aged | Mice | Gastrointestinal Microbiome - immunology | Genome, Human | Development and progression | Microbiota (Symbiotic organisms) | Gastrointestinal diseases | Genes | Inflammatory bowel diseases | Host-parasite relationships | Genetic aspects | Gene expression | Observations | Health aspects | Inflammatory bowel disease | Microbiology | Genetics | Index Medicus | Human | Membranes | Microorganisms | Vesicles | Government regulations | Pathways | Risk
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4. Full Text Disease-Specific Alterations in the Enteric Virome in Inflammatory Bowel Disease
by Norman, Jason M and Handley, Scott A and Baldridge, Megan T and Droit, Lindsay and Liu, Catherine Y and Keller, Brian C and Kambal, Amal and Monaco, Cynthia L and Zhao, Guoyan and Fleshner, Phillip and Stappenbeck, Thaddeus S and McGovern, Dermot P.B and Keshavarzian, Ali and Mutlu, Ece A and Sauk, Jenny and Gevers, Dirk and Xavier, Ramnik J and Wang, David and Parkes, Miles and Virgin, Herbert W
Cell (Cambridge), ISSN 0092-8674, 01/2015, Volume 160, Issue 3, pp. 447 - 460
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Colitis, Ulcerative - virology | Humans | Crohn Disease - virology | Dysbiosis - microbiology | Case-Control Studies | Dysbiosis - virology | Feces - microbiology | Feces - virology | Bacteria - classification | Caudovirales - genetics | Colitis, Ulcerative - therapy | Caudovirales - isolation & purification | Crohn Disease - microbiology | Colitis, Ulcerative - pathology | Microviridae - isolation & purification | Bacteria - genetics | Dysbiosis - therapy | Crohn Disease - therapy | Bacteria - isolation & purification | Colitis, Ulcerative - microbiology | Crohn Disease - pathology | Dysbiosis - pathology | Microviridae - genetics | Metagenome | Cohort Studies | Analysis | Ulcerative colitis | Index Medicus
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5. Full Text Reprograming of gut microbiome energy metabolism by the FUT2 Crohn's disease risk polymorphism
by Tong, Maomeng and McHardy, Ian and Ruegger, Paul and Goudarzi, Maryam and Kashyap, Purna C and Haritunians, Talin and Li, Xiaoxiao and Graeber, Thomas G and Schwager, Emma and Huttenhower, Curtis and Fornace, Albert J and Sonnenburg, Justin L and McGovern, Dermot P.B and Borneman, James and Braun, Jonathan
The ISME Journal, ISSN 1751-7362, 11/2014, Volume 8, Issue 11, pp. 2193 - 2206
multi'omic analysis | microbial ecology | glycan foraging | intestinal microbiome | Environmental Sciences & Ecology | Ecology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Crohn Disease - genetics | Fucosyltransferases - genetics | Bacteria - metabolism | Humans | Mice, Inbred C57BL | Middle Aged | Risk Factors | Metabolome | Male | Intestinal Mucosa - microbiology | Mice, Knockout | Polymorphism, Genetic | Microbiota | Animals | Adult | Female | Aged | Mice | Energy Metabolism - genetics | Metagenome | Proteome - metabolism | Index Medicus | Original
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6. Full Text CX3CR1+, mononuclear phagocytes control immunity to intestinal fungi
by Leonardi, Irina and Li, Xin and Semon, Alexa and Li, Dalin and Doron, Itai and Putzel, Gregory and Bar, Agnieszka and Prieto, Daniel and Rescigno, Maria and McGovern, Dermot P.B and Pla, Jesus and Iliev, Iliyan D
Science (American Association for the Advancement of Science), ISSN 0036-8075, 01/2018, Volume 359, Issue 6372, pp. 232 - 236
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Crohn Disease - genetics | Antibodies, Fungal - biosynthesis | Immunoglobulin G - blood | Dendritic Cells - immunology | Humans | Immunity, Mucosal | Gastrointestinal Microbiome - physiology | Mutation, Missense | Candida albicans - growth & development | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | Immunoglobulin G - biosynthesis | CX3C Chemokine Receptor 1 - genetics | Colitis - drug therapy | Mycobiome - physiology | Antibodies, Fungal - blood | Candida albicans - immunology | Phagocytes - immunology | Crohn Disease - immunology | CX3C Chemokine Receptor 1 - analysis | Mycobiome - immunology | Animals | Colitis - microbiology | Intestines - microbiology | Phagocytes - microbiology | Th17 Cells - immunology | Mice | Gastrointestinal Microbiome - immunology | Inflammatory bowel diseases | Fungicides | Communities | Antibodies | Homeostasis | CX3CR1 protein | Leukocytes (mononuclear) | Immunity | Inflammatory diseases | Fungi | Phagocytes | Receptors | Microbiota | Missense mutation | Clonal deletion | Intestine | Bacteria | Digestive tract | Immune system | Immune response | Digestive system | Medical treatment | Crohn's disease | Patients | Ablation | Inflammatory bowel disease | Syk protein | Colitis | Index Medicus
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7. Full Text A common missense variant in NUDT15 confers susceptibility to thiopurine-induced leukopenia
by Yang, Suk-Kyun and Hong, Myunghee and Baek, Jiwon and Choi, Hyunchul and Zhao, Wanting and Jung, Yusun and Haritunians, Talin and Ye, Byong Duk and Kim, Kyung-Jo and Park, Sang Hyoung and Park, Soo-Kyung and Yang, Dong-Hoon and Dubinsky, Marla and Lee, Inchul and McGovern, Dermot P.B and Liu, Jianjun and Song, Kyuyoung
Nature genetics, ISSN 1061-4036, 08/2014, Volume 46, Issue 9, pp. 1017 - 1020
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Crohn Disease - genetics | Genetic Predisposition to Disease | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Methyltransferases - genetics | Leukopenia - genetics | Male | Mutation, Missense | Case-Control Studies | Adolescent | Leukopenia - chemically induced | Leukopenia - enzymology | Adult | Female | Aged | Polymorphism, Single Nucleotide | Child | Purines | Genetic variation | Leucopenia | Genetic aspects | Properties | Identification and classification | Risk factors | Population | Data analysis | Genomes | Biomedical research | Mutation | Crohns disease | Index Medicus
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8. Full Text Malassezia Is Associated with Crohn’s Disease and Exacerbates Colitis in Mouse Models
by Limon, Jose J and Tang, Jie and Li, Dalin and Wolf, Andrea J and Michelsen, Kathrin S and Funari, Vince and Gargus, Matthew and Nguyen, Christopher and Sharma, Purnima and Maymi, Viviana I and Iliev, Iliyan D and Skalski, Joseph H and Brown, Jordan and Landers, Carol and Borneman, James and Braun, Jonathan and Targan, Stephan R and McGovern, Dermot P.B and Underhill, David M
Cell host & microbe, ISSN 1931-3128, 03/2019, Volume 25, Issue 3, pp. 377 - 388.e6
Crohn disease | mycobiome | Malassezia | c-type lectin | CARD9 | Parasitology | Life Sciences & Biomedicine | Microbiology | Science & Technology | Virology | CARD Signaling Adaptor Proteins - metabolism | Malassezia - isolation & purification | Animals | Colitis - physiopathology | Cytokines - metabolism | Crohn Disease - pathology | Gastrointestinal Tract - microbiology | Colitis - pathology | Crohn Disease - physiopathology | Mice | Malassezia - growth & development | Disease Models, Animal | Microbiota (Symbiotic organisms) | Analysis | Antifungal agents | Lectins | Crohn's disease | Colitis | Genetic polymorphisms | Index Medicus
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