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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2011, Volume 127, Issue 3, pp. 661 - 667
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 121, Issue 4, pp. 872 - 877.e9
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 121, Issue 4, pp. 940 - 946.e3
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 121, Issue 5, pp. 1294 - 1295
  [...]this study has only 50% power to detect the primary effect size of 1.8. [...]the likelihood of this replicating the primary observation was as likely to... 
Allergy and Immunology | ATOPIC-DERMATITIS | ALLERGY | RISK | PHENOTYPES | MUTATIONS | BIRTH | IMMUNOLOGY | Genetic Predisposition to Disease | Mutation | Humans | Asthma - genetics | Hypersensitivity, Immediate - genetics | Intermediate Filament Proteins - genetics | Studies | Genotype & phenotype | Eczema
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 132, Issue 5, pp. 1121 - 1129
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 135, Issue 1, pp. 164 - 170.e4
Background History and severity of atopic dermatitis (AD) are risk factors for peanut allergy. Recent evidence suggests that children can become sensitized to... 
Allergy and Immunology | peanut sensitization | peanut allergy | Atopic dermatitis | environmental peanut exposure | dust | PROTEIN | FILAGGRIN | OF-FUNCTION VARIANTS | FOOD ALLERGY | TOLERANCE | IMMUNOLOGY | RISK-FACTOR | GENE | ALLERGY | INFANTS | MUTATIONS | CONSUMPTION | Antigens, Plant - analysis | Dust - analysis | Allergens - analysis | Skin Tests | Dermatitis, Atopic - epidemiology | Humans | Antigens, Plant - immunology | Dust - immunology | Infant | Male | Plant Proteins - immunology | Plant Proteins - analysis | Environmental Exposure - analysis | Peanut Hypersensitivity - immunology | Housing | Environmental Exposure - adverse effects | Peanut Hypersensitivity - diagnosis | Allergens - immunology | Peanut Hypersensitivity - epidemiology | Arachis - immunology | Dermatitis, Atopic - immunology | Female | Odds Ratio | Allergens | Medical colleges | Skin | Inflammation | Dermatitis | Peanut allergy | Studies | Food allergies | Mutation | Peanuts | Allergies | Children & youth | Index Medicus | Abridged Index Medicus | LR, Logistic regression | AD, Atopic dermatitis | PA, Peanut allergy | SPT, Skin prick test | FLG, Filaggrin | sIgE, Specific IgE | Atopic Dermatitis and Skin Disease | CoFAR, Consortium of Food Allergy Research | IQR, Interquartile range | OR, Odds ratio | LLQ, Lower limit of quantitation | EPE, Environmental peanut exposure
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 1, pp. 166 - 172
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2014, Volume 134, Issue 4, pp. 867 - 875.e1
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2012, Volume 129, Issue 4, pp. 1031 - 1039.e1
Background Filaggrin (FLG) mutations result in reduced stratum corneum (SC) natural moisturizing factor (NMF) components and consequent increased SC pH.... 
Allergy and Immunology | skin barrier | transepidermal water loss | confocal Raman spectroscopy | Flgft/Flgft (FlgdelAPfal) mice | Atopic dermatitis | IL-1 | IL-18 | FLG gene mutations | eczema | filaggrin | natural moisturizing factor | UNCONVENTIONAL PROTEIN SECRETION | INTERLEUKIN-1 RECEPTOR ANTAGONIST | TERM CULTURE | IMMUNOLOGY | PERMEABILITY BARRIER HOMEOSTASIS | HUMAN KERATINOCYTES | IN-VITRO | ALLERGY | Flg(ft)/Flg(ft)(Flg(delAPfal)) mice | EPIDERMIS | IL-1-BETA | Interleukin-1 - genetics | Interleukin-1 - metabolism | Dermatitis, Atopic - genetics | Cytokines - metabolism | Skin - metabolism | Humans | Mice, Inbred C57BL | Child, Preschool | Genotype | Infant | Male | Mice, Knockout | Animals | Adolescent | Alleles | Intermediate Filament Proteins - genetics | Female | Mice | Mutation | Child | Dermatitis, Atopic - metabolism | Intermediate Filament Proteins - metabolism | Hydrogen-Ion Concentration | Medical colleges | Cytokines | Proteases | Genes | Respiratory agents | Genetic aspects | Skin | Inflammation | Universities and colleges | Dermatitis | Proteins | Enzymes | Genotype & phenotype | Hypotheses | Families & family life | Values | Homeostasis | Animal models | Stratum corneum | Interleukin 1 receptor antagonist | Proteinase | Keratinocytes | mRNA | Filaggrin | Raman spectroscopy | pH effects | Tails | Interleukin 18 | Polymerase chain reaction | Water loss | Primers | Interleukin 1 | Interleukin 8 | FLG | SC, Stratum corneum | Flgft | Patient homozygous for null alleles (ie, 2 null alleles) | AD, Atopic dermatitis | FLG, Filaggrin gene | Homozygote wild-type (ie, 0 null alleles) | TEWL, Transepidermal water loss | Heterozygote null allele | Flgft (FlgdelAPfal) mice | IL-1RA, IL-1 receptor antagonist | wild-type (ie, 1 null allele) | NMF, Natural moisturizing factor | ADFLG, AD with FLG mutations | Atopic Dermatitis and Skin Disease | NESS, Nottingham Eczema Severity Score | ADNON-FLG, AD without FLG mutations
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 5, pp. 1268 - 1276