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Journal Article
Nature medicine, ISSN 1078-8956, 12/2010, Volume 16, Issue 12, pp. 1400 - 1408
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 11/2008, Volume 19, Issue 11, pp. 4875 - 4887
Members of the Snail family of transcription factors have been shown to induce epithelial-mesenchymal transition (EMT), a fundamental mechanism of... 
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, p. e42913
Hemangiomas are tumors formed by hyper-proliferation of vascular endothelial cells. This is caused by elevated vascular endothelial growth factor (VEGF)... 
HIF-1 | RESPONSES | THERAPY | PATHWAY | BIOLOGY | HYPOXIA | CANCER | MTOR | Autocrine Communication | Cell Line | Gene Expression | Cell Proliferation | Antibiotics, Antineoplastic - pharmacology | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | TOR Serine-Threonine Kinases - metabolism | Endothelial Cells - metabolism | Humans | Gene Expression Regulation, Neoplastic | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Hypoxia-Inducible Factor 1 - metabolism | Sirolimus - pharmacology | Hemangioma - metabolism | Hypoxia-Inducible Factor 1 - genetics | RNA Interference | Signal Transduction - drug effects | Phosphorylation - drug effects | Vascular Endothelial Growth Factor A - pharmacology | Hemangioma - genetics | Endothelial Cells - drug effects | Proteins | Hemangioma | RNA | Rapamycin | Genetic translation | Vascular endothelial growth factor | Endothelium | TOR protein | Cell proliferation | Pediatrics | Hypoxia-inducible factor 1 | Incorporation | mRNA | Kinases | Medical schools | Angiogenesis | Physiology | Autocrine signalling | Localization | Immunoglobulins | Developmental biology | Therapeutic applications | siRNA | Dentistry | Endothelial cells | Medicine | Overexpression | Hypoxia | Protein expression | Cancer | Tumors
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2008, Volume 14, Issue 11, pp. 1236 - 1246
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 04/2006, Volume 17, Issue 4, pp. 1871 - 1879
Transforming growth factor beta 1 (TGF-β1) has been shown to induce epithelial-mesenchymal transition (EMT) during various stages of embryogenesis and... 
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 08/2012, Volume 27, Issue 8, p. 1619
  Heterotopic ossification (HO) is a process by which bone forms in soft tissues, in response to injury, inflammation, or genetic disease. This usually occurs... 
Journal Article
Journal Article
Matrix Biology, ISSN 0945-053X, 2010, Volume 29, Issue 3, pp. 161 - 165
Collagen I has been shown to promote epithelial–mesenchymal transition (EMT), a critical process of embryonic development and disease progression. However,... 
Collagen I | NF-κB | ILK | Epithelial–mesenchymal transition | EMT | LEF-1 | Epithelial-mesenchymal transition | CELLS | INTEGRIN-LINKED KINASE | TGF-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PANCREATIC-CANCER | TRANSCRIPTION | CELL BIOLOGY | NF-kappa B | SIGNALING PATHWAY | CADHERIN GENE-EXPRESSION | CARCINOMA | PROGRESSION | SNAIL | epithelial-mesenchymal transition | collagen I
Journal Article