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Molecular Cancer, ISSN 1476-4598, 05/2017, Volume 16, Issue 1, pp. 93 - 14
Background: The MET receptor tyrosine kinase represents a promising target in cancer. PIK3CA activating mutations are common in several tumor types and can... 
Head and neck cancer | PI3K pathway | PIK3CA mutations | MET receptor tyrosine kinase | Resistance mechanisms | HEAD | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | SENSITIVITY | GROWTH-FACTOR RECEPTOR | PHOSPHATIDYLINOSITOL 3-KINASES | ONCOLOGY | RAS MUTATIONS | C-MET | RESISTANCE | NECK | EXPRESSION | Neoplasms - metabolism | Proto-Oncogene Proteins c-met - metabolism | Cell Survival - drug effects | NIH 3T3 Cells | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-met - genetics | Phosphatidylinositol 3-Kinases - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Neoplasms - genetics | Tumor Burden - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Mutation | Neoplasms - pathology | Disease Models, Animal | Tyrosine | Care and treatment | Gene mutations | Genetic aspects | Research | Gene expression | Health aspects | Cell proliferation | Genomics | Lung cancer | Colorectal cancer | Activation | Metastasis | Kinases | Cancer therapies | Neoplasms | Anticancer properties | c-Met protein | Cell activation | Cell growth | Antitumor agents | Epidermal growth factor | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Wound healing | Colonies | Mortality | Forming | Breast cancer | 1-Phosphatidylinositol 3-kinase | Chemotherapy | Inhibitors | Plasmids | Cell death | Point mutation | Ligands | Healing | Aberration | Tumors | Cancer | Apoptosis
Journal Article
Molecular Cancer, ISSN 1476-4598, 02/2018, Volume 17, Issue 1, pp. 1 - 12
Journal Article
Molecular cancer, 02/2018, Volume 17, Issue 1, p. 27
Tumor metabolism is a thrilling discipline that focuses on mechanisms used by cancer cells to earn crucial building blocks and energy to preserve growth and... 
Neoplasms - metabolism | Animals | Signal Transduction - drug effects | Protein-Tyrosine Kinases - metabolism | Humans | Lipid Metabolism - drug effects | Glucose - metabolism | Enzyme Inhibitors - therapeutic use | Glycolysis - drug effects | Energy Metabolism - drug effects
Journal Article
Oncogene, ISSN 0950-9232, 07/2018, Volume 37, Issue 30, pp. 4181 - 4196
Journal Article
Cytometry Part A, ISSN 1552-4922, 04/2018, Volume 93, Issue 4, pp. 406 - 410
To optimize OMIP‐045, cancer cell lines and a HNSCC tumor were processed into a single cell suspension and stained with the protocol outlined. Cells were then... 
cancer | mass cytometry | phenotyping | head and neck squamous cell carcinoma | RELEVANCE | BIOCHEMICAL RESEARCH METHODS | RECEPTOR | CELL BIOLOGY | Squamous cell carcinoma | Cytometry | Head | Cell lines | Head and neck cancer | Tumor cell lines | Cancer | Tumors
Journal Article
Molecular Cancer Research, ISSN 1541-7786, 12/2018, Volume 16, Issue 12, pp. 1912 - 1926
Metastases and tumor recurrence have a major prognostic impact in head and neck squamous cell carcinoma (HNSCC); however, cellular models that comprehensively... 
RATES | TO-MESENCHYMAL TRANSITION | ONCOLOGY | LANDSCAPE | PATHWAY | MUTATIONS | OUTCOMES | CARCINOMA | EXPRESSION | DISCOVERY | CELL BIOLOGY
Journal Article
FRONTIERS IN ONCOLOGY, ISSN 2234-943X, 10/2019, Volume 9
In human cells, three closely related RAS genes, termed HRAS, KRAS, and NRAS, encode four highly homologous proteins. RAS proteins are small GTPases involved... 
ONCOGENIC K-RAS | GTP | ACTIVATION | rare codons | N-RAS | RAS profile | MOUSE | PROLIFERATION | RAS mutations | KRAS MUTATIONS | H-RAS | GENE | ONCOLOGY | GROWTH | treatment responses | RAS-mutated cancers | GDP binding | ASSOCIATION | RAS-related omics | RAS signaling | Research | Genetic transcription | Gene mutations | Cancer cells | GTP/GDP binding
Journal Article
Journal Article
Molecular Oncology, ISSN 1574-7891, 08/2015, Volume 9, Issue 7, pp. 1434 - 1446
Journal Article
BMC BIOINFORMATICS, ISSN 1471-2105, 11/2019, Volume 20, Issue 1, pp. 563 - 563
Journal Article