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1. Full Text Mutations in SQSTM1 encoding p62 in amyotrophic lateral sclerosis: genetics and neuropathology
by Teyssou, Elisa and Takeda, Takahiro and Lebon, Vincent and Boillée, Séverine and Doukouré, Brahima and Bataillon, Guillaume and Sazdovitch, Véronique and Cazeneuve, Cécile and Meininger, Vincent and LeGuern, Eric and Salachas, François and Seilhean, Danielle and Millecamps, Stéphanie
Acta Neuropathologica, ISSN 0001-6322, 4/2013, Volume 125, Issue 4, pp. 511 - 522
Mutations in SQSTM1 encoding the sequestosome 1/p62 protein have recently been identified in familial and sporadic cases of amyotrophic lateral sclerosis... 
Pathology | Neurosciences | Medicine & Public Health | Neuropathology | Motor neuron disease | Paget’s disease | FTLD–ALS | p62 | Familial and sporadic ALS | FTLD-ALS | Paget's disease | DOMAIN | UBIQUITIN-BINDING | BINDING PROTEIN P62 | TDP-43 PATHOLOGY | ALS PATIENTS | PATHOLOGY | FRONTOTEMPORAL LOBAR DEGENERATION | C9ORF72 | NEUROSCIENCES | CLINICAL NEUROLOGY | PAGETS-DISEASE | BONE | HEXANUCLEOTIDE REPEAT | Sequestosome-1 Protein | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Ubiquitin - metabolism | Male | Ubiquitin - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - metabolism | Amyotrophic Lateral Sclerosis - pathology | Adaptor Proteins, Signal Transducing - genetics | Aged, 80 and over | Brain - pathology | Adult | Female | Heterozygote | Aged | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Ubiquitin | Amyotrophic lateral sclerosis | Genetic aspects | Gene mutations | Analysis | Genes | Index Medicus
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2. Full Text Chromosome 9p21 in sporadic amyotrophic lateral sclerosis in the UK and seven other countries: a genome-wide association study
by Shatunov, Aleksey, PhD and Mok, Kin, MSc and Newhouse, Stephen, PhD and Weale, Michael E, PhD and Smith, Bradley, PhD and Vance, Caroline, PhD and Johnson, Lauren, PhD and Veldink, Jan H, MD and van Es, Michael A, MD and van den Berg, Leonard H, Prof and Robberecht, Wim, Prof and Van Damme, Philip, MD and Hardiman, Orla, Prof and Farmer, Anne E, Prof and Lewis, Cathryn M, Prof and Butler, Amy W, PhD and Abel, Olubunmi, MSc and Andersen, Peter M, Prof and Fogh, Isabella, PhD and Silani, Vincenzo, Prof and Chiò, Adriano, MD and Traynor, Bryan J, MD and Melki, Judith, Prof and Meininger, Vincent, Prof and Landers, John E, PhD and McGuffin, Peter, Prof and Glass, Jonathan D, MD and Pall, Hardev, MD and Leigh, P Nigel, Prof and Hardy, John, Prof and Brown, Robert H, Prof and Powell, John F, DPhil and Orrell, Richard W, MD and Morrison, Karen E, Prof and Shaw, Pamela J, Prof and Shaw, Christopher E, Prof and Al-Chalabi, Ammar, Prof and Medicinska fakulteten and Institutionen för farmakologi och klinisk neurovetenskap and Neurologi and Umeå universitet
Lancet Neurology, The, ISSN 1474-4422, 2010, Volume 9, Issue 10, pp. 986 - 994
Summary Background Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease of motor neurons that results in progressive weakness and death from... 
Neurology | POPULATION | DEMENTIA | GENE | SUSCEPTIBILITY | ALS | LOCI | FRONTOTEMPORAL LOBAR DEGENERATION | COPY NUMBER VARIATION | LINKAGE | IRISH | CLINICAL NEUROLOGY | Frontotemporal Dementia - genetics | United States - epidemiology | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Europe - epidemiology | Case-Control Studies | Chromosomes, Human, Pair 9 - genetics | Internationality | Aged, 80 and over | Polymorphism, Single Nucleotide - genetics | Aged | Genome-Wide Association Study - methods | Frontotemporal Dementia - epidemiology | United Kingdom - epidemiology | Amyotrophic Lateral Sclerosis - epidemiology | Cohort Studies | Genetic research | Amyotrophic lateral sclerosis | Analysis | Genomics | Risk factors | Index Medicus | Fast track
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3. Full Text TARDBP mutations in individuals with sporadic and familial amyotrophic lateral sclerosis
by Meininger, Vincent and Spiegelman, Dan and Salachas, Francois and Dupre, Nicolas and Dion, Patrick and Velde, Christine Vande and Pradat, Pierre-Francois and Kabashi, Edor and Bouchard, Jean-Pierre and Lacomblez, Lucette and Valdmanis, Paul N and McConkey, Brendan J and Camu, William and Pochigaeva, Ksenia and Rouleau, Guy A
Nature Genetics, ISSN 1061-4036, 05/2008, Volume 40, Issue 5, pp. 572 - 574
Recently, TDP-43 was identified as a key component of ubiquitinated aggregates in amyotrophic lateral sclerosis (ALS), an adult-onset neurological disorder... 
BODIES | PROTEIN | FRONTOTEMPORAL LOBAR DEGENERATION | TDP-43 | DISEASE | GENETICS & HEREDITY | Amino Acid Sequence | Protein Biosynthesis | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Molecular Sequence Data | Male | Mutation, Missense | DNA-Binding Proteins - genetics | DNA-Binding Proteins - metabolism | Pedigree | Base Sequence | Amyotrophic Lateral Sclerosis - metabolism | Adult | Female | Aged | Motor neurons | Amyotrophic lateral sclerosis | Genetic aspects | Gene mutations | Health aspects | Risk factors | Proteins | Medical research | Data analysis | Genetics | Cystic fibrosis | Amino acids | Mutation | Chromosomes | Neurological disorders | Muscular dystrophy | Index Medicus
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4. Full Text Early diaphragm pacing in patients with amyotrophic lateral sclerosis (RespiStimALS): a randomised controlled triple-blind trial
by Gonzalez-Bermejo, Jésus, Dr and Morélot-Panzini, Capucine, MD and Tanguy, Marie-Laure, PharmD and Meininger, Vincent, Prof and Pradat, Pierre-François, MD and Lenglet, Timothée, MD and Bruneteau, Gaëlle, MD and Forestier, Nadine Le, MD and Couratier, Philippe, Prof and Guy, Nathalie, MD and Desnuelle, Claude, Prof and Prigent, Hélène, MD and Perrin, Christophe, MD and Attali, Valérie, MD and Fargeot, Catherine, PharmD and Nierat, Marie-Cécile, PhD and Royer, Catherine, MD and Ménégaux, Fabrice, Prof and Salachas, François, MD and Similowski, Thomas, Prof
Lancet Neurology, The, ISSN 1474-4422, 2016, Volume 15, Issue 12, pp. 1217 - 1227
Summary Background Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder associated with respiratory muscle weakness and respiratory failure.... 
Neurology | SURVIVAL | STIMULATION | PHRENIC-NERVE | MECHANICAL VENTILATION | ALS | ELECTRODES | MUSCLE | COHORT | EXPERIENCE | NONINVASIVE VENTILATION | CLINICAL NEUROLOGY | Double-Blind Method | Amyotrophic Lateral Sclerosis - therapy | Humans | Middle Aged | Male | Laparoscopy | Respiration Disorders | Respiration, Artificial | Amyotrophic Lateral Sclerosis - complications | Early Termination of Clinical Trials | Electric Stimulation Therapy - methods | Respiratory Insufficiency - prevention & control | Electric Stimulation Therapy - adverse effects | Phrenic Nerve | Female | Aged | Diaphragm - innervation | Diaphragm - physiopathology | Amyotrophic lateral sclerosis | Care and treatment | Nervous system diseases | Neurophysiology | Palliative care | Ventilation | Ostomy | Surgery | Mortality | Index Medicus
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5. Full Text Mutations in the profilin 1 gene cause familial amyotrophic lateral sclerosis
by Wu, Chi-Hong and Fallini, Claudia and Ticozzi, Nicola and Keagle, Pamela J and Sapp, Peter C and Piotrowska, Katarzyna and Lowe, Patrick and Koppers, Max and McKenna-Yasek, Diane and Baron, Desiree M and Kost, Jason E and Gonzalez-Perez, Paloma and Fox, Andrew D and Adams, Jenni and Taroni, Franco and Tiloca, Cinzia and Leclerc, Ashley Lyn and Chafe, Shawn C and Mangroo, Dev and Moore, Melissa J and Zitzewitz, Jill A and Xu, Zuo-Shang and Van Den Berg, Leonard H and Glass, Jonathan D and Siciliano, Gabriele and Cirulli, Elizabeth T and Goldstein, David B and Salachas, Francois and Meininger, Vincent and Rossoll, Wilfried and Ratti, Antonia and Gellera, Cinzia and Bosco, Daryl A and Bassell, Gary J and Silani, Vincenzo and Drory, Vivian E and Brown Jr, Robert H and Landers, John E
Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7412, pp. 499 - 503
Amyotrophic lateral sclerosis (ALS) is a late-onset neurodegenerative disorder resulting from motor neuron death. Approximately 10% of cases are familial... 
PROTEIN | NEURITE OUTGROWTH | ACTIN | SPASTIC PARAPLEGIA | INCREASE | MULTIDISCIPLINARY SCIENCES | EXPANSION | ALS | MUTANT SOD1 | HEXANUCLEOTIDE REPEAT | C9ORF72 | Humans | Actins - metabolism | Amyotrophic Lateral Sclerosis - diagnosis | Molecular Sequence Data | Male | Jews - genetics | Ubiquitination | Profilins - genetics | Motor Neurons - cytology | Female | Amino Acid Sequence | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease - genetics | Amyotrophic Lateral Sclerosis - genetics | Cells, Cultured | Mutant Proteins - genetics | Axons - metabolism | Models, Molecular | Mutant Proteins - metabolism | Mutation - genetics | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Exome - genetics | Animals | Growth Cones - metabolism | Pedigree | Axons - pathology | Amyotrophic Lateral Sclerosis - metabolism | Protein Conformation | High-Throughput Nucleotide Sequencing | Mice | Profilins - metabolism | Amyotrophic lateral sclerosis | Genetic aspects | Research | Gene mutations | Health aspects | Risk factors | Proteins | Genomes | Polypeptides | Mutation | Pathogenesis | Genes | Index Medicus
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6. Amyotrophic lateral sclerosis
by Brown, Robert H., 1952 and Meininger, Vincent and Swash, Michael
2000, ISBN 1853174211, xvi, 479
Amyotrophic lateral sclerosis
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7. Full Text Low-Dose Mineralocorticoid Receptor Blockade Prevents Western Diet–Induced Arterial Stiffening in Female Mice
by DeMarco, Vincent G and Habibi, Javad and Jia, Guanghong and Aroor, Annayya R and Ramirez-Perez, Francisco I and Martinez-Lemus, Luis A and Bender, Shawn B and Garro, Mona and Hayden, Melvin R and Sun, Zhe and Meininger, Gerald A and Manrique, Camila and Whaley-Connell, Adam and Sowers, James R
Hypertension, ISSN 0194-911X, 07/2015, Volume 66, Issue 1, pp. 99 - 107
Women are especially predisposed to development of arterial stiffening secondary to obesity because of consumption of excessive calories. Enhanced activation... 
vascular stiffness | spironolactone | aldosterone | obesity | AORTIC STIFFNESS | ENDOTHELIAL FUNCTION | PRIMARY ALDOSTERONISM | VASCULAR-DISEASE | DIASTOLIC DYSFUNCTION | DIABETES-MELLITUS | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SERUM ALDOSTERONE | Pulse Wave Analysis | Receptors, Mineralocorticoid - physiology | Dose-Response Relationship, Drug | Spironolactone - pharmacology | Mineralocorticoid Receptor Antagonists - pharmacology | Female | Aorta - physiopathology | Nitric Oxide Synthase Type III - metabolism | Myocytes, Smooth Muscle - drug effects | Spironolactone - therapeutic use | Arteriosclerosis - etiology | Aorta - drug effects | Femoral Artery - physiopathology | Mice, Inbred C57BL | Obesity - physiopathology | Femoral Artery - drug effects | Diet, Western - adverse effects | Mineralocorticoid Receptor Antagonists - therapeutic use | Arteriosclerosis - prevention & control | Animals | Vascular Stiffness - drug effects | Inflammation - prevention & control | Mice | Oxidative Stress - drug effects | Vasodilation - drug effects | Endothelial Cells - drug effects | Index Medicus | Aldosterone
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8. Full Text Genome-wide association analyses identify new risk variants and the genetic architecture of amyotrophic lateral sclerosis
by van Rheenen, Wouter and Shatunov, Aleksey and Dekker, Annelot M and McLaughlin, Russell L and Diekstra, Frank P and Pulit, Sara L and van der Spek, Rick A. A and Võsa, Urmo and de Jong, Simone and Robinson, Matthew R and Yang, Jian and Fogh, Isabella and van Doormaal, Perry Tc and Tazelaar, Gijs H. P and Koppers, Max and Blokhuis, Anna M and Sproviero, William and Jones, Ashley R and Kenna, Kevin P and van Eijk, Kristel R and Harschnitz, Oliver and Schellevis, Raymond D and Brands, William J and Medic, Jelena and Menelaou, Anoniki and Vajda, Alice and Ticozzi, Nicola and Lin, Kuang and Rogelj, Boris and Vrabec, Katarina and Ravnik-Glavač, Metka and Koritnik, Blaž and Zidar, Janez and Leonardis, Lea and Grošelj, Leja Dolenc and Millecamps, Stéphanie and Salachas, François and Meininger, Vincent and de Carvalho, Mamede and Pinto, Susana and Mora, Jesus S and Rojas-García, Ricardo and Polak, Meraida and Chanan, Siddharthan and Colville, Shuna and Swingler, Robert and Morrison, Karen E and Shaw, Pamela J and Hardy, John and Orrell, Richard W and Pittman, Alan and Sidle, Katie and Fratta, Pietro and Malaspina, Anea and Topp, Simon and Petri, Susanne and Abdulla, Susanne and pper, Carsten and Sendtner, Michael and Meyer, Thomas and Ophoff, Roel A and Staats, Kim A and Wiedau-Pazos, Martina and Lomen-Hoerth, Catherine and van Deerlin, Vivianna M and Trojanowski, John Q and Elman, Lauren and McCluskey, Leo and Basak, A. Nazli and Tunca, Ceren and Hamzeiy, Hamid and Parman, Yesim and Meitinger, Thomas and Lichtner, Peter and Radivojkov-Blagojevic, Milena and Anes, Christian R and Maurel, Cindy and Bensimon, Gilbert and Landwehrmeyer, Bernhard and Brice, Alexis and Payan, Christine A. M and Saker-Delye, Safaa and Dürr, Alexana and Wood, Nicholas W and Tittmann, Lukas and Lieb, Wolfgang and Franke, Ane and Rietschel, Marcella and Cichon, Sven and Nöthen, Markus M and Amouyel, Philippe and Tzourio, Christophe and Dartigues, Jean-François and Uitterlinden, Ane G and Rivadeneira, Fernando and Estrada, Karol and Hofman, Albert and Curtis, Charles and Blauw, Hylke M and van der Kooi, Anneke J and ... and PARALS Registry and FALS Sequencing Consortium and SLALOM Grp and SLAGEN Consortium and NNIPPS Study Grp and SLAP Registry and SLALOM Group and NNIPPS Study Group and Medicinska fakulteten and Institutionen för farmakologi och klinisk neurovetenskap and Klinisk neurovetenskap and Umeå universitet
Nature genetics, ISSN 1061-4036, 2016, Volume 48, Issue 9, pp. 1043 - 1048
To elucidate the genetic architecture of amyotrophic lateral sclerosis (ALS) and find associated loci, we assembled a custom imputation reference panel from... 
PATHWAYS | POPULATION | COMPLEX | FRONTOTEMPORAL DEMENTIA | SUSCEPTIBILITY | SEQUENCE | GENETICS & HEREDITY | ALS | PITFALLS | HEXANUCLEOTIDE REPEAT | EFFICIENT | Proteins - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Munc18 Proteins - genetics | Amyotrophic Lateral Sclerosis - genetics | Humans | Netherlands - epidemiology | Mutation - genetics | Myelin Proteins - genetics | Case-Control Studies | Amyotrophic Lateral Sclerosis - epidemiology | Cohort Studies | Studies | Haplotypes | Consortia | Amyotrophic lateral sclerosis | Genomes | Quality control | Index Medicus | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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9. Full Text Endothelial Mineralocorticoid Receptor Mediates Diet-Induced Aortic Stiffness in Females
by Jia, Guanghong and Habibi, Javad and Aroor, Annayya R and Martinez-Lemus, Luis A and DeMarco, Vincent G and Ramirez-Perez, Francisco I and Sun, Zhe and Hayden, Melvin R and Meininger, Gerald A and Mueller, Katelee Barrett and Jaffe, Iris Z and Sowers, James R
Circulation Research, ISSN 0009-7330, 03/2016, Volume 118, Issue 6, pp. 935 - 943
RATIONALE:Enhanced activation of the mineralocorticoid receptors (MRs) in cardiovascular tissues increases oxidative stress, maladaptive immune responses, and... 
macrophages | nitric oxide | vascular stiffness | inflammation | Western diet | CAVEOLIN-1-DEFICIENT MICE | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIAC STRUCTURE | ANGIOTENSIN-II | VASCULAR ENDOTHELIUM | BLOOD-PRESSURE | DIASTOLIC DYSFUNCTION | INSULIN-RESISTANCE | SHEAR-STRESS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | INDUCED OBESITY | Aorta - pathology | Animals | Vascular Stiffness - physiology | Female | Mice | Aorta - physiology | Diet, Western - adverse effects | Endothelium, Vascular - physiology | Mice, Knockout | Receptors, Mineralocorticoid - physiology | Index Medicus | endothelial cell mineralocorticoid receptor | stiffness | endothelial dysfunction | Vascular stiffness
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10. Full Text Safety and efficacy of ozanezumab in patients with amyotrophic lateral sclerosis: a randomised, double-blind, placebo-controlled, phase 2 trial
by Meininger, Vincent, MD and Genge, Angela, MD and van den Berg, Leonard H, Prof and Robberecht, Wim, Prof and Ludolph, Albert, Prof and Chio, Adriano, Prof and Kim, Seung H, Prof and Leigh, P Nigel, Prof and Kiernan, Matthew C, Prof and Shefner, Jeremy M, Prof and Desnuelle, Claude, Prof and Morrison, Karen E, Prof and Petri, Susanne, Prof and Boswell, Diane, BSc and Temple, Jane, PhD and Mohindra, Rajat, MD and Davies, Matt, MSc and Bullman, Jonathan, BSc and Rees, Paul, PhD and Lavrov, Arseniy, Dr and NOG112264 Study Grp and NOG112264 Study Group
Lancet Neurology, The, ISSN 1474-4422, 2017, Volume 16, Issue 3, pp. 208 - 216
Summary Background Neurite outgrowth inhibitor A (Nogo-A) is thought to have a role in the pathophysiology of amyotrophic lateral sclerosis (ALS). A monoclonal... 
Neurology | DIAGNOSIS | RILUZOLE SERUM CONCENTRATIONS | NEURITE OUTGROWTH INHIBITOR | MARKER | DISEASE | ALS | NOGO-A | DENERVATION | EXPRESSION | CLINICAL NEUROLOGY | Antibodies, Monoclonal, Humanized - therapeutic use | Nogo Proteins - immunology | Age Factors | Double-Blind Method | Humans | Middle Aged | Male | Treatment Outcome | International Cooperation | Amyotrophic Lateral Sclerosis - drug therapy | Amyotrophic Lateral Sclerosis - mortality | Young Adult | Adolescent | Survival Analysis | Aged, 80 and over | Electrocardiography | Adult | Female | Aged | Immunologic Factors - therapeutic use | Medical colleges | Neurosciences | Hospitals | Analysis | Clinical trials | Monoclonal antibodies | Amyotrophic lateral sclerosis | Musculoskeletal system | Rodents | Index Medicus | Life Sciences | Human health and pathology | Neurons and Cognition
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11. Full Text Efficacy and Safety of Canagliflozin in Patients with Type 2 Diabetes and Stage 3 Nephropathy
by Yamout, Hala and Perkovic, Vlado and Davies, Melanie and Woo, Vincent and de Zeeuw, Dick and Mayer, Cristiana and Vijapurkar, Ujjwala and Kline, Irina and Usiskin, Keith and Meininger, Gary and Bakris, George
American Journal of Nephrology, ISSN 0250-8095, 08/2014, Volume 40, Issue 1, pp. 64 - 74
Background/Aims: Some sodium glucose co-transporter 2 (SGLT2) inhibitors are approved for the treatment of patients with type 2 diabetes mellitus (T2DM) with... 
Original Report: Patient-Oriented, Translational Research | Hypertension | Nephropathy | Diabetes | Kidney | METFORMIN | GLYCEMIC CONTROL | PLACEBO | MONOTHERAPY | SULFONYLUREA | MANAGEMENT | SITAGLIPTIN | MELLITUS | DIET | UROLOGY & NEPHROLOGY | Blood Pressure | Thiophenes - therapeutic use | Body Weight | Renal Insufficiency, Chronic - etiology | Diabetic Nephropathies - etiology | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Male | Diabetes Mellitus, Type 2 - metabolism | Renal Insufficiency, Chronic - metabolism | Clinical Trials, Phase III as Topic | Female | Glucosides - therapeutic use | Diabetes Mellitus, Type 2 - complications | Hypoglycemic Agents - therapeutic use | Severity of Illness Index | Glomerular Filtration Rate | Diabetic Nephropathies - metabolism | Treatment Outcome | Randomized Controlled Trials as Topic | Canagliflozin | Hypertension - complications | Aged | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Index Medicus
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