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International Journal of Molecular Sciences, ISSN 1661-6596, 06/2016, Volume 17, Issue 6, pp. 990 - 990
Pulmonary hypertension (PH) is defined by a mean pulmonary arterial pressure over 25 mmHg at rest and is diagnosed by right heart catheterization. Among the... 
Oxidative stress | DNA-damage response | Inflammation | DNA damage | Pulmonary hypertension | Animals | Oxidative Stress | DNA Repair | Humans | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | DNA Damage | Genes, Mitochondrial | Hypertension, Pulmonary - pathology | Pathogenesis | DNA repair | inflammation | pulmonary hypertension | oxidative stress
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2011, Volume 301, Issue 5, pp. H1798 - H1809
Paulin R, Meloche J, Jacob MH, Bisserier M, Courboulin A, Bonnet S. Dehydroepiandrosterone inhibits the Src/STAT3 constitutive activation in pulmonary arterial... 
Therapies | Proliferation | Remodeling | Signal transducer and activator of transcription 3 | Apoptosis | remodeling | therapies | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | proliferation | CA2+ CHANNELS | apoptosis | signal transducer and activator of transcription 3 | ONCOGENIC TRANSFORMATION | GROWTH | STAT3 PHOSPHORYLATION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | EXPRESSION | MORPHOGENETIC PROTEIN-RECEPTOR | K+ CHANNELS | C-SRC | Antihypertensive Agents - pharmacology | Apoptosis - drug effects | Calcium - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Middle Aged | Monocrotaline | Proto-Oncogene Proteins c-pim-1 - metabolism | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Membrane Potential, Mitochondrial - drug effects | Hypertension, Pulmonary - chemically induced | Muscle, Smooth - drug effects | src-Family Kinases - metabolism | Adult | Female | Hypertension, Pulmonary - drug therapy | Inhibitor of Apoptosis Proteins - metabolism | Myocytes, Smooth Muscle - drug effects | Real-Time Polymerase Chain Reaction | Hypertension, Pulmonary - enzymology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | In Situ Nick-End Labeling | Vasodilator Agents - pharmacology | Dehydroepiandrosterone - pharmacology | Myocytes, Smooth Muscle - enzymology | NFATC Transcription Factors - metabolism | Cells, Cultured | Rats | Familial Primary Pulmonary Hypertension | Pulmonary Artery - drug effects | Rats, Sprague-Dawley | Blotting, Western | Bone Morphogenetic Protein Receptors, Type II - metabolism | Pulmonary Artery - enzymology | Animals | Signal Transduction - drug effects | Muscle, Smooth - physiopathology | Cell Proliferation - drug effects | Enzyme Activation | Hypertension, Pulmonary - pathology | Muscle, Smooth - pathology | Pulmonary Artery - pathology | Muscle, Smooth - enzymology | Dehydroepiandrosterone | Phenotype | Physiological aspects | Development and progression | Genetic aspects | Research | T cells | Health aspects | Pulmonary hypertension
Journal Article
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 03/2011, Volume 208, Issue 3, pp. 535 - 548
Journal Article
Current Vascular Pharmacology, ISSN 1570-1611, 07/2015, Volume 13, Issue 3, pp. 331 - 340
Journal Article
Circulation, ISSN 0009-7322, 02/2014, Volume 129, Issue 7, pp. 786 - 797
Journal Article
Handbook of Experimental Pharmacology, ISSN 0171-2004, 2013, Volume 218, pp. 437 - 476
Conference Proceeding
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2015, Volume 309, Issue 6, pp. C363 - C372
Journal Article
Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue Suppl_3 Suppl 3, pp. A16419 - A16419
IntroductionPulmonary arterial hypertension (PAH) is a devastating vasculopathy. Recent progress in understanding its pathogenesis led to an increase in... 
Journal Article