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Science, ISSN 0036-8075, 11/2005, Volume 310, Issue 5749, pp. 791 - 793
Bullock et al detail how neuroscientists, after a century, are rethinking the Neuron Doctrine--the fundamental principle of neuroscience. The proposition,... 
Axons | Brain | Neuroscience | Neurons | Gap junctions | Neuroglia | Ribosomes | Nervous system | Dendrites | Perspectives | Synapses | ACTION-POTENTIALS | DENDRITES | CELLS | SYNAPSES | TRIGGERS | MULTIDISCIPLINARY SCIENCES | DYNAMICS | BRAIN | Neurosciences | Research | Analysis | Neurology | Science history
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2016, Volume 113, Issue 25, p. E3558
  A major hallmark of oxidative DNA damage after stroke is the induction of apurinic/apyrimidinic (AP) sites and strand breaks. To mitigate cell loss after... 
Brain | Neurology | Oxidative stress | Stroke | Rodents | DNA damage
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 03/2013, Volume 110, Issue 12, p. 4738
  The phosphoinositide signaling system is a crucial regulator of neural development, cell survival, and plasticity. Phosphatase and tensin homolog deleted on... 
Autism | Protein synthesis | Morphology | Mutation | Behavior | Kinases | Chromosomes
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 06/2016, Volume 113, Issue 25, pp. E3558 - E3558
A major hallmark of oxidative DNA damage after stroke is the induction of apurinic/apyrimidinic (AP) sites and strand breaks. To mitigate cell loss after... 
Journal Article
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 04/2012, Volume 109, Issue 16, pp. E962 - E971
Dysregulation of the transcriptional repressor element-1 silencing transcription factor (REST)/neuron-restrictive silencer factor is important in a broad range... 
alpha -Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptors | Histone deacetylase | Stroke | Transcription factors | Diabetes mellitus | Cognitive ability | Capsaicin receptors | Promoters | epigenetics | Gene silencing | Ischemia | Repressors | MeCP2 protein | Neurodegeneration | Heart diseases | Hippocampus | Injuries | Cancer
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 02/2012, Volume 109, Issue 9, pp. 573 - 582
Trafficking and turnover of transmitter receptors required to maintain and modify the strength of chemical synapses have been characterized extensively. In... 
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 11/2011, Volume 108, Issue 44, pp. 18102 - 18107
Persistent protein synthesis inhibition (PSI) is a robust predictor of eventual neuronal death following cerebral ischemia. We thus tested the hypothesis that... 
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 12/2010, Volume 107, Issue 52, pp. 22659 - 22664
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2013, Volume 33, Issue 11, pp. 4741 - 4753
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2010, Volume 107, Issue 7, pp. 3204 - 3209
Inducible DNA repair via the base-excision repair pathway is an important prosurvival mechanism activated in response to oxidative DNA damage. Elevated levels... 
Transcriptional regulatory elements | Ischemia | Genetic vectors | Neurons | Cell death | DNA damage | DNA | Brain ischemia | Mathematical vectors | DNA repair | Delayed neurodegeneration | Oxidative stress | Activating transcription factor 2 | cAMP response-element binding | activating transcription factor 2 | CYCLASE-ACTIVATING POLYPEPTIDE | BASE-EXCISION-REPAIR | MULTIDISCIPLINARY SCIENCES | TRANSIENT FOCAL ISCHEMIA | DNA-DAMAGE | HIPPOCAMPAL-NEURONS | CELL-DEATH | delayed neurodegeneration | RAT-BRAIN | ADENYLATE-CYCLASE | SUPEROXIDE-DISMUTASE | oxidative stress | EARLY DECREASE | Phosphorylation | Humans | Oxidative Stress - physiology | DNA Repair - physiology | Gene Expression Regulation - physiology | Rats | Brain Ischemia - prevention & control | DNA-(Apurinic or Apyrimidinic Site) Lyase - physiology | Hippocampus - cytology | Rats, Sprague-Dawley | Activating Transcription Factor 2 - metabolism | Hippocampus - metabolism | Animals | Analysis of Variance | Chromatin Immunoprecipitation | DNA-(Apurinic or Apyrimidinic Site) Lyase - metabolism | Luciferases | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Signal Transduction - physiology | Electrophoretic Mobility Shift Assay | Apoptosis - physiology | Prevention | Nucleases | Cerebral ischemia | Transcription factors | Cyclic adenosine monophosphate response element-binding protein | Properties | Observations | Risk factors | Biological Sciences
Journal Article
Nature Neuroscience, ISSN 1097-6256, 04/2001, Volume 4, Issue 4, pp. 382 - 390
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 03/2008, Volume 105, Issue 12, pp. 4892 - 4897
Transient forebrain or global ischemia induces delayed neuronal death in vulnerable CA1 pyramidal cells with many features of apoptosis. A brief period of... 
Journal Article