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Frontiers in Oncology, ISSN 2234-943X, 2013, Volume 3, pp. 38 - 38
Current drug development in oncology is non-selective as it typically focuses on pathways essential for the survival of all dividing cells. The unique... 
Dichloroacetate | Hypoxia-inducible factor 1a | Mitochondria | Tumor metabolism | Pyruvate dehydrogenase kinase | Apoptosis resistance | Apoptosis Resistance | Hypoxia-Inducible Factor 1α | Pyruvate Dehydrogenase Kinase | tumor metabolism
Journal Article
Circulation Research, ISSN 0009-7330, 08/2017, Volume 121, Issue 4, pp. 323 - 325
Journal Article
Circulation Research, ISSN 0009-7330, 06/2018, Volume 122, Issue 12, pp. 1635 - 1637
Journal Article
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 11/2013, Volume 91, Issue 11, pp. 1315 - 1327
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e90965 - e90965
Bronchopulmonary dysplasia (BPD), the chronic lung disease of prematurity, remains a major health problem. BPD is characterized by impaired alveolar... 
Bronchopulmonary Dysplasia - drug therapy | Lung Injury - pathology | Reactive Oxygen Species - metabolism | Humans | Bronchopulmonary Dysplasia - pathology | Oxygen - adverse effects | Prodrugs - chemistry | Organothiophosphorus Compounds - chemistry | Organothiophosphorus Compounds - pharmacology | Protective Agents - pharmacology | Hyperoxia - metabolism | Pulmonary Alveoli - metabolism | Lung Injury - metabolism | Pulmonary Alveoli - drug effects | Lung Injury - prevention & control | Lung - metabolism | Infant, Newborn | Disease Models, Animal | Animals, Newborn | Hydrogen Sulfide - pharmacology | Lung - pathology | Pulmonary Alveoli - pathology | Endothelial Cells - metabolism | Morpholines - pharmacology | Rats | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Rats, Sprague-Dawley | Hyperoxia - chemically induced | Morpholines - chemistry | Hyperoxia - pathology | Reactive Oxygen Species - antagonists & inhibitors | Animals | Bronchopulmonary Dysplasia - metabolism | Lung - drug effects | Hyperoxia - drug therapy | Endothelial Cells - pathology | Lung Injury - chemically induced | Prodrugs - pharmacology | Endothelial Cells - drug effects | Sulfide | Heart | Cell proliferation | Neonates | Reactive oxygen species | Epithelial cells | Lung | Smooth muscle | Kinases | Vasodilation | Hydrogen sulfide | Biological effects | Mitochondria | Rodents | Biocompatibility | Carbon monoxide | Alveoli | Chemical synthesis | Hydrogen ion concentration | Hypertension | Medical research | Dysplasia | Network formation | Lung diseases | Muscles | Pulmonary artery | Endothelial cells | Nitric oxide | Hyperoxia | In vivo methods and tests | Ventricle | Viability | Hypertrophy | Veins & arteries | Index Medicus
Journal Article
Translational Cancer Research, ISSN 2218-676X, 03/2019, Volume 8, Issue S2, pp. S156 - S161
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2007, Volume 104, Issue 27, pp. 11418 - 11423
In pulmonary arterial hypertension (PAH), antiapoptotic, proliferative, and inflammatory diatheses converge to create an obstructive vasculopathy. A selective... 
T lymphocytes | Bonnets | Mitochondria | Cell growth | Cyclosporins | Hypoxia | Leukocytes | Down regulation | Renovations | Apoptosis | channel | POTASSIUM CHANNELS | mitochondria | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIAL-FUNCTION | CARDIAC MYOCYTES | RATS | apoptosis | PREVENTS | K+ channel | SMOOTH-MUSCLE | GENE | IN-VIVO | EXPRESSION | Mitochondrial Size - drug effects | Apoptosis - drug effects | Humans | NFATC Transcription Factors - physiology | Male | Hypertension, Pulmonary - chemically induced | Hypertension, Pulmonary - therapy | Pulmonary Artery - metabolism | Kv1.5 Potassium Channel - genetics | Lung - metabolism | Mitochondrial Size - physiology | Monocrotaline - administration & dosage | Cell Line | Gene Targeting | Lung - pathology | Rats | Hypertension, Pulmonary - metabolism | Random Allocation | Rats, Sprague-Dawley | Animals | Cyclosporine - administration & dosage | Kv1.5 Potassium Channel - antagonists & inhibitors | Calcium Channels - biosynthesis | Kv1.5 Potassium Channel - biosynthesis | Oligopeptides - administration & dosage | Apoptosis - physiology | Lung - blood supply | Hypertension, Pulmonary - pathology | NFATC Transcription Factors - antagonists & inhibitors | Pulmonary Artery - pathology | NFATC Transcription Factors - genetics | Care and treatment | Usage | Therapeutics | Smooth muscle | Genetic aspects | Research | T cells | Health aspects | Pulmonary hypertension | Homeopathy | Materia medica and therapeutics | Hypertension | Enzymes | Lungs | Cells | Index Medicus | Biological Sciences
Journal Article
Circulation, ISSN 0009-7322, 05/2002, Volume 105, Issue 20, pp. 2398 - 2403
Journal Article
Chest, ISSN 0012-3692, 10/2012, Volume 142, Issue 4, pp. 816 - 820
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A17922 - A17922
IntroductionTraditional risk factors for aortic stenosis (AS) include age, male sex, hypertension, smoking, and dyslipidemia, while inflammation and increased... 
Journal Article
Circulation, ISSN 0009-7322, 11/2016, Volume 134, Issue Suppl_1 Suppl 1, pp. A17957 - A17957
IntroductionPulmonary arterial hypertension (PAH) is a proliferative vascular disease characterized by mitochondrial suppression in the pulmonary artery and... 
Journal Article
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