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BMC Bioinformatics, ISSN 1471-2105, 05/2010, Volume 11, Issue 1, pp. 228 - 228
Background: Recently, supervised learning methods have been exploited to reconstruct gene regulatory networks from gene expression data. The reconstruction of... 
BIOTECHNOLOGY & APPLIED MICROBIOLOGY | BIOCHEMICAL RESEARCH METHODS | MATHEMATICAL & COMPUTATIONAL BIOLOGY | Databases, Genetic | Data Mining | Gene Expression Profiling - methods | Gene Regulatory Networks - genetics | Artificial Intelligence | Usage | Research | Genetic regulation | Computational biology | Genetic algorithms | Index Medicus
Journal Article
Science, ISSN 0036-8075, 9/2012, Volume 337, Issue 6099, pp. 1231 - 1235
The brain tumor glioblastoma multiforme (GBM) is among the most lethal forms of human cancer. Here, we report that a small subset of GBMs (3.1%; 3 of 97 tumors... 
Exons | Neurons | Genes | REPORTS | Stem cells | Aneuploidy | Chromosomes | Cells | Tumors | Daughter cells | Cancer | ANEUPLOIDY | SELECTIVE INHIBITOR | POTENT | MULTIDISCIPLINARY SCIENCES | CANCER | RECEPTOR TYROSINE KINASE | DISCOVERY | CHROMOSOMAL INSTABILITY | FAMILY | Microtubule-Associated Proteins - chemistry | Neoplasm Transplantation | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - chemistry | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Mitosis | Humans | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - antagonists & inhibitors | Fetal Proteins - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Brain Neoplasms - metabolism | Oncogene Proteins, Fusion - chemistry | Spindle Apparatus - metabolism | Glioblastoma - genetics | Oncogene Fusion | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Nuclear Proteins - genetics | Chromosomal Instability | Pyrazoles - pharmacology | Protein Structure, Tertiary | Receptor, Fibroblast Growth Factor, Type 3 - genetics | Enzyme Inhibitors - pharmacology | Brain Neoplasms - genetics | Nuclear Proteins - metabolism | Pyrimidines - pharmacology | Nuclear Proteins - chemistry | Piperazines - pharmacology | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Xenograft Model Antitumor Assays | Animals | Cell Transformation, Neoplastic | Oncogene Proteins, Fusion - genetics | Fetal Proteins - genetics | Mice | Receptor, Fibroblast Growth Factor, Type 1 - chemistry | Fetal Proteins - chemistry | Physiological aspects | Development and progression | Fibroblast growth factors | Genetic aspects | Research | Health aspects | Glioblastoma multiforme | Proteins | Kinases | Brain cancer | Genomics | Pharmaceutical sciences | Index Medicus
Journal Article
Image and Vision Computing, ISSN 0262-8856, 2007, Volume 25, Issue 6, pp. 792 - 804
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, pp. e30427 - e30427
Aberrant activation of PI3K/AKT signalling represents one of the most common molecular alterations in lung cancer, though the relative contribution of the... 
KRAS MUTATIONS | PIK3CA GENE | PATHWAY | COLORECTAL-CANCER | THYROID-TUMORS | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC TARGET | HIGH-FREQUENCY | PHOSPHORYLATED AKT | CARCINOMA | PTEN EXPRESSION | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Cell Proliferation | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Lung Neoplasms - pathology | Male | Phosphatidylinositol 3-Kinases - metabolism | Neoplasm Proteins - metabolism | DNA Mutational Analysis | Base Sequence | Aged, 80 and over | Adult | Female | Neoplasm Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung Neoplasms - enzymology | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | PTEN Phosphohydrolase - metabolism | In Situ Hybridization, Fluorescence | Proto-Oncogene Proteins - genetics | Phosphoserine - metabolism | Epithelial Cells - enzymology | Cell Line, Tumor | Aged | Carcinoma, Non-Small-Cell Lung - enzymology | Enzyme Activation | Cell Transformation, Neoplastic - pathology | Immunohistochemistry | Inositol | RNA | Gene mutations | Analysis | Genes | DNA binding proteins | Nucleotide sequencing | B cells | Lung cancer, Non-small cell | DNA sequencing | Cell proliferation | Transcription factors | Phosphorylation | Laboratories | Epithelial cells | Copy number | Lung | Lung cancer | AKT protein | Genomes | Myc protein | Kinases | Cancer therapies | K-Ras protein | Gene sequencing | Signal transduction | Cell activation | Alterations | Fos protein | Cell cycle | Environmental studies | Catalysis | Lesions | Deoxyribonucleic acid--DNA | AKT1 protein | Review boards | AKT2 protein | Non-small cell lung carcinoma | Breast cancer | Ribonucleic acid--RNA | 1-Phosphatidylinositol 3-kinase | Medicine | Studies | Signaling | Medical prognosis | Alleles | Genetic research | Mutation | Clinical medicine | Aberration | PTEN protein | Tumors | Cancer | Index Medicus | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e62227 - e62227
Journal Article
Journal of Physical Chemistry B, ISSN 1520-6106, 04/2005, Volume 109, Issue 14, pp. 6714 - 6721
Metadynamics is a powerful technique that has been successfully exploited to explore the multidimensional free energy surface of complex polyatomic systems and... 
MOLECULAR-DYNAMICS | FORCE | PHASE-TRANSITIONS | CHEMISTRY, PHYSICAL | CONSTANT | SIMULATIONS | FREE-ENERGY | Molecular dynamics | Chemistry, Physical and theoretical | Research
Journal Article
ANZ Journal of Surgery, ISSN 1445-1433, 07/2019, Volume 89, Issue 7-8, pp. E329 - E330
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, pp. e0178865 - e0178865
Hyperactivation of the phosphatydil-inositol-3' phosphate kinase (PI3K)/AKT pathway is observed in most NSCLCs, promoting proliferation, migration, invasion... 
BREAST-CANCER | PROMOTES CELL-SURVIVAL | AKT ACTIVATION | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | MACROPHAGE INHIBITORY CYTOKINE-1 | PROSTATE-CANCER | ENDOTHELIAL-CELLS | CYCLIN-DEPENDENT KINASE | LIPOCALIN 2 | ACTIVATING TRANSCRIPTION FACTOR-3 | Lung Neoplasms - genetics | Gene Expression | Lung Neoplasms - enzymology | Humans | Lung Neoplasms - metabolism | PTEN Phosphohydrolase - metabolism | Lung Neoplasms - pathology | Phosphatidylinositol 3-Kinases - metabolism | Mutation | Proto-Oncogene Proteins c-akt - metabolism | Cell Line, Transformed | Inositol | Epithelial cells | Dosage and administration | Genetic aspects | Research | Gene expression | Lung cancer, Non-small cell | Therapy | Transcription factors | Leukocyte migration | Lung cancer | Lung | AKT protein | Metastasis | Signal transduction | Alterations | Catalysis | Growth factors | Activation analysis | Medical research | Activating transcription factor 3 | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Molecular modelling | Inhibitors | Human performance | Aberration | Cell migration | Phosphates | Cell proliferation | Enrichment | FHL2 protein | Biotechnology | Phosphorylation | Insulin-like growth factor-binding protein 2 | Genes | Activation | C cells | Kinases | Cell adhesion & migration | Phosphate | Cell cycle | Insulin-like growth factor-binding protein 3 | Localization | Signatures | AKT1 protein | Pharmacology | Breast cancer | Derivatives | Tumor cell lines | Cyclin-dependent kinase 2 | Signaling | Effectors | Growth hormones | Cost analysis | Prostate cancer | Tumors | Cancer | Apoptosis | Index Medicus
Journal Article