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Cancer Science, ISSN 1347-9032, 04/2010, Volume 101, Issue 4, pp. 831 - 835
The cellular response to genotoxic stress that damages DNA includes cell cycle arrest, activation of DNA repair, and in the event of irreparable damage,... 
Journal Article
International Journal of Clinical Oncology, ISSN 1341-9625, 2/2018, Volume 23, Issue 1, pp. 36 - 44
Journal Article
Cancer Science, ISSN 1347-9032, 11/2004, Volume 95, Issue 11, pp. 866 - 871
BRCA1 (BReast‐CAncer susceptibility gene 1) and BRCA2 are tumor suppressor genes, the mutant phenotypes of which predispose to breast and ovarian cancers.... 
Phosphorylation | Humans | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - genetics | Breast Neoplasms - genetics | Cell Cycle | Genes, BRCA1 - physiology | DNA Repair | Genes, BRCA2 - physiology | Female | Transcription, Genetic | DNA Damage | Mutation | Apoptosis
Journal Article
Japanese Journal of Cancer and Chemotherapy, ISSN 0385-0684, 02/2017, Volume 44, Issue 2, pp. 102 - 106
Journal Article
Gan to kagaku ryoho. Cancer & chemotherapy, ISSN 0385-0684, 02/2017, Volume 44, Issue 2, p. 102
Journal Article
Nutrition, ISSN 0899-9007, 2010, Volume 26, Issue 5, pp. 515 - 521
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 03/2017, Volume 108, Issue 3, pp. 380 - 389
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 10/2014, Volume 6, Issue 10, pp. 1279 - 1293
Epithelial‐mesenchymal transition (EMT) is a reversible and dynamic process hypothesized to be co‐opted by carcinoma during invasion and metastasis. Yet, there... 
drug response | microarray | gene expression signature | epithelial‐mesenchymal transition | prognosis | Drug response | Epithelial-mesenchymal transition | Prognosis | Microarray | Gene expression signature | MOLECULAR SUBTYPES | MEDICINE, RESEARCH & EXPERIMENTAL | GENE-EXPRESSION SIGNATURE | STEM-CELLS | GENOMIC ANALYSES | OVARIAN-CANCER | NEGATIVE BREAST-CANCER | CLINICAL-RELEVANCE | MICRORNA CONTROL | CLAUDIN-LOW | epithelial-mesenchymal transition | SIGNATURE PREDICTS RESISTANCE | Lung Neoplasms - drug therapy | Oligonucleotide Array Sequence Analysis | Colorectal Neoplasms - genetics | Humans | Epithelial-Mesenchymal Transition - drug effects | Antineoplastic Agents - therapeutic use | Epithelial-Mesenchymal Transition - genetics | Ovarian Neoplasms - genetics | Urinary Bladder Neoplasms - genetics | Neoplasms - genetics | Colorectal Neoplasms - drug therapy | Female | Gene Expression Regulation, Neoplastic - drug effects | Ovarian Neoplasms - drug therapy | Lung Neoplasms - genetics | Stomach Neoplasms - genetics | Treatment Outcome | Transcriptome - drug effects | Transcriptome - genetics | Stomach Neoplasms - drug therapy | Urinary Bladder Neoplasms - drug therapy | Breast Neoplasms - drug therapy | Neoplasms - drug therapy | Disease-Free Survival | Breast Neoplasms - genetics | Cell Line, Tumor | Cancer patients | Chemotherapy | Patient outcomes | Stem cells | Development and progression | Drug therapy | Gene expression | Ovarian cancer | Cancer | Mesenchyme | Colorectal carcinoma | Genomes | Breast cancer | Metastasis | Kinases | Cancer therapies | Metastases | Breast carcinoma | Genotype & phenotype | Cell lines | Paclitaxel | Breast | Software | Tumors
Journal Article