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Critical Reviews in Oncology and Hematology, ISSN 1040-8428, 2016, Volume 102, pp. 73 - 81
Abstract Tumor growth rate (TGR), usually defined as the ratio between the slope of tumor growth before the initiation of treatment and the slope of tumor... 
Hematology, Oncology and Palliative Medicine | Targeted therapies | RECIST | Tumor response | Everolimus | mRCC
Journal Article
Journal Article
Current Pharmaceutical Design, ISSN 1381-6128, 2014, Volume 20, Issue 24, pp. 3861 - 3862
Journal Article
Infection, ISSN 0300-8126, 10/2018, Volume 46, Issue 5, pp. 607 - 615
Journal Article
Critical Reviews in Oncology/Hematology, ISSN 1040-8428, 06/2016, Volume 102, pp. 73 - 81
Journal Article
Journal Article
SCIENTIFIC REPORTS, ISSN 2045-2322, 08/2019, Volume 9, Issue 1, pp. 12286 - 13
Pancreatic ductal adenocarcinoma (PDA) has a highly immunosuppressive microenvironment, which is contributed by the complex interaction between cancer cells... 
SUPPRESSOR | MAINTENANCE | PROGENITORS | T-CELL EXCLUSION | INHIBITION | 7 C7 | DUCTAL ADENOCARCINOMA | IMMUNOTHERAPY | MULTIDISCIPLINARY SCIENCES | TUMOR | CANCER | Adenocarcinoma | Carcinoma | Cell survival | Transforming growth factor-b1 | Syngeneic grafts | CD8 antigen | Pancreatic islet transplantation | Lymphocytes T | Metastasis | Leukocytes | Macrophages | Metastases | Organoids | Stromal cells | Pancreas transplantation | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0130142
Background The potential predictive role of programmed death-ligand-1 (PD-L1) expression on tumor cells in the context of solid tumor treated with checkpoint... 
CTLA-4 BLOCKADE | ANTI-PD-L1 ANTIBODY | EFFICACY | SAFETY | MULTIDISCIPLINARY SCIENCES | OPEN-LABEL | ANTITUMOR IMMUNITY | CLINICAL ACTIVITY | IPILIMUMAB | CHEMOTHERAPY | CHECKPOINT BLOCKADE | Lung Neoplasms - drug therapy | Melanoma - diagnosis | Prognosis | Humans | Gene Expression Regulation, Neoplastic | Antibodies, Monoclonal - therapeutic use | Antineoplastic Agents - therapeutic use | Carcinoma, Non-Small-Cell Lung - diagnosis | Neoplasms - diagnosis | Urogenital System - pathology | Neoplasms - genetics | Melanoma - genetics | Urogenital Neoplasms - drug therapy | Lung - metabolism | Lung Neoplasms - genetics | Lung - pathology | Antibodies, Monoclonal, Humanized - therapeutic use | Carcinoma, Non-Small-Cell Lung - genetics | Treatment Outcome | Urogenital System - drug effects | B7-H1 Antigen - genetics | Neoplasms - drug therapy | Urogenital System - metabolism | Urogenital Neoplasms - genetics | Lung - drug effects | Melanoma - drug therapy | Carcinoma, Non-Small-Cell Lung - drug therapy | Lung Neoplasms - diagnosis | Urogenital Neoplasms - diagnosis | Lung cancer, Non-small cell | Care and treatment | Tumors | PD-1 protein | Lung cancer | Clinical trials | Antibodies | Oncology | Cancer therapies | Confidence intervals | Immunotherapy | Sensitivity analysis | Tumor cells | Melanoma | Non-small cell lung carcinoma | Patients | Pathology | Immune checkpoint | Medical prognosis | PD-L1 protein | Response rates | Biomarkers | Ligands | Mutation | Solid tumors | Blood diseases | Cancer | Apoptosis
Journal Article
2011, ISBN 9780857291653
Web Resource
Scientific Reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, pp. 43013 - 43013
Combined MAPK/PI3K pathway inhibition represents an attractive, albeit toxic, therapeutic strategy in oncology. Since PTEN lies at the intersection of these... 
LUNG-CANCER | METASTATIC MELANOMA | SIGNALING PATHWAYS | RAS/RAF/MEK/ERK | MULTIDISCIPLINARY SCIENCES | DEREGULATION | PI3K/PTEN/AKT/MTOR | COMBINATION | NF-KAPPA-B | MAPK PATHWAY | STEM-CELL POPULATION | Neoplastic Stem Cells - cytology | TOR Serine-Threonine Kinases - metabolism | Neoplastic Stem Cells - drug effects | Humans | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | TOR Serine-Threonine Kinases - antagonists & inhibitors | Janus Kinase 1 - metabolism | PTEN Phosphohydrolase - antagonists & inhibitors | Neoplastic Stem Cells - metabolism | RNA Interference | Janus Kinase 1 - genetics | Female | Pyrimidinones - pharmacology | MAP Kinase Kinase Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | PTEN Phosphohydrolase - genetics | Everolimus - pharmacology | PTEN Phosphohydrolase - metabolism | MAP Kinase Kinase Kinases - metabolism | Drug Synergism | Animals | Mice, Nude | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Pyridones - pharmacology | RNA, Small Interfering - metabolism | TOR protein | Stat3 protein | MAP kinase | Janus kinase | mRNA | Tumor cell lines | PTEN protein | Cancer | 1-Phosphatidylinositol 3-kinase | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2013, Volume 133, Issue 4, pp. 1016 - 1022
Journal Article