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Nutrition Research and Practice, ISSN 1976-1457, 2019, Volume 13, Issue 2, pp. 87 - 94
BACKGROUND/OBJECTIVES: Oxidative stress is a major effector of various diseases; accordingly, antioxidants are frequently ingested in order to prevent or... 
Antioxidants | NUTRITION & DIETETICS | kimchi | fermentation | VEGETABLES | raspberries | oxidative stress | Oxidative stress | Animal models | Transaminase | Liver | Pyruvic acid | Superoxide dismutase | Superoxide | Acidity | Fermentation | Phenolic compounds | Powder | Feeding | Cirrhosis | Thioacetamide | Bioactive compounds | Oxidation | Lactic acid | Liver cirrhosis | Original Research | 생활과학
Journal Article
Immunology and Cell Biology, ISSN 0818-9641, 07/2012, Volume 90, Issue 6, pp. 601 - 610
Journal Article
by Yun, HY and Kim, MW and Lee, HS and Kim, W and Shin, JH and Kim, H and Shin, HC and Park, H and Oh, BH and Kim, WK and Bae, KH and Lee, SC and Lee, EW and Ku, B and Kim, SJ
PLOS BIOLOGY, ISSN 1544-9173, 07/2019, Volume 17, Issue 7, pp. e3000367 - e3000367
Human papillomaviruses (HPVs) are causative agents of various diseases associated with cellular hyperproliferation, including cervical cancer, one of the most... 
COMPLEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | BIOLOGY | INDEPENDENT FUNCTIONS | DIMERIZATION | DEGRADATION | TYROSINE PHOSPHATASES | MECHANISMS | IDENTIFICATION | TYPE-16 E7 | Oncogene Proteins, Viral - chemistry | Humans | Uterine Cervical Neoplasms - pathology | DNA-Binding Proteins - metabolism | Protein Tyrosine Phosphatases, Non-Receptor - chemistry | Uterine Cervical Neoplasms - metabolism | Protein Tyrosine Phosphatases, Non-Receptor - genetics | HEK293 Cells | Protein Domains | Female | Oncogene Proteins, Viral - genetics | Retinoblastoma Protein - chemistry | Oncogene Proteins, Viral - metabolism | Amino Acid Sequence | Cell Line | Retinoblastoma Protein - metabolism | Models, Molecular | DNA-Binding Proteins - genetics | Uterine Cervical Neoplasms - genetics | DNA-Binding Proteins - chemistry | Sequence Homology, Amino Acid | Cell Transformation, Neoplastic | Retinoblastoma Protein - genetics | Protein Tyrosine Phosphatases, Non-Receptor - metabolism | Cell Line, Tumor | Protein Binding | HeLa Cells | Physiological aspects | Tumor suppressor genes | Structure | Papillomavirus infections | Oncogenes | Cell proliferation | Biotechnology | Transformation | Phosphorylation | Leukocyte migration | Funding | Kinases | Phosphatase | Degradation | Proteins | Human papillomavirus | Human papilloma viruses | Tumorigenesis | Catalysis | Supervision | Genotypes | Crystal structure | Binding | Tyrosine | Retinoblastoma protein | Cervix | Metabolism | Mutagenesis | Software | Disruption | Retinoblastoma | Cell migration | Cervical cancer | Cancer | Tumors | Structural analysis | Protein-tyrosine-phosphatase | Index Medicus | Risk groups
Journal Article
Obesity, ISSN 1930-7381, 04/2010, Volume 18, Issue 4, pp. 780 - 787
Obesity‐induced inflammation contributes to the development of obesity‐related metabolic disorders such as insulin resistance, type 2 diabetes, fatty liver... 
CRUCIAL ROLE | PPAR-ALPHA | METABOLIC SYNDROME | ACTIVATION | NUTRITION & DIETETICS | ADIPONECTIN | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | RECEPTOR | TNF-ALPHA | ADIPOSE-TISSUE | Glucose Intolerance - metabolism | Dietary Fats - metabolism | Obesity - drug therapy | Capsaicin - pharmacology | Capsaicin - therapeutic use | Male | Insulin - blood | RNA, Messenger - metabolism | Adipose Tissue - metabolism | TRPV Cation Channels - metabolism | Flow Cytometry | Leptin - blood | Glucose Intolerance - etiology | Adiponectin - genetics | Dietary Fats - administration & dosage | Anti-Inflammatory Agents - therapeutic use | Adiponectin - metabolism | Disease Models, Animal | Hypoglycemic Agents - therapeutic use | Gene Expression | Fatty Liver - metabolism | Anti-Inflammatory Agents - pharmacology | Obesity - complications | Liver - metabolism | Mice, Inbred C57BL | Insulin Resistance | PPAR alpha - genetics | Reverse Transcriptase Polymerase Chain Reaction | Fatty Liver - drug therapy | TRPV Cation Channels - genetics | Hypoglycemic Agents - pharmacology | Obesity - metabolism | Triglycerides - metabolism | Animals | Lipid Metabolism - drug effects | Inflammation - genetics | Mice, Obese | Mice | PPAR alpha - metabolism | Blood Glucose - metabolism | Dietary Supplements | PPAR alpha - chemistry | Fatty Liver - etiology
Journal Article
Nature Communications, ISSN 2041-1723, 09/2016, Volume 7, Issue 1, pp. 12961 - 12961
Nicotinic acetylcholine receptors (nAChRs) binding to the tobacco-specific carcinogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) induces Ca2+... 
CANCER GROWTH | INSULIN | AKT ACTIVATION | MULTIDISCIPLINARY SCIENCES | RISK | 4-(METHYLNITROSAMINO)-1-(3-PYRIDYL)-1-BUTANONE | NICOTINIC ACETYLCHOLINE-RECEPTORS | SRC FAMILY | BINDING | RAB GTPASES | GROWTH-FACTOR-I
Journal Article
Journal Article
Scientific reports, ISSN 2045-2322, 03/2019, Volume 9, Issue 1, pp. 3644 - 9
Molecular testing is increasingly important in cancer diagnosis. Targeted next generation sequencing (NGS) is widely accepted method but structural variation... 
MULTIDISCIPLINARY SCIENCES | IN-SITU HYBRIDIZATION | FISH | FRAMEWORK | 19Q | TUMORS | DELETIONS | Next-generation sequencing | Copy number | Neural networks | Brain tumors | Brain cancer | Data processing | Single-nucleotide polymorphism | Genomes | Tumors | Cancer
Journal Article