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SAE technical paper series, Volume 2015-01-0980.
This study aims to build a conceptual simulation used at the early stage of PHEV development. This simulation enables to design vehicle concept and fundamental... 
Product development | Simulation and modeling | Hybrid electric vehicles
eJournal
Nature Medicine, ISSN 1078-8956, 03/2012, Volume 18, Issue 3, pp. 405 - 412
The signaling molecule Wnt regulates bone homeostasis through beta-catenin-dependent canonical and beta-catenin-independent noncanonical pathways. Impairment... 
RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECESSIVE ROBINOW-SYNDROME | CELL BIOLOGY | BONE-FORMATION | PATHWAY | DIFFERENTIATION | ROR2 | C-JUN | WNT | EXPRESSION | RECEPTOR TYROSINE KINASE | Arthritis - metabolism | Wnt Proteins - deficiency | Bone Diseases - metabolism | Wnt-5a Protein | Osteoclasts - cytology | Wnt Proteins - metabolism | X-Ray Microtomography | MAP Kinase Kinase 4 - metabolism | Wnt Proteins - genetics | Osteoblasts - cytology | Receptor Activator of Nuclear Factor-kappa B - metabolism | Wnt Signaling Pathway | Promoter Regions, Genetic | Proto-Oncogene Proteins c-jun - genetics | Receptor Tyrosine Kinase-like Orphan Receptors - metabolism | Gene Expression Regulation | Receptor Tyrosine Kinase-like Orphan Receptors - deficiency | Osteoclasts - metabolism | Cell Lineage | Animals | Proto-Oncogene Proteins c-jun - metabolism | Skull - cytology | Receptor Activator of Nuclear Factor-kappa B - genetics | Receptor Tyrosine Kinase-like Orphan Receptors - genetics | Mice | Osteoblasts - metabolism | Skull - growth & development | Osteoporosis | Care and treatment | Physiological aspects | Cellular signal transduction | Genetic aspects | Research | Osteoclasts (Biology) | Proteins | Signal transduction | Bone density | Cellular biology | Gene expression
Journal Article
by Yamagami, Keitaro and Kurogi, Ryota and Kurogi, Ai and Nishimura, Hiroyuki and Nishimura, Shinjitsu and Nishimura, Yasuaki and Nishimura, Kunihiro and Nishimura, Ataru and Onozuka, Daisuke and Ren, Nice and Kada, Akiko and Arimura, Koichi and Ido, Keisuke and Mizoguchi, Masahiro and Sakamoto, Seisaburo and Sakamoto, Tetsuya and Kayama, Takamasa and Suzuki, Nozomi and Suzuki, Michiyasu and Suzuki, Sachio and Suzuki, Hidenori and Suzuki, Ichiro and Suzuki, Susumu and Arai, Yoshinori and Arai, Hajime and Arai, Motohiro and Hagihara, Akihito and Iihara, Koji and Takigami, Masayoshi and Kamiyama, Kenji and Houkin, Kiyohiro and Nishi, Shougo and Yoshimoto, Tetsuyuki and Yoshimoto, Junpei and Kaneko, Sadao and Oka, Hirofumi and Oka, Koji and Ooyama, Hiroshi and Kamada, Kyousuke and Makino, Kenichi and Tokumitsu, Naoki and Sako, Kazuhiro and Izumi, Naoto and Nitta, Hisashi and Nitta, Kazumi and Ootaki, Masahumi and Isobe, Masanori and Nishiya, Mikio and Yamazaki, Takaaki and Mabuchi, Syouji and Mabuchi, Eiichiro and Ogasawara, Kuniaki and Kubo, Naohiko and Shimizu, Yukihiko and Saito, Hitoshi and Saito, Keiichi and Yamanome, Tatumi and Yoshino, Kimihiro and Yoshino, Atsuo and Fujitsuka, Mitsuyuki and Takami, Masaaki and Ohtaka, Hirotoshi and Hirano, Teruyuki and Shiokawa, Yosiaki and Okada, Takaharu and Kohno, Michihiro and Haraoka, Jou and Kawamura, Noriyoshi and Kawamura, Tadao and Isoshima, Akira and Yasue, Masaharu and Takayoshi Kobayashi, Mitsuhiko Hokari and Kawai, Kensuke and Maehara, Taketoshi and Noguchi, Makoto and Hoshino, Haruhiko and Hiyama, Hirofumi and Yoshida, Kensaku and Yoshida, Kazunari and Utsugi, Osamu and Takeda, Yasuaki and Takeda, Yuu and Tamaki, Kouichi and Karasudani, Hirohide and Urabe, Takao and Kobayashi, Nozomu and Kobayashi, Shiro and Nakamura, Yusaku and Nakamura, Kazuhito and Nakamura, Michio and Nakamura, Yoshinari and Koguchi, Yorio and Ono, Junichi and Suda, Sumio and Hadeishi, Hiromu and Fukutake, Toshio and Wakui, Kenji and Tanno, Hirokazu and Ishige, Naoki and Ohasi, Takashi and ... and J-ASPECT Study Collaborators
World Neurosurgery, ISSN 1878-8750, 10/2019, Volume 130, pp. e26 - e46
The epidemiology of patients with traumatic brain injury (TBI) has changed dramatically over recent decades as a result of rapid advances in aging societies.... 
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma | SURGERY | CARE | CLINICAL NEUROLOGY | IMPACT | CLINICAL-VARIABLES | HOSPITAL VOLUME | GENDER | COMA
Journal Article
Developmental Dynamics, ISSN 1058-8388, 01/2018, Volume 247, Issue 1, pp. 24 - 32
Journal Article
Journal of the Acoustical Society of America, ISSN 0001-4966, 10/2016, Volume 140, Issue 4, pp. 3060 - 3060
In this study, we attempt a simultaneous recognition method of phone and speaker using a single energy-based model, a three-way restricted Boltzmann machine... 
Journal Article
Science Signaling, ISSN 1945-0877, 08/2017, Volume 10, Issue 494, p. eaan0023
Cytoskeletal reorganization in osteoclasts to form actin rings is necessary for these cells to attach to bone and resorb bone matrices. We delineated the... 
PODOSOME ORGANIZATION | SEALING ZONE | ACTIN | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RHO | RECEPTOR | SRC KINASE | DIFFERENTIATION | LIGAND | ALPHA(V)BETA INTEGRIN | CELL BIOLOGY | Protein Kinase C - genetics | Phosphorylation | Wnt-5a Protein - genetics | Humans | Actins - metabolism | Arthritis, Rheumatoid - metabolism | Focal Adhesion Kinase 2 - genetics | Wnt-5a Protein - metabolism | Focal Adhesion Kinase 2 - metabolism | Osteoporosis - metabolism | Protein Kinase C - metabolism | src-Family Kinases - metabolism | Bone Resorption - metabolism | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Disease Models, Animal | Receptor Tyrosine Kinase-like Orphan Receptors - metabolism | rho GTP-Binding Proteins - genetics | Mice, Transgenic | Osteoclasts - metabolism | Arthritis, Rheumatoid - pathology | Periodontal Diseases - metabolism | Animals | Periodontal Diseases - pathology | Osteoporosis - pathology | rho GTP-Binding Proteins - metabolism | Bone Resorption - pathology | Receptor Tyrosine Kinase-like Orphan Receptors - genetics | Mice | src-Family Kinases - genetics | Wnt protein | Proline | Reversing | Arthritis | Bone diseases | Kinases | Metastases | Bone resorption | Proteins | Osteoporosis | Signal transduction | Receptors | Actin | Protein-tyrosine kinase receptors | Biocompatibility | Bone density | Protein-tyrosine kinase | Binding | Tyrosine | Periodontics | Therapeutic applications | RhoA protein | Src protein | Signaling | Periodontal disease | Bone mass | Rheumatoid arthritis | Cytoskeleton | Osteoclasts | Cancer | Guanosinetriphosphatase
Journal Article
Genes to Cells, ISSN 1356-9597, 07/2018, Volume 23, Issue 7, pp. 606 - 613
Malignant pleural mesothelioma (MPM) is a highly aggressive tumor with poor prognosis and closely related to exposure to asbestos. MPM is a heterogeneous tumor... 
malignant pleural mesothelioma | invasion | proliferation | Ror receptor tyrosine kinases | Rac | Cdc42 | SURVIVAL | PROGNOSTIC-FACTORS | YAP | CELL BIOLOGY | INVASIVENESS | MORPHOGENESIS | DISEASES | PATHWAY | JAPAN | GENETICS & HEREDITY | EXPRESSION | Tyrosine | Phenols | Mesothelioma | G proteins | Cdc42 protein | Cell proliferation | Asbestos | Prognosis | Tumor cells | Invasiveness | Cyclin-dependent kinases | Cell lines | Rac1 protein
Journal Article
Cancer Science, ISSN 1347-9032, 03/2016, Volume 107, Issue 3, pp. 290 - 297
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e102797
In diabetics, methylglyoxal (MG), a glucose-derived metabolite, plays a noxious role by inducing oxidative stress, which causes and exacerbates a series of... 
LRP5 GENE | HIGH-BONE-MASS | INDUCED OXIDATIVE STRESS | MULTIDISCIPLINARY SCIENCES | PROMOTER REGION | RECEPTOR-RELATED PROTEIN-5 | METHYLATION STATUS | HYDROGEN-PEROXIDE | MICE | TRANSCRIPTIONAL REGULATION | GLYCATION END-PRODUCTS | Diabetes Mellitus - genetics | Gene Expression - genetics | Methyl-CpG-Binding Protein 2 - metabolism | Wnt Proteins - metabolism | Promoter Regions, Genetic - genetics | Methyl-CpG-Binding Protein 2 - genetics | Pyruvaldehyde - metabolism | Wnt Proteins - genetics | Epigenomics - methods | Receptor Activator of Nuclear Factor-kappa B - metabolism | Proto-Oncogene Proteins - metabolism | Cell Line | Oxidative Stress - genetics | Diabetes Mellitus - metabolism | Mesenchymal Stromal Cells - metabolism | Proto-Oncogene Proteins - genetics | Up-Regulation - genetics | DNA Methylation - genetics | Signal Transduction - genetics | beta Catenin - metabolism | Down-Regulation - genetics | beta Catenin - genetics | Bone Diseases, Metabolic - metabolism | Animals | Epigenesis, Genetic - genetics | Receptor Activator of Nuclear Factor-kappa B - genetics | Mice | Bone Diseases, Metabolic - genetics | Oxidative stress | Bisulfite | Frizzled-related protein | Wnt protein | Frizzled protein | Genes | DNA damage | Inactivation | Osteoblasts | Gene sequencing | Osteoprotegerin | Proteins | Osteoporosis | β-catenin | Demethylation | Biomedical materials | Methyl-CpG binding protein | Rodents | DNA methylation | Bone marrow | Aging | Biocompatibility | Physiology | TRANCE protein | Deoxyribonucleic acid--DNA | Osteopenia | University graduates | CpG islands | NF-κB protein | Deactivation | Complications | Legal medicine | Diabetes mellitus | Stroma | Gene expression | Medicine | Pathology | Signaling | Deoxyguanosine | Sodium | MeCP2 protein | Derepression | Epigenetics | Biomarkers | Diabetes | Pyruvaldehyde | Deoxyribonucleic acid | DNA
Journal Article