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physics (38) 38
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arrangements or circuits for control of indicating devicesusing static means to present variable information (9) 9
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climate change mitigation technologies in information andcommunication technologies [ict], i.e. information andcommunication technologies aiming at the reduction of thir ownenergy use (7) 7
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immunologic diseases. allergy (7) 7
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Journal of Nihon University Medical Association, ISSN 0029-0424, 04/2018, Volume 77, Issue 2, pp. 115 - 117
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2014, Volume 124, Issue 9, pp. 3987 - 4003
Journal Article
International Journal of Cell Biology, ISSN 1687-8876, 2014, Volume 2014, pp. 502676 - 12
Autophagy represents a homeostatic cellular mechanism for the turnover of organelles and proteins, through a lysosome-dependent degradation pathway. During... 
Autophagy (Cytology) | Cell interaction | Identification and classification | Proteins | Mitochondria | Disease | Pathogenesis | Homeostasis | Ligands | Autophagy | Apoptosis | Review
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2013, Volume 123, Issue 12, pp. 5212 - 5230
Chronic obstructive pulmonary disease (COPD) involves aberrant airway inflammatory responses to cigarette smoke (CS) that are associated with epithelial cell... 
SMOKE-INDUCED EMPHYSEMA | MEDICINE, RESEARCH & EXPERIMENTAL | OBSTRUCTIVE PULMONARY-DISEASE | UNFOLDED PROTEIN RESPONSE | EPITHELIAL-CELLS | MONITORING AUTOPHAGY | UBIQUITIN | IN-VIVO | CIGARETTE-SMOKE | ENDOPLASMIC-RETICULUM STRESS | HDAC6 | Cilia - physiology | NF-E2-Related Factor 2 - physiology | Humans | Male | Autophagy - physiology | Mucus | Pulmonary Disease, Chronic Obstructive - etiology | Pulmonary Disease, Chronic Obstructive - pathology | Cytosol - enzymology | Ubiquitination | Tobacco Products | Apoptosis Regulatory Proteins - deficiency | Cilia - ultrastructure | Phenylbutyrates - pharmacology | Female | Microtubule-Associated Proteins - deficiency | Sirtuin 1 - deficiency | Beclin-1 | Microscopy, Electron, Scanning | Trachea - cytology | Histone Deacetylase 6 | Mice, Inbred C57BL | Cells, Cultured | Epithelial Cells - ultrastructure | NF-E2-Related Factor 2 - deficiency | Mice, Knockout | Pulmonary Disease, Chronic Obstructive - enzymology | Phenotype | Animals | Tobacco Smoke Pollution - adverse effects | Sirtuin 1 - physiology | Histone Deacetylase Inhibitors - pharmacology | Mice | Protein Processing, Post-Translational | Histone Deacetylases - physiology | Proteasome Endopeptidase Complex - metabolism | Physiological aspects | Lung diseases, Obstructive | Research | Epithelial cells | Health aspects | Smoking | Proteins | Statistical analysis | Womens health | Pathogenesis | Lung diseases | Mortality | Chronic obstructive pulmonary disease | Genomes | Grants | Autophagy | Binding sites | Apoptosis
Journal Article
Frontiers in Pharmacology, ISSN 1663-9812, 2014, Volume 5, p. 244
Autophagy was originally described as a highly conserved system for the degradation of cytosol through a lysosome-dependent pathway. In response to starvation,... 
Xenophagy | Ciliophagy | Mitophagy | Autophagy | Lung diseases | lung diseases | autophagy | PULMONARY-HYPERTENSION | RECOGNITION | ciliophagy | TUBERCULOSIS | INDUCTION | xenophagy | PATHOGENESIS | mitophagy | PINK1 | MITOCHONDRIAL DYSFUNCTION | TARGETS | PHARMACOLOGY & PHARMACY | CONTRIBUTES | Lung Diseases
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, p. e0121246
Background Epithelial cell death is a major contributor to fibrogenesis in the lung. In this study, we sought to determine the function of mitochondria and... 
PARKIN | RECRUITMENT | ACTIVATION | DISEASES | MULTIDISCIPLINARY SCIENCES | DAMAGED MITOCHONDRIA | DEGRADATION | MITOPHAGY | AUTOPHAGY | PINK1-DEFICIENT MICE | PROMOTES | Protein Kinases - metabolism | Cell Line | Gene Expression Regulation, Enzymologic - drug effects | Pulmonary Alveoli - pathology | Reactive Oxygen Species - metabolism | Epithelial Cells - drug effects | Humans | Epithelial Cells - pathology | Male | Pulmonary Fibrosis - pathology | Bleomycin - adverse effects | Mitochondria - drug effects | Mitochondria - pathology | Membrane Potential, Mitochondrial - drug effects | Animals | Mitochondrial Degradation - drug effects | Pulmonary Fibrosis - enzymology | Pulmonary Fibrosis - chemically induced | Mice | Cell Death - drug effects | Transforming Growth Factor beta1 - pharmacology | Cell death | Analysis | Fibrosis | Bone morphogenetic proteins | Respiratory tract diseases | Mitochondrial DNA | B cells | Transforming growth factors | Oxidative stress | Flow cytometry | Epithelial cells | Homeostasis | Critical care | Kinases | Autophagy | Experiments | Medical schools | Western blotting | Depolarization | Mitochondria | Bleomycin | Rodents | Animal tissues | Alveoli | Growth factors | Transforming growth factor-b1 | Mortality | Lung diseases | Gene expression | Electron microscopy | Medicine | Cytometry | Hospitals | Transmission electron microscopy | Pulmonary fibrosis | Lungs | Womens health | PTEN-induced putative kinase | Laboratory animals | Alzheimers disease | Immunofluorescence | PTEN protein | Apoptosis
Journal Article
Journal Article
Annals of the American Thoracic Society, ISSN 2325-6621, 03/2016, Volume 13, Issue Suppl 1, pp. S40 - S47
Journal Article
Journal Article
Respiration and Circulation, ISSN 0452-3458, 01/2014, Volume 62, Issue 1, pp. 55 - 60
Journal Article
The Journal of Allergy and Clinical Immunology: In Practice, ISSN 2213-2198, 01/2019, Volume 7, Issue 1, pp. 134 - 145.e1
Asthma-chronic obstructive pulmonary disease (COPD) overlap (ACO) has been proposed as a different diagnosis from asthma and COPD. However, little is known... 
Type 2 biomarker | Asthma-COPD overlap | YKL-40 | Periostin | AIR-FLOW LIMITATION | LUNG |