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1. Full Text High-grade glioma associated immunosuppression does not prevent immune responses induced by therapeutic vaccines in combination with T.sub.reg depletion
by Lohr, Mario and Freitag, Benjamin and Technau, Antje and Krauss, Jurgen and Monoranu, Camelia-Maria and Rachor, Johannes and Lutz, Manfred B
Cancer Immunology, Immunotherapy, ISSN 0340-7004, 10/2018, Volume 67, Issue 10, p. 1545
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00262-018-2214-0... 
Cancer vaccines | Evaluation | Immune response | Gliomas | Analysis | Vaccination | T cells | Health aspects
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2. Full Text Reduced H3K27me3 and DNA Hypomethylation Are Major Drivers of Gene Expression in K27M Mutant Pediatric High-Grade Gliomas
by Bender, Sebastian and Tang, Yujie and Lindroth, Anders M and Hovestadt, Volker and Jones, David T.W and Jones, Chris and Kool, Marcel and Zapatka, Marc and Northcott, Paul A and Sturm, Dominik and Wang, Wei and Radlwimmer, Bernhard and Højfeldt, Jonas W and Truffaux, Nathalène and Castel, David and Schubert, Simone and Ryzhova, Marina and Şeker-Cin, Huriye and Gronych, Jan and Johann, Pascal David and Stark, Sebastian and Meyer, Jochen and Milde, Till and Schuhmann, Martin and Ebinger, Martin and Monoranu, Camelia-Maria and Ponnuswami, Anitha and Chen, Spenser and Witt, Olaf and Collins, V. Peter and von Deimling, Andreas and Jabado, Nada and Puget, Stephanie and Grill, Jacques and Helin, Kristian and Korshunov, Andrey and Lichter, Peter and Monje, Michelle and Plass, Christoph and Cho, Yoon-Jae and Pfister, Stefan M
Cancer Cell, ISSN 1535-6108, 11/2013, Volume 24, Issue 5, pp. 660 - 672
Two recurrent mutations, K27M and G34R/V, within histone variant H3.3 were recently identified in ∼50% of pHGGs. Both mutations define clinically and... 
METHYLATION | GLIOBLASTOMA | HYPERMETHYLATION | ONCOLOGY | EMBRYONIC STEM-CELLS | MUTATION | INTRINSIC PONTINE GLIOMAS | TARGETS | HISTONE H3.3 | POLYCOMB | CANCER | CELL BIOLOGY | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | Brain Neoplasms - genetics | Molecular Sequence Data | Mutation, Missense | Brain Neoplasms - metabolism | Brain Stem Neoplasms - metabolism | DNA Methylation | Brain Stem Neoplasms - genetics | Histones - genetics | Genes, Dominant | Glioblastoma - genetics | Histone-Lysine N-Methyltransferase - metabolism | Cell Line, Tumor | Protein Binding | Glioblastoma - metabolism | Transcription, Genetic | Protein Processing, Post-Translational | Histones - metabolism | Methylation | Polycomb Repressive Complex 2 - metabolism | Child | Pediatrics | Genetic aspects | Gene expression | Gliomas | DNA | Cancer
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3. Full Text DNA methylation-based classification of central nervous system tumours
by Capper, David and Jones, David T. W and Sill, Martin and Hovestadt, Volker and Schrimpf, Daniel and Sturm, Dominik and Koelsche, Christian and Sahm, Felix and Chavez, Lukas and Reuss, David E and Kratz, Annekathrin and Wefers, Annika K and Huang, Kristin and Pajtler, Kristian W and Schweizer, Leonille and Stichel, Damian and Olar, Adriana and Engel, Nils W and Lindenberg, Kerstin and Harter, Patrick N and Braczynski, Anne K and Plate, Karl H and Dohmen, Hildegard and Garvalov, Boyan K and Coras, Roland and Hölsken, Annett and Hewer, Ekkehard and Bewerunge-Hudler, Melanie and Schick, Matthias and Fischer, Roger and Beschorner, Rudi and Schittenhelm, Jens and Staszewski, Ori and Wani, Khalida and Varlet, Pascale and Pages, Melanie and Temming, Petra and Lohmann, Dietmar and Selt, Florian and Witt, Hendrik and Milde, Till and Witt, Olaf and Aronica, Eleonora and Giangaspero, Felice and Rushing, Elisabeth and Scheurlen, Wolfram and Geisenberger, Christoph and Rodriguez, Fausto J and Becker, Albert and Preusser, Matthias and Haberler, Christine and Bjerkvig, Rolf and Cryan, Jane and Farrell, Michael and Deckert, Martina and Hench, Jürgen and Frank, Stephan and Serrano, Jonathan and Kannan, Kasthuri and Tsirigos, Aristotelis and Brück, Wolfgang and Hofer, Silvia and Brehmer, Stefanie and Seiz-Rosenhagen, Marcel and Hänggi, Daniel and Hans, Volkmar and Rozsnoki, Stephanie and Hansford, Jordan R and Kohlhof, Patricia and Kristensen, Bjarne W and Lechner, Matt and Lopes, Beatriz and Mawrin, Christian and Ketter, Ralf and Kulozik, Andreas and Khatib, Ziad and Heppner, Frank and Koch, Arend and Jouvet, Anne and Keohane, Catherine and Mühleisen, Helmut and Mueller, Wolf and Pohl, Ute and Prinz, Marco and Benner, Axel and Zapatka, Marc and Gottardo, Nicholas G and Driever, Pablo Hernáiz and Kramm, Christof M and Müller, Hermann L and Rutkowski, Stefan and Von Hoff, Katja and Frühwald, Michael C and Gnekow, Astrid and Fleischhack, Gudrun and Tippelt, Stephan and Calaminus, Gabriele and Monoranu, Camelia-Maria and Perry, Arie and Jones, Chris and ...
Nature, ISSN 0028-0836, 03/2018, Volume 555, Issue 7697, pp. 469 - 474
Accurate pathological diagnosis is crucial for optimal management of patients with cancer. For the approximately 100 known tumour types of the central nervous... 
MOLECULAR CLASSIFICATION | MULTICENTER | ENTITY | GLIOMA | MULTIDISCIPLINARY SCIENCES | PILOCYTIC ASTROCYTOMA | CLASS PROBABILITY ESTIMATION | SUBGROUPS | MEDULLOBLASTOMA | EXPRESSION | BIOCONDUCTOR | Central Nervous System Neoplasms - classification | Reproducibility of Results | Central Nervous System Neoplasms - genetics | Humans | Middle Aged | Child, Preschool | Infant | Male | Unsupervised Machine Learning | Young Adult | Central Nervous System Neoplasms - pathology | DNA Methylation | Adolescent | Aged, 80 and over | Adult | Female | Aged | Central Nervous System Neoplasms - diagnosis | Child | Cohort Studies | Genetic aspects | Nucleotide sequencing | Diagnosis | Methylation | Observations | Methods | DNA sequencing | Tumors | Cancer | Analysis | DNA | Machine learning | Central nervous system | Classifiers | Brain cancer | Standardization | Clinical trials | Probability | Data processing | Nervous system | Genomes | Gene expression | Generalized linear models | Learning algorithms | Classification | Diagnostic software | DNA methylation | Diagnostic systems | Bioinformatics | Deoxyribonucleic acid--DNA
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4. Full Text Changes in the expression of genes related to neuroinflammation over the course of sporadic Alzheimer’s disease progression: CX3CL1, TREM2, and PPARγ
by Strobel, S and Grünblatt, E and Riederer, P and Heinsen, H and Arzberger, T and Al-Sarraj, S and Troakes, C and Ferrer, I and Monoranu, Camelia Maria
Journal of Neural Transmission, ISSN 0300-9564, 7/2015, Volume 122, Issue 7, pp. 1069 - 1076
The role of neuroinflammation in the pathogenesis of neurodegenerative diseases has become more evident in recent years. Research on the etiology and... 
Neurology | Neurosciences | Medicine & Public Health | Sporadic Alzheimer’s disease | Neuroinflammation | TREM2 | Pharmacology/Toxicology | Psychiatry | CX3CL1 | PPARγ
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5. Full Text New Brain Tumor Entities Emerge from Molecular Classification of CNS-PNETs
by Sturm, Dominik and Orr, Brent A and Toprak, Umut H and Hovestadt, Volker and Jones, David T.W and Jones, Chris and Capper, David and Sill, Martin and Buchhalter, Ivo and Northcott, Paul A and Leis, Irina and Ryzhova, Marina and Koelsche, Christian and Pfaff, Elke and Allen, Sariah J and Balasubramanian, Gnanaprakash and Worst, Barbara C and Pajtler, Kristian W and Brabetz, Sebastian and Johann, Pascal D and Sahm, Felix and Reimand, Jüri and Mackay, Alan and Carvalho, Diana M and Remke, Marc and Phillips, Joanna J and Perry, Arie and Cowdrey, Cynthia and Drissi, Rachid and Fouladi, Maryam and Giangaspero, Felice and Łastowska, Maria and Grajkowska, Wiesława and Scheurlen, Wolfram and Pietsch, Torsten and Hagel, Christian and Gojo, Johannes and Lötsch, Daniela and Berger, Walter and Slavc, Irene and Haberler, Christine and Jouvet, Anne and Holm, Stefan and Hofer, Silvia and Prinz, Marco and Keohane, Catherine and Fried, Iris and Mawrin, Christian and Scheie, David and Mobley, Bret C and Schniederjan, Matthew J and Santi, Mariarita and Buccoliero, Anna M and Dahiya, Sonika and Kramm, Christof M and von Bueren, André O and von Hoff, Katja and Rutkowski, Stefan and Herold-Mende, Christel and Frühwald, Michael C and Milde, Till and Hasselblatt, Martin and Wesseling, Pieter and Rößler, Jochen and Schüller, Ulrich and Ebinger, Martin and Schittenhelm, Jens and Frank, Stephan and Grobholz, Rainer and Vajtai, Istvan and Hans, Volkmar and Schneppenheim, Reinhard and Zitterbart, Karel and Collins, V. Peter and Aronica, Eleonora and Varlet, Pascale and Puget, Stephanie and Dufour, Christelle and Grill, Jacques and Figarella-Branger, Dominique and Wolter, Marietta and Schuhmann, Martin U and Shalaby, Tarek and Grotzer, Michael and van Meter, Timothy and Monoranu, Camelia-Maria and Felsberg, Jörg and Reifenberger, Guido and Snuderl, Matija and Forrester, Lynn Ann and Koster, Jan and Versteeg, Rogier and Volckmann, Richard and van Sluis, Peter and Wolf, Stephan and Mikkelsen, Tom and Gajjar, Amar and Aldape, Kenneth and Moore, Andrew S and Taylor, Michael D and ...
Cell, ISSN 0092-8674, 02/2016, Volume 164, Issue 5, pp. 1060 - 1072
Primitive neuroectodermal tumors of the central nervous system (CNS-PNETs) are highly aggressive, poorly differentiated embryonal tumors occurring... 
PRIMITIVE NEUROECTODERMAL TUMORS | METHYLATION | AMPLIFICATION | DNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENOMIC ANALYSIS | CENTRAL-NERVOUS-SYSTEM | MUTATIONS | ABUNDANT NEUROPIL | MEDULLOBLASTOMA | TRUE ROSETTES | CELL BIOLOGY | Amino Acid Sequence | Central Nervous System Neoplasms - classification | Repressor Proteins - chemistry | Signal Transduction | Central Nervous System Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Repressor Proteins - genetics | Proto-Oncogene Proteins - genetics | Gene Expression Profiling | Proto-Oncogene Proteins - chemistry | Forkhead Transcription Factors - genetics | Neuroectodermal Tumors - diagnosis | Central Nervous System Neoplasms - pathology | DNA Methylation | Neuroectodermal Tumors - genetics | Tumor Suppressor Proteins - genetics | Neuroectodermal Tumors - classification | Neuroectodermal Tumors - pathology | Central Nervous System Neoplasms - diagnosis | Child | Brain tumors | Life Sciences | Neurons and Cognition | Neurobiology | Cancer
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6. Full Text Changes in the expression of genes related to neuroinflammation over the course of sporadic Alzheimer's disease progression: CX3CL1, TREM2, and PPAR gamma
by Strobel, S and Grunblatt, E and Riederer, P and Heinsen, H and Arzberger, T and Al-Sarraj, S and Troakes, C and Ferrer, I and Monoranu, CM
JOURNAL OF NEURAL TRANSMISSION, ISSN 0300-9564, 07/2015, Volume 122, Issue 7, pp. 1069 - 1076
The role of neuroinflammation in the pathogenesis of neurodegenerative diseases has become more evident in recent years. Research on the etiology and... 
VAGUS NERVE | FRACTALKINE | CLEARANCE | Sporadic Alzheimer's disease | CHEMOKINES | Neuroinflammation | BRAIN-STEM | TREM2 | PLAQUES | MILD COGNITIVE IMPAIRMENT | PPAR gamma | NEUROSCIENCES | CX3CL1 | FAR-FIELD POTENTIALS | CLINICAL NEUROLOGY | LIGANDS | RECEPTORS | Membrane Glycoproteins - metabolism | Humans | Middle Aged | Male | Chemokines, CXC - genetics | PPAR gamma - metabolism | Case-Control Studies | Disease Progression | Alzheimer Disease - pathology | Membrane Glycoproteins - genetics | Brain - metabolism | Analysis of Variance | Gene Expression - physiology | Alzheimer Disease - metabolism | Chemokines, CXC - metabolism | Aged, 80 and over | Brain - pathology | Female | Aged | Alzheimer Disease - genetics | Receptors, Immunologic - genetics | RNA, Messenger | Receptors, Immunologic - metabolism | PPAR gamma - genetics
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7. Full Text Comparison of the amino acid tracers 18F-FET and 18F-DOPA in high-grade glioma patients
by Lapa, Constantin and Linsenmann, Thomas and Monoranu, Camelia Maria and Samnick, Samuel and Buck, Andreas K and Bluemel, Christina and Czernin, Johannes and Kessler, Almuth F and Homola, Gyoergy A and Ernestus, Ralf-Ingo and Löhr, Mario and Herrmann, Ken
Journal of Nuclear Medicine, ISSN 0161-5505, 10/2014, Volume 55, Issue 10, pp. 1611 - 1616
High-grade gliomas (HGGs) are the most common malignant primary tumors of the central nervous system. PET probes of amino acid transport such as... 
Amino acids | FET | Positron emission tomography | F-DOPA | PET | Glioblastoma | Recurrence | Basal Ganglia - diagnostic imaging | Brain - diagnostic imaging | Prospective Studies | Brain Neoplasms - diagnostic imaging | Humans | Middle Aged | Dihydroxyphenylalanine - analogs & derivatives | Logistic Models | Male | Dihydroxyphenylalanine - pharmacokinetics | Tyrosine - analogs & derivatives | Glioma - diagnostic imaging | Biopsy | Radionuclide Imaging | Aged, 80 and over | Amino Acids - pharmacokinetics | Adult | Female | Aged | Tyrosine - pharmacokinetics
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8. Full Text BAFF is produced by astrocytes and up-regulated in multiple sclerosis lesions and primary central nervous system lymphoma
by Krumbholz, Markus and Theil, Diethilde and Derfuss, Tobias and Rosenwald, Andreas and Schrader, Frank and Monoranu, Camelia-Maria and Kalled, Susan L and Hess, Donna M and Serafini, Barbara and Aloisi, Francesca and Wekerle, Hartmut and Hohlfeld, Reinhard and Meinl, Edgar
Journal of Experimental Medicine, ISSN 0022-1007, 01/2005, Volume 201, Issue 2, pp. 195 - 200
We report that B cell-activating factor of the tumor necrosis factor (TNF) family (BAFF) is expressed in the normal human brain at similar to10% of that in... 
MEDICINE, RESEARCH & EXPERIMENTAL | MENINGES | HOMEOSTASIS | B-CELL | CELL-ACTIVATING FACTOR | CLONAL EXPANSION | T-CELL | IMMUNOLOGY | CEREBROSPINAL-FLUID | RECEPTORS | EXPRESSION | BRAIN | Receptors, Tumor Necrosis Factor - metabolism | Tumor Necrosis Factor-alpha - metabolism | Central Nervous System Neoplasms - metabolism | Up-Regulation | Membrane Proteins - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | B-Lymphocytes | Organ Specificity | RNA, Messenger - metabolism | B-Cell Activation Factor Receptor | Brain - metabolism | Lymphoma - metabolism | B-Cell Activating Factor | Membrane Proteins - metabolism | Astrocytes - metabolism | Multiple Sclerosis - metabolism | Brief Definitive Report
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9. Full Text Differential Alterations in Metabolism and Proteolysis-Related Proteins in Human Parkinson’s Disease Substantia Nigra
by Grünblatt, Edna and Ruder, Josefine and Monoranu, Camelia Maria and Riederer, Peter and Youdim, Moussa BH and Mandel, Silvia A
Neurotoxicity Research, ISSN 1029-8428, 4/2018, Volume 33, Issue 3, pp. 560 - 568
Parkinson’s disease is the most common neurodegenerative disorder after Alzheimer’s disease, with the majority of cases being sporadic or “idiopathic”. The... 
Neurochemistry | Immunohistochemistry | Neurosciences | Substantia nigra | Neurobiology | HSC70 | Cell Biology | Neurology | Biomedicine | PSMC4 | ALDH1A1 | Pharmacology/Toxicology | SKP1A | Parkinson’s disease
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10. Full Text Expression of D-amino acid oxidase (DAO/DAAO) and D-amino acid oxidase activator (DAOA/G72) during development and aging in the human post-mortem brain
by Jagannath, Vinita and Marinova, Zoya and Monoranu, Camelia-Maria and Walitza, Susanne and Grünblatt, Edna
Frontiers in Neuroanatomy, ISSN 1662-5129, 04/2017, Volume 11, p. 31
In the brain, D-amino acid oxidase (DAO/DAAO) mainly oxidizes D-serine, a co-agonist of the N-methyl-D-aspartate (NMDA) receptors. Thus, DAO can regulate the... 
Human post-mortem brain regions | DNA methylation | DAO/DAAO | DAOA/G72 | Gene and protein expression | GENE G72 | ANATOMY & MORPHOLOGY | SCHIZOPHRENIA | QUANTITATIVE PCR DATA | NEUROSCIENCES | D-SERINE | CORTEX | gene and protein expression | DISORDER | G72/G30 | ASSOCIATION | BINDING | human post-mortem brain regions | Oxidases | Aging | Brain research | Research | Attention deficit disorder | Cerebellum | Brain | Post-transcription | Neurosciences | Amygdala | Mental disorders | Working groups | Glutamic acid receptors (ionotropic) | Gene regulation | Genomics | Schizophrenia | Reverse transcription | Amino acids | Genomes | Glutamic acid receptors | Gestation | Proteins | Consortia | Autopsy | Cortex (frontal) | Amino acid oxidase | Enzyme-linked immunosorbent assay | Deoxyribonucleic acid--DNA | CpG islands | D-Serine | D-Amino-acid oxidase | N-Methyl-D-aspartic acid receptors | Gene expression | Polymerase chain reaction | Life span | Epigenetics | Protein expression | Receptor mechanisms | Methylation | Age composition | Child & adolescent psychiatry
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11. Full Text Post-mortem cerebrospinal fluid diagnostics: cytology and immunocytochemistry method suitable for routine use to interpret pathological processes in the central nervous system
by Bohnert, Simone and Ondruschka, Benjamin and Bohnert, Michael and Schuhmann, Michael K and Monoranu, Camelia-Maria
International Journal of Legal Medicine, ISSN 0937-9827, 7/2019, Volume 133, Issue 4, pp. 1141 - 1146
Due to its protected anatomical location, cerebrospinal fluid (CSF) is a very stable fluid which undergoes comparatively little change in the early post-mortem... 
Forensic Medicine | Medicine & Public Health | Immunocytochemistry | CSF | Forensic neuropathology | Cytology | Medical Law | Cerebrospinal fluid | Medicine/Public Health, general | Forensic neurotraumatology | NSE | TRAUMATIC BRAIN-INJURY | MEDICINE, LEGAL | DEATH | TIME-COURSE | S100B | Cytochemistry | Analysis | Methods | Central nervous system
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12. Full Text Astrocyte- and Microglia-Specific Mitochondrial DNA Deletions Levels in Sporadic Alzheimer’s Disease
by Strobel, Sabrina and Grünblatt, Edna and Heinsen, Helmut and Riederer, Peter and Espach, Thomas and Meder, Michael and Monoranu, Camelia-Maria
Journal of Alzheimer's Disease, ISSN 1387-2877, 01/2019, Volume 67, Issue 1, pp. 149 - 157
Oxidative stress is implicated in the pathogenesis of neurodegenerative diseases, including sporadic Alzheimer’s disease (AD). Mitochondrial DNA (mtDNA)... 
Cerebellum | Brain | Oxidative stress | Disease | Neurodegenerative diseases | Astrocytes | Pathogenesis | Brain stem | Mitochondrial DNA | Disease control | Microglia | Neurological diseases | Clonal deletion | Correlation analysis | Deletion | Damage accumulation | Alzheimer's disease | Age | Hippocampus | Deoxyribonucleic acid--DNA
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13. Full Text O-(2-(18F)fluoroethyl)-L-tyrosine PET for the differentiation of tumour recurrence from late pseudoprogression in glioblastoma
by Mihovilovic, Milena I and Kertels, Olivia and Hänscheid, Heribert and Löhr, Mario and Monoranu, Camelia-Maria and Kleinlein, Irene and Samnick, Samuel and Kessler, Almuth F and Linsenmann, Thomas and Ernestus, Ralf-Ingo and Buck, Andreas K and Lapa, Constantin
Journal of Neurology, Neurosurgery & Psychiatry, ISSN 0022-3050, 02/2019, Volume 90, Issue 2, pp. 238 - 239
Typically understood to occur within the first 12 weeks following radiotherapy, PsP has recently been demonstrated to also appear significantly later (‘late... 
neurooncology | ligand studies | pet | Studies | Nuclear medicine | Histopathology | Brain research | Medical imaging | Working groups | Brain cancer | Tomography | Amino acids | Oncology | Radiation therapy | Patients
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14. Full Text Astrocyte- and Microglia-Specific Mitochondrial DNA Deletions Levels in Sporadic Alzheimer's Disease
by Strobel, Sabrina and Grünblatt, Edna and Heinsen, Helmut and Riederer, Peter and Espach, Thomas and Meder, Michael and Monoranu, Camelia-Maria
Journal of Alzheimer's Disease, ISSN 1387-2877, 2019, Volume 67, Issue 1, pp. 149 - 157
Oxidative stress is implicated in the pathogenesis of neurodegenerative diseases, including sporadic Alzheimer's disease (AD). Mitochondrial DNA (mtDNA)... 
hippocampus | mitochondrial DNA | brainstem | astrocytes | selective vulnerability | Alzheimer's disease | microglia | cerebellum | oxidative stress | VAGUS NERVE | CELLS | BRAIN-STEM | NEUROSCIENCES | NEURONS | DYSFUNCTION
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15. Full Text Region-specific regulation of the serotonin 2A receptor expression in development and ageing inpost mortemhuman brain
: Serotonin 2A receptor developmental and region-specific expression in brain
by Marinova, Zoya and Monoranu, Camelia-Maria and Fetz, Sonja and Walitza, Susanne and Grünblatt, Edna
Neuropathology and Applied Neurobiology, ISSN 0305-1846, 06/2015, Volume 41, Issue 4, pp. 520 - 532
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