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The New England Journal of Medicine, ISSN 0028-4793, 11/2003, Volume 349, Issue 21, pp. 1993 - 2003
Journal Article
Tissue Engineering - Part A, ISSN 1937-3341, 11/2017, Volume 23, Issue 21-22, pp. 1251 - 1261
Journal Article
2013, 2013, ISBN 8847056322, 410
This volume presents a selection of contributions to the XXIV Villa Mondragone International Economic Seminar on "Public debt, global governance and economic... 
Economics | Debts, Public | International cooperation | International organization
eBook
Rambam Maimonides Medical Journal, ISSN 2076-9172, 01/2015, Volume 6, Issue 1, p. e0009
Journal Article
Economic Modelling, ISSN 0264-9993, 2019
This paper proposes a small-scale general equilibrium model of structural transformation with a non-agricultural labour market characterized by search... 
Structural transformation | Dual economies | Solow residual | Growth accounting | Non-agricultural unemployment | Matching frictions
Journal Article
1966, 1. ed., Novelistas contemporáneos, 235
Book
2001, 1. ed., ISBN 9681908066, 153
Book
PLoS ONE, ISSN 1932-6203, 10/2011, Volume 6, Issue 10, p. e25545
Background: Amyotrophic lateral sclerosis (ALS) is a fatal progressive motor neuron disease, for which there are still no diagnostic/prognostic test and... 
INCREASED 3-NITROTYROSINE | MOTOR-NEURONS | SKELETAL-MUSCLE | UNFOLDED PROTEIN RESPONSE | OXIDATIVE DAMAGE | HUMAN CEREBROSPINAL-FLUID | MULTIDISCIPLINARY SCIENCES | EXTEND SURVIVAL | ENDOPLASMIC-RETICULUM STRESS | PROTEOMIC ANALYSIS | TRANSGENIC MICE | Biomarkers - metabolism | Leukocytes, Mononuclear - metabolism | Reproducibility of Results | Animals | Humans | Translational Medical Research | Amyotrophic Lateral Sclerosis - metabolism | Proteomics | Rats | Proteome - metabolism | Disease Progression | Proteins | Medical research | Nervous system diseases | RNA | Analysis | Medicine, Experimental | Amyotrophic lateral sclerosis | Development and progression | Genetic engineering | Research | Biological markers | Spinal cord | Neurosciences | Change detection | Pathogenesis | Parkinsons disease | Transgenic | Superoxide dismutase | Leukocytes (mononuclear) | Biochemistry | Blood | Occupational safety | Motor neuron diseases | Neurodegeneration | Rodents | Peripheral blood mononuclear cells | Differential diagnosis | Trends | Diagnosis | RNA processing | Gel electrophoresis | Cystic fibrosis | Pharmacology | Metabolism | Ribonucleic acid--RNA | Patients | Neurological diseases | Correlation analysis | Biomarkers | Scientific imaging | Diagnostic systems | Human behavior | Endoplasmic reticulum | Mass spectrometry | Immunoassays | Ribonucleic acid
Journal Article
Experimental Cell Research, ISSN 0014-4827, 04/2014, Volume 322, Issue 2, pp. 313 - 323
Quinolinic acid (QUIN) is a glutamate agonist which markedly enhances the vulnerability of neural cells to excitotoxicity. QUIN is produced from the amino acid... 
Astrocyte, cell signaling | GFAP phosphorylation | Quinolinic acid | Cytoskeleton remodelling | ALZHEIMERS-DISEASE | DEPENDENT PROTEIN-KINASE | AMYOTROPHIC-LATERAL-SCLEROSIS | CORTICAL ASTROCYTES | CEREBRAL-CORTEX | INTERMEDIATE-FILAMENT PHOSPHORYLATION | CELL BIOLOGY | ONCOLOGY | DIPHENYL DITELLURIDE | THYROID-HORMONES | REACTIVE ASTROCYTES | YOUNG-RATS | Astrocytes - cytology | Astrocytes - drug effects | Actin Cytoskeleton - metabolism | Rats, Wistar | Apoptosis - drug effects | Calcium - metabolism | Vimentin - metabolism | Cells, Cultured | Rats | Glutamates - metabolism | Glial Fibrillary Acidic Protein - metabolism | Corpus Striatum - cytology | Corpus Striatum - metabolism | Blotting, Western | Immunoenzyme Techniques | Pregnancy | Quinolinic Acid - pharmacology | Animals | Actin Cytoskeleton - drug effects | Corpus Striatum - drug effects | Female | Cell Proliferation - drug effects | Phosphorylation - drug effects | Astrocytes - metabolism | Methyl aspartate | Calcium channels | Analysis | Tryptophan | Calcium-binding proteins | Glutamate | Intermediate filament proteins | Cytoskeleton | Phosphorylation | Neurotoxicity | Neurological disorders | Index Medicus | KYNURENINE | TRYPTOPHAN | ACTIN | MICROTUBULES | PHOSPHORYLATION | CALCIUM IONS | INCUBATION | 60 APPLIED LIFE SCIENCES | EGTA | CALMODULIN | CYTOPLASM | PHOSPHATES | PHOSPHORUS 32 | RECEPTORS | MORPHOLOGY
Journal Article
1979, Colección de arte (Universidad Nacional Autónoma de México. Coordinación de Humanidades), ISBN 9685825564, Volume 35, 82
Book
BMC Cancer, ISSN 1471-2407, 07/2011, Volume 11, Issue 1, pp. 296 - 296
Background: Systemic therapy for cancer metastatic lesions is difficult and generally renders a poor clinical response. Structural analogs of cisplatin, the... 
PERMEABILITY TRANSITION PORE | PALLADIUM(II) COMPLEXES | ONCOLOGY | CALCIUM | BAX | ENDOPLASMIC-RETICULUM | DEATH | ANTITUMOR-ACTIVITY | PROTEINS | CYTOTOXICITY | CANCER-THERAPY | Rats, Wistar | Apoptosis - drug effects | Mitochondria, Liver - metabolism | Humans | Lung Neoplasms - metabolism | Melanoma, Experimental - drug therapy | Drug Resistance, Neoplasm | Male | Melanoma, Experimental - metabolism | Membrane Potential, Mitochondrial - drug effects | Mitochondria, Liver - physiology | Antineoplastic Agents - metabolism | Mitochondrial Proteins - drug effects | Sulfhydryl Compounds - metabolism | Caspases - metabolism | Lung Neoplasms - secondary | Mitochondrial Proteins - metabolism | Antineoplastic Agents - pharmacology | Molecular Structure | Mice, Inbred C57BL | Palladium - pharmacology | bcl-2-Associated X Protein - metabolism | Melanoma, Experimental - pathology | Rats | Cisplatin - pharmacology | Antineoplastic Agents - chemistry | Enzyme Activation - drug effects | Mitochondrial Membranes - metabolism | Mitochondria, Liver - drug effects | Animals | Lung Neoplasms - prevention & control | Organometallic Compounds - metabolism | Cell Line, Tumor | Palladium - metabolism | Organometallic Compounds - chemistry | Mice | Palladium - chemistry | Organometallic Compounds - pharmacology | Melanoma | Physiological aspects | Genetic aspects | Mitochondrial DNA | Research | Metastasis | Apoptosis
Journal Article
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 08/2014, Volume 40, Issue 3, pp. 2471 - 2478
Although the accumulation of the neurotoxic peptide β‐amyloid (Aβ) in the central nervous system is a hallmark of Alzheimer's disease, whether Aβ acts in... 
Bax | amyloid‐beta peptide | apoptosis | Alzheimer's disease | endoplasmic reticulum | calcium signaling | Calcium signaling | Endoplasmic reticulum | Amyloid-beta peptide | Apoptosis
Journal Article