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by Yamagami, Keitaro and Kurogi, Ryota and Kurogi, Ai and Nishimura, Hiroyuki and Nishimura, Shinjitsu and Nishimura, Yasuaki and Nishimura, Kunihiro and Nishimura, Ataru and Onozuka, Daisuke and Ren, Nice and Kada, Akiko and Arimura, Koichi and Ido, Keisuke and Mizoguchi, Masahiro and Sakamoto, Seisaburo and Sakamoto, Tetsuya and Kayama, Takamasa and Suzuki, Nozomi and Suzuki, Michiyasu and Suzuki, Sachio and Suzuki, Hidenori and Suzuki, Ichiro and Suzuki, Susumu and Arai, Yoshinori and Arai, Hajime and Arai, Motohiro and Hagihara, Akihito and Iihara, Koji and Takigami, Masayoshi and Kamiyama, Kenji and Houkin, Kiyohiro and Nishi, Shougo and Yoshimoto, Tetsuyuki and Yoshimoto, Junpei and Kaneko, Sadao and Oka, Hirofumi and Oka, Koji and Ooyama, Hiroshi and Kamada, Kyousuke and Makino, Kenichi and Tokumitsu, Naoki and Sako, Kazuhiro and Izumi, Naoto and Nitta, Hisashi and Nitta, Kazumi and Ootaki, Masahumi and Isobe, Masanori and Nishiya, Mikio and Yamazaki, Takaaki and Mabuchi, Syouji and Mabuchi, Eiichiro and Ogasawara, Kuniaki and Kubo, Naohiko and Shimizu, Yukihiko and Saito, Hitoshi and Saito, Keiichi and Yamanome, Tatumi and Yoshino, Kimihiro and Yoshino, Atsuo and Fujitsuka, Mitsuyuki and Takami, Masaaki and Ohtaka, Hirotoshi and Hirano, Teruyuki and Shiokawa, Yosiaki and Okada, Takaharu and Kohno, Michihiro and Haraoka, Jou and Kawamura, Noriyoshi and Kawamura, Tadao and Isoshima, Akira and Yasue, Masaharu and Takayoshi Kobayashi, Mitsuhiko Hokari and Kawai, Kensuke and Maehara, Taketoshi and Noguchi, Makoto and Hoshino, Haruhiko and Hiyama, Hirofumi and Yoshida, Kensaku and Yoshida, Kazunari and Utsugi, Osamu and Takeda, Yasuaki and Takeda, Yuu and Tamaki, Kouichi and Karasudani, Hirohide and Urabe, Takao and Kobayashi, Nozomu and Kobayashi, Shiro and Nakamura, Yusaku and Nakamura, Kazuhito and Nakamura, Michio and Nakamura, Yoshinari and Koguchi, Yorio and Ono, Junichi and Suda, Sumio and Hadeishi, Hiromu and Fukutake, Toshio and Wakui, Kenji and Tanno, Hirokazu and Ishige, Naoki and Ohasi, Takashi and ... and J-ASPECT Study Collaborators
World Neurosurgery, ISSN 1878-8750, 10/2019, Volume 130, pp. e26 - e46
The epidemiology of patients with traumatic brain injury (TBI) has changed dramatically over recent decades as a result of rapid advances in aging societies.... 
Aging | Hospital mortality | Prognosis | Traumatic brain injury | Subdural hematoma
Journal Article
Journal Article
International Immunology, ISSN 0953-8178, 11/2011, Volume 23, Issue 11, pp. 701 - 712
Rheumatoid arthritis (RA) is a chronic inflammatory disease that causes irreversible joint damage and significant disability. However, the fundamental... 
Joint destruction | Chronic inflammation | Collagen-induced arthritis | Rheumatoid arthritis | IL-6 | RHEUMATOID-ARTHRITIS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | STAT3 | MONOCLONAL-ANTIBODY | IMMUNOLOGY | rheumatoid arthritis | OSTEOCLASTOGENESIS | RODENT MODELS | TUMOR-NECROSIS-FACTOR | collagen-induced arthritis | REGULATORY T-CELLS | DOUBLE-BLIND | joint destruction | NF-KAPPA-B | chronic inflammation | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Arthritis, Experimental - drug therapy | RANK Ligand - metabolism | Humans | Arthritis, Rheumatoid - complications | Interleukin-1beta - adverse effects | Arthritis, Experimental - pathology | RANK Ligand - immunology | Inflammation - complications | Signal Transduction - immunology | Osteoclasts - immunology | Arthritis, Rheumatoid - drug therapy | Inflammation - drug therapy | Interleukin-1beta - metabolism | Piperidines | Gene Expression - immunology | STAT3 Transcription Factor - deficiency | Tumor Necrosis Factor-alpha - immunology | Mice, Inbred DBA | Pyrroles - therapeutic use | Interleukin-6 - metabolism | Joints - metabolism | STAT3 Transcription Factor - genetics | Joints - drug effects | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Interleukin-1beta - immunology | Joints - pathology | Arthritis, Experimental - immunology | Inflammation - immunology | Pyrimidines - pharmacology | Arthritis, Rheumatoid - pathology | Pyrroles - pharmacology | Animals | Tumor Necrosis Factor-alpha - adverse effects | Interleukin-6 - pharmacology | RANK Ligand - antagonists & inhibitors | Pyrimidines - therapeutic use | Fibroblasts - drug effects | Interleukin-6 - immunology | Fibroblasts - immunology | Mice | STAT3 Transcription Factor - immunology | Arthritis, Rheumatoid - immunology | STAT3 Transcription Factor - antagonists & inhibitors | Chronic Disease | Osteoclasts - drug effects | Animal models | Transcription | Leukemia inhibitory factor | Stat3 protein | Data processing | Inflammation | Joint diseases | Osteoblasts | Inflammatory diseases | Interleukin 6 | Osteoclastogenesis | Collagen | Interleukin 1 | TRANCE protein | Tumor necrosis factor- alpha
Journal Article
Journal of Orthopaedic Science, ISSN 0949-2658, 09/2010, Volume 15, Issue 5, pp. 654 - 660
Journal Article
BMC Musculoskeletal Disorders, ISSN 1471-2474, 12/2019, Volume 20, Issue 1
Journal Article
BMC MUSCULOSKELETAL DISORDERS, ISSN 1471-2474, 07/2019, Volume 20, Issue 1, pp. 345 - 5
BackgroundSuperior shoulder dislocation is a rare type of shoulder dislocation. Its occurrence is thought to be associated with rupture of the deltoid;... 
Rotator cuff tear | Superior shoulder dislocation | Deltoid rupture | SUBLUXATION | RHEUMATOLOGY | ORTHOPEDICS | Conservative treatment | Case studies | Usage | Shoulder joint | Care and treatment | Magnetic resonance imaging | Diagnosis | Dislocation
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 41, pp. 16568 - 16573
Journal Article
Cancer Science, ISSN 1347-9032, 12/2014, Volume 105, Issue 12, pp. 1550 - 1559
Angiopoietin‐like protein 2 ( ANGPTL 2) plays an important role in inflammatory carcinogenesis and tumor metastasis by activating tumor angiogenesis and tumor... 
ANGPTL | apoptosis | chemoresistance | BCL | BCL-2 | ANGPTL2 | Syk | Chemoresistance | Apoptosis | STEM-CELLS | DRUG-RESISTANCE | FLUOROURACIL | P53 | FAMILY | 5-FLUOROURACIL | ONCOLOGY | INFLAMMATION | BAX | MODULATION | Angiopoietins - metabolism | Signal Transduction | Apoptosis - drug effects | Protein-Tyrosine Kinases - metabolism | Humans | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Intracellular Signaling Peptides and Proteins - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Sequence Analysis, RNA | Angiopoietin-like Proteins | Colorectal Neoplasms - drug therapy | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Angiopoietins - genetics | Colorectal Neoplasms - pathology | Syk Kinase | Colorectal Neoplasms - metabolism | Transcription factors | Angiopoietin | Colorectal carcinoma | Colorectal cancer | Cytotoxicity | NF-AT protein | Kinases | Drug resistance | Cancer therapies | Carcinogenesis | Metastases | Proteins | Angiogenesis | Protein-tyrosine kinase | Polyclonal antibodies | Spleen | Tyrosine | Medical research | Antineoplastic drugs | Cell survival | Tumor cells | Invasiveness | Inflammation | Tumor cell lines | Ribonucleic acid--RNA | Chemotaxis | 1-Phosphatidylinositol 3-kinase | Chemotherapy | Cell lines | Syk protein | Tumors | Cancer | Index Medicus | Original
Journal Article