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PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 8, pp. e72301 - e72301
Progressive aggregation of protein Tau into oligomers and fibrils correlates with cognitive decline and synaptic dysfunction, leading to neurodegeneration in... 
NEUROFIBRILLARY TANGLES | IMMUNOTHERAPY | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | AMYLOID-BETA-PEPTIDE | MOUSE MODEL | PATHOLOGY | CONFORMATION | PASSIVE-IMMUNIZATION | BRAIN | DEGENERATION | Phosphoproteins - immunology | Phosphorylation | Tauopathies - immunology | tau Proteins - immunology | Humans | Vaccination | Peptides - administration & dosage | Phosphoproteins - chemical synthesis | Peptides - chemical synthesis | tau Proteins - genetics | Tauopathies - physiopathology | tau Proteins - antagonists & inhibitors | Alzheimer Vaccines - immunology | Disease Models, Animal | Brain - physiopathology | Peptides - immunology | Alzheimer Vaccines - administration & dosage | Mice, Transgenic | Treatment Outcome | Psychomotor Performance - drug effects | Brain - drug effects | Liposomes - chemistry | Animals | Mice | Tauopathies - drug therapy | Brain - immunology | Antibodies, Neutralizing - blood | Phosphoproteins - administration & dosage | Oligomers | Medical research | Peptides | Medicine, Experimental | Disease susceptibility | Vaccines | Alzheimer's disease | Antigenic determinants | Brain | Neurosciences | Cognitive ability | Clinical trials | Lipids | Antisera | Proteins | Neurodegeneration | Transgenic animals | Immunotherapy | Rodents | Genetics | Neurodegenerative diseases | Medical treatment | Fibrils | Pharmacology | Pathology | Tau protein | Liposomes | Mutation | Alzheimers disease | Animal cognition | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 11/2013, Volume 110, Issue 46, pp. E4369 - E4374
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 13556 - 18
Tau pathology is associated with cognitive decline in Alzheimer's disease, and missense tau mutations cause frontotemporal dementia. Hyperphosphorylation and... 
PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | TANGLES | PAIRED HELICAL FILAMENTS | NEURONS | TAUOPATHY | MUTATIONS | MODEL | DEFICITS | EXPRESSION | SEVERITY | Phosphorylation | Vinblastine | Neurodegenerative diseases | Cognitive ability | Forebrain | Proline | Pathology | Tau protein | Microtubules | Dementia disorders | Cytoskeleton | Mutation | Frontotemporal dementia | Dementia | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 10, pp. 8585 - 8596
The identification of toxic Aβ species and/or the process of their formation is crucial for understanding the mechanism(s) of Aβ neurotoxicity in Alzheimer... 
Oligomers | Molecular Bases of Disease | Protein Self-assembly | Neurodegeneration | Cell Death | Amyloid | Alzheimer Disease | Nucleated Polymerization
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 1 - 15
The aggregation of intracellular tau protein is a major hallmark of Alzheimer’s disease (AD). The extent and the stereotypical spread of tau pathology in the... 
Brain | Oligomerization | Tau protein | Site selection | Proteolysis | Edman degradation | Cognitive ability | Mass spectroscopy | Calpain | Alzheimer's disease
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2007, Volume 104, Issue 23, pp. 9810 - 9815
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, pp. e0152471 - e0152471
In Down syndrome (DS) or trisomy of chromosome 21, the beta-amyloid (A beta) peptide product of the amyloid precursor protein (APP) is present in excess.... 
PATHOGENESIS | IMMUNIZATION | TRANSPORT | DEMENTIA | ABNORMALITIES | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | RECEPTOR | NEURODEGENERATION | PEPTIDE | TRANSGENIC MICE | Inflammation - pathology | Antibodies - metabolism | Memory | Septal Nuclei - pathology | Vaccination | Male | Vaccines - therapeutic use | Atrophy | Behavior, Animal | Brain - metabolism | Amyloid beta-Peptides - genetics | Down Syndrome - complications | Disease Models, Animal | Animals, Newborn | Biomarkers - metabolism | Cholinergic Neurons - metabolism | Gene Expression Regulation | Mice, Transgenic | Cognition Disorders - drug therapy | Cognition Disorders - complications | Animals | Brain - pathology | Down Syndrome - drug therapy | Amyloid beta-Peptides - immunology | Hemorrhage - pathology | Brain | Neurosciences | Neuropathology | Pathogenesis | Immunoglobulin G | Medical services | Cognitive ability | Forebrain | Antibodies | Vaccines | Hemorrhage | Lipid A | Reduction | Down's syndrome | Neurodegeneration | Proteolysis | Rodents | Immunotherapy | Dementia disorders | Monophosphoryl lipid A | Drug dosages | Alzheimer's disease | Age | Immunization | Immunological memory | Neurodegenerative diseases | Medical treatment | Pattern recognition | Down syndrome | Object recognition | Forebrain (basal) | Immunological tolerance | Amyloid precursor protein | Fear conditioning | Medicine | Neurology | Chromosome 21 | β-Amyloid | Cholinergic nerves | Mice | Infiltration | Liposomes | Alzheimers disease | Dementia | Index Medicus
Journal Article