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Neuropharmacology, ISSN 0028-3908, 11/2016, Volume 110, p. 251
Depressive disorders cause a substantial burden for the individual and the society. Key depressive symptoms can be modeled in animals and enable the... 
Phenols | Neurosciences | Cyclic adenylic acid | Antidepressants | Depression, Mental | Stress (Psychology) | Neurons
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 04/2016, Volume 41, Issue 5, p. 1416
  Cannabis is the most commonly used illicit drug worldwide, and use is typically initiated during adolescence. The endocannabinoid system has an important... 
Journal Article
CNS Neuroscience & Therapeutics, ISSN 1755-5930, 05/2019, Volume 25, Issue 5, p. 549
Prenatal maternal immune activation (MIA) is associated with a risk to develop schizophrenia and affects dopamine systems in the ventral tegmental area (VTA),... 
Fenofibrate | Neurons | Schizophrenia | Phenols | Missing in action | Neurophysiology
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 04/2012, Volume 37, Issue 5, pp. 1164 - 1176
The rostromedial tegmental nucleus (RMTg), a structure located just posterior to the ventral tegmental area (VTA), is an important site involved in aversion... 
cannabinoids | patch clamp | electrophysiology | rat | morphine | GABAERGIC TAIL | NEGATIVE REWARD SIGNALS | AVERSIVE STIMULI | PSYCHIATRY | SYNAPTIC-TRANSMISSION | LATERAL HABENULA | SUBSTANTIA-NIGRA | CANNABINOID CB1 | NEUROSCIENCES | IN-VITRO | AREA | PHARMACOLOGY & PHARMACY | OPIOID RECEPTORS | Electric Stimulation | Cannabinoids - antagonists & inhibitors | Male | Ventral Tegmental Area - cytology | Neural Pathways - physiology | Nicotine - pharmacology | Neural Inhibition - physiology | Piperidines - pharmacology | Time Factors | Dopaminergic Neurons - drug effects | Dopaminergic Neurons - physiology | Action Potentials - drug effects | Nicotinic Agonists - pharmacology | Pyrazoles - pharmacology | Physical Stimulation | Morphine - pharmacology | Tegmentum Mesencephali - cytology | Morpholines - pharmacology | Rats | Rats, Sprague-Dawley | Naphthalenes - pharmacology | Action Potentials - physiology | Cocaine - pharmacology | Patch-Clamp Techniques | Animals | Analysis of Variance | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Benzoxazines - pharmacology | Cannabinoids - agonists | Narcotics - pharmacology | In Vitro Techniques | Neural Inhibition - drug effects | Inhibitory Postsynaptic Potentials - drug effects | cannabinoid | neuropharmacology | addiction & substance abuse | neurophysiology | Original | dopamine
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 02/2011, Volume 36, Issue 3, pp. 589 - 602
Journal Article
Psychopharmacology, ISSN 0033-3158, 6/2016, Volume 233, Issue 12, pp. 2241 - 2251
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2008, Volume 28, Issue 51, pp. 13985 - 13994
Nicotine stimulates the activity of mesolimbic dopamine neurons, which is believed to mediate the rewarding and addictive properties of tobacco use.... 
Peroxisome proliferator-activated receptor | Electrophysiology | Fatty acid amide hydrolase | Patch clamp | Dopamine neurons | Endocannabinoids | Nicotine | PPAR-ALPHA | nicotine | patch clamp | ANANDAMIDE HYDROLYSIS | NEUROSCIENCES | MOUSE THALAMIC SYNAPTOSOMES | VENTRAL TEGMENTAL AREA | AMIDE HYDROLASE | dopamine neurons | ACETYLCHOLINE-RECEPTORS | fatty acid amide hydrolase | ESTER URB597 | electrophysiology | CORTICAL-NEURONS | endocannabinoids | INFLAMMATORY PAIN | peroxisome proliferator-activated receptor | CB1 RECEPTORS | PPAR alpha - drug effects | Cannabinoid Receptor Antagonists | Protein-Tyrosine Kinases - metabolism | Injections, Intraventricular | Male | Ventral Tegmental Area - cytology | Nicotine - pharmacology | Receptors, Cannabinoid - metabolism | Piperidines - pharmacology | Benzamides - pharmacology | Neurons - metabolism | Oleic Acids - pharmacology | Palmitic Acids - pharmacology | Neurons - drug effects | Dopamine - metabolism | Organ Culture Techniques | Carbamates - pharmacology | Pyrazoles - pharmacology | Amidohydrolases - metabolism | Ventral Tegmental Area - drug effects | Appetite Depressants - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Amidohydrolases - antagonists & inhibitors | Enzyme Activation - drug effects | Ethanolamines | Nicotine - antagonists & inhibitors | Rats, Sprague-Dawley | Arachidonic Acids - pharmacology | Patch-Clamp Techniques | Animals | Rimonabant | Ventral Tegmental Area - metabolism | PPAR alpha - metabolism | Lipoxygenase Inhibitors - pharmacology
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 04/2016, Volume 41, Issue 5, pp. 1416 - 1426
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 06/2002, Volume 15, Issue 11, pp. 1795 - 1802
The nucleus accumbens (NAc) represents a critical site for the rewarding properties of diverse classes of drugs of abuse. Glutamatergic afferents to the NAc... 
medium spiny neurons | medial prefrontal cortex | baso‐lateral amygdala | electrophysiology | rat | Electrophysiology | Medial prefrontal cortex | Rat | Medium spiny neurons | Baso-lateral amygdala | AMYGDALOID PROJECTIONS | STIMULATION | NEUROSCIENCES | DOPAMINE NEURONS | FIMBRIA INPUT | MORPHINE-DEPENDENT RATS | baso-lateral amygdala | SINGLE-UNIT RESPONSES | OPIOID RECEPTORS | SYNAPTIC TRANSMISSION | BRAIN | CB1 RECEPTORS | Analgesics - pharmacology | Amygdala - drug effects | Electric Stimulation | Neural Pathways - drug effects | Synaptic Transmission - physiology | Nucleus Accumbens - metabolism | Male | Benzoxazines | Dose-Response Relationship, Drug | Neural Inhibition - physiology | Prefrontal Cortex - drug effects | Piperidines - pharmacology | Dronabinol - analogs & derivatives | Nucleus Accumbens - cytology | Synaptic Transmission - drug effects | Dronabinol - pharmacology | Action Potentials - drug effects | Dopamine - metabolism | Pyrazoles - pharmacology | Amygdala - metabolism | Morpholines - pharmacology | Rats | Amygdala - cytology | Prefrontal Cortex - cytology | Rats, Sprague-Dawley | Receptors, Drug - metabolism | Cannabinoids - pharmacology | Naphthalenes - pharmacology | Action Potentials - physiology | Animals | Receptors, Drug - drug effects | Rimonabant | Dopamine Antagonists - pharmacology | Narcotic Antagonists - pharmacology | Neural Pathways - cytology | Prefrontal Cortex - metabolism | Neural Pathways - metabolism | Receptors, Cannabinoid | Glutamic Acid - metabolism | Nucleus Accumbens - drug effects | Neural Inhibition - drug effects
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2004, Volume 24, Issue 1, pp. 53 - 62
Journal Article
Journal Article
Epilepsia, ISSN 0013-9580, 10/2017, Volume 58, Issue 10, pp. 1762 - 1770
Summary Objective Nocturnal frontal lobe epilepsy (NFLE) is an idiopathic partial epilepsy with a family history in about 25% of cases, with autosomal dominant... 
Fenofibrate | Neuronal nicotinic acetylcholine receptors | Nocturnal frontal lobe epilepsy | Peroxisome proliferator‐activated receptor alpha | Peroxisome proliferator-activated receptor alpha | DEFINITION | DEPRESSION | SLEEP | AROUSAL | NICOTINIC ACETYLCHOLINE-RECEPTORS | CLINICAL NEUROLOGY | MINOR MOTOR EVENTS | SEIZURES | HYPOTHESIS | MICE | HEALTH | Epilepsy, Frontal Lobe - genetics | Triazines - therapeutic use | Humans | Middle Aged | Male | Electroencephalography | Anticonvulsants - pharmacology | Fenofibrate - therapeutic use | Drug Resistant Epilepsy - drug therapy | Young Adult | Fenofibrate - pharmacology | Piracetam - therapeutic use | Drug Resistant Epilepsy - genetics | Adult | Female | Valproic Acid - therapeutic use | Drug Therapy, Combination | Epilepsy, Frontal Lobe - drug therapy | Disease Models, Animal | Carbamazepine - analogs & derivatives | Carbamazepine - therapeutic use | Mice, Transgenic | Polysomnography | Animals | Piracetam - analogs & derivatives | PPAR alpha - agonists | Mice | Mutation | Benzodiazepines - therapeutic use | Receptors, Nicotinic - genetics | Brain | Epilepsy | Genetic engineering | Frontal lobe | Cortex | Transgenic mice | Heredity | Antiepileptic agents | Motor task performance | Sleep | Pyramidal cells | Rodents | Acetylcholine receptors (nicotinic) | Peroxisome proliferator-activated receptors | Receptor mechanisms | Genotypes | Nicotine | Seizures | Inhibitory postsynaptic potentials
Journal Article