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PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e18734
Background: Dendritic cells (DC) and regulatory cells (Treg) play pivotal roles in controlling both normal and autoimmune adaptive immune responses. DC are the... 
WEGENERS-GRANULOMATOSIS | RHEUMATOID-ARTHRITIS | AUTOANTIBODIES | SYSTEMIC-LUPUS-ERYTHEMATOSUS | MULTIPLE-SCLEROSIS | IMMUNE SUPPRESSION | MULTIDISCIPLINARY SCIENCES | SUPPRESSOR FUNCTION | ANCA-ASSOCIATED VASCULITIS | EXPRESSION | TH17 CELLS | Acute Disease | Cell Count | Coculture Techniques | Humans | Middle Aged | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - physiopathology | Dendritic Cells - pathology | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - blood | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - pathology | Remission Induction | Case-Control Studies | T-Lymphocytes, Regulatory - pathology | L-Selectin - metabolism | Interleukin-17 - metabolism | Flow Cytometry | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - immunology | Th17 Cells - metabolism | Viral antibodies | Care and treatment | Vasculitis | Dendritic cells | Analysis | Antibodies | T cells | Statistics | Cell culture | Flow cytometry | Multiple sclerosis | Transplants & implants | Disease | Pathogenesis | Helper cells | Lymphocytes T | Arthritis | Blood | Lymphocytes | Flares | Antineutrophil cytoplasmic antibodies | CD25 antigen | Population | Foxp3 protein | Remission | Growth factors | Lupus | Antigens | Immune response | Immunoregulation | CD62L protein | T cell receptors | Patients | CD4 antigen | Cytometry | Immunosuppression | Antigen-presenting cells | Diabetes | Autoimmune diseases
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 11/2017, Volume 69, Issue 11, pp. 2175 - 2186
Objective In most patients with nonsevere systemic necrotizing vasculitides (SNVs), remission is achieved with glucocorticoids alone, but one‐third experience... 
Glucocorticoids | Rhinosinusitis | Leukocytes (eosinophilic) | Granulomatosis | Patients | Placebo effect | Asthma | Randomization | Remission | Azathioprine | Failure | Steroid hormones | Steroids | Life Sciences | Human health and pathology | Rhumatology and musculoskeletal system
Journal Article
Journal Article
Autoimmunity Reviews, ISSN 1568-9972, 2014, Volume 13, Issue 10, pp. 1035 - 1041
Journal Article
Circulation, ISSN 0009-7322, 11/2015, Volume 132, Issue 18, pp. 1693 - 1700
BACKGROUND—The goal of this work was to assess the safety and efficacy of biologics (ie, tumor necrosis factor-α antagonists and tocilizumab) in patients with... 
Treatment outcome | Biological therapy | Vasculitis | Takayasu arteritis | CARDIAC & CARDIOVASCULAR SYSTEMS | METAANALYSIS | vasculitis | COMPETING RISK | biological therapy | REFERRAL CENTER | treatment outcome | DISEASE-ACTIVITY | TOCILIZUMAB | FACTOR THERAPY | TERM-FOLLOW-UP | MALIGNANCIES | PERIPHERAL VASCULAR DISEASE | NECROSIS-FACTOR INHIBITORS | TNF-ALPHA | Blood Sedimentation | Angiography | Humans | Middle Aged | Drug Evaluation | Takayasu Arteritis - diagnostic imaging | Male | Infliximab - therapeutic use | Adalimumab - adverse effects | Young Adult | Infliximab - adverse effects | Takayasu Arteritis - blood | Antirheumatic Agents - adverse effects | Adult | Female | Retrospective Studies | C-Reactive Protein - analysis | Drug Therapy, Combination | Drug Resistance | Antirheumatic Agents - therapeutic use | Antibodies, Monoclonal, Humanized - adverse effects | Takayasu Arteritis - drug therapy | Etanercept - adverse effects | Molecular Targeted Therapy - adverse effects | Antibodies, Monoclonal, Humanized - therapeutic use | Proportional Hazards Models | Treatment Outcome | Disease-Free Survival | Adalimumab - therapeutic use | Prednisone - therapeutic use | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Etanercept - therapeutic use | Care and treatment | Usage | Diagnosis | Takayasu's arteritis | Tumor necrosis factor inhibitors
Journal Article
Blood, ISSN 0006-4971, 07/2017, Volume 130, Issue 2, pp. 167 - 175
Langerhans cell histiocytosis (LCH) and Erdheim-Chester disease (ECD) are rare histiocytic disorders induced by somatic mutation of MAPK pathway genes.... 
MANAGEMENT | DENDRITIC CELLS | NEOPLASMS | DISTINCT | MONOCYTES | SUBSETS | NOTCH | MUTATIONS | MATURATION | HEMATOLOGY | BLOOD | Antigens, CD - immunology | Dendritic Cells - immunology | Histiocytosis, Langerhans-Cell - diagnosis | Histiocytosis, Langerhans-Cell - genetics | Humans | Lectins, C-Type - immunology | Dendritic Cells - pathology | Hematopoietic Stem Cells - pathology | Erdheim-Chester Disease - immunology | Male | Receptors, Notch - genetics | Monocytes - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Hematopoietic Stem Cells - immunology | Histiocytosis, Langerhans-Cell - pathology | Bone Marrow Cells - immunology | Monocytes - pathology | Adult | Female | Cell Differentiation | Foam Cells - pathology | Glycoproteins - genetics | Transforming Growth Factor beta - immunology | Antigens, CD1 - immunology | Diagnosis, Differential | Erdheim-Chester Disease - genetics | Gene Expression | Bone Marrow Cells - pathology | Immunophenotyping | Lipopolysaccharide Receptors - immunology | Histiocytosis, Langerhans-Cell - immunology | Erdheim-Chester Disease - pathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Glycoproteins - immunology | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Mannose-Binding Lectins - genetics | Mannose-Binding Lectins - immunology | Transforming Growth Factor beta - genetics | Proto-Oncogene Proteins B-raf - genetics | Proto-Oncogene Proteins B-raf - immunology | Alleles | Erdheim-Chester Disease - diagnosis | Lipopolysaccharide Receptors - genetics | Foam Cells - immunology | Antigens, CD1 - genetics | Mutation | Receptors, Notch - immunology | Myeloid Neoplasia
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 02/2014, Volume 34, Issue 4, pp. 595 - 605
Classifications Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
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