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1. Full Text CD8+ cytotoxic T lymphocytes in cancer immunotherapy: A review
by Farhood, Bagher and Najafi, Masoud and Mortezaee, Keywan
Journal of cellular physiology, ISSN 0021-9541, 06/2019, Volume 234, Issue 6, pp. 8509 - 8521
dendritic cells (DCs) | interferon‐γ (IFN‐γ) | tumor microenvironment (TME) | CD8+ cytotoxic T lymphocytes (CTLs) | regulatory T cells (Tregs) | immunotherapy | CTL‐associated antigen 4 (CTLA‐4) | programmed death‐1 receptor (PD‐1)–ligand (PD‐L1) | cancer cells | CD8 | programmed death-1 receptor (PD-1)–ligand (PD-L1) | CTL-associated antigen 4 (CTLA-4) | interferon-γ (IFN-γ) | cytotoxic T lymphocytes (CTLs) | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | T-Lymphocytes, Cytotoxic - immunology | Tumor Escape - drug effects | Cytokines - metabolism | Signal Transduction | Humans | Tumor Microenvironment | Lymphocytes, Tumor-Infiltrating - drug effects | Immunotherapy - adverse effects | T-Lymphocytes, Cytotoxic - drug effects | Antineoplastic Agents, Immunological - adverse effects | Neoplasms - therapy | Animals | T-Lymphocytes, Cytotoxic - metabolism | Antineoplastic Agents, Immunological - therapeutic use | Lymphocyte Activation - drug effects | Neoplasms - immunology | Cell Communication - drug effects | Lymphocytes, Tumor-Infiltrating - metabolism | Neoplasms - pathology | Lymphocytes, Tumor-Infiltrating - immunology | Antigens | Care and treatment | Dendritic cells | Immunotherapy | Development and progression | Interferon | Drug therapy | Biological response modifiers | T cells | Cancer | CD8 antigen | Cytotoxicity | Innate immunity | Lymphocytes T | Macrophages | Immunity | Receptors | Cell activation | Lymphocytes | Fibroblasts | Natural killer cells | Immune system | Immunoregulation | Priming | Inflammation | Immunological tolerance | CD4 antigen | Immunosuppression | Immune checkpoint | Exhaustion | Antitumor activity | Tumors | Apoptosis | Index Medicus
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2. Full Text Melatonin ameliorates radiation-induced oxidative stress at targeted and nontargeted lung tissue
by Ghobadi, Alireza and Shirazi, Alireza and Najafi, Masoud and Kahkesh, Masoud and Rezapoor, Saeed
Journal of medical physics, ISSN 0971-6203, 10/2017, Volume 42, Issue 4, pp. 241 - 244
Glutathione peroxidase | malondialdehyde | nontargeted effect | melatonin | radiation | superoxide dismutase | Ionizing radiation | Melatonin | Oxidative stress | Lipid peroxidation | Lipids | Superoxide dismutase | Pelvis | Antioxidants | Rodents | Peroxidase | Pretreatment | Drug dosages | Glutathione | Radiation effects | Medical research | Enzymes | Damage assessment | Free radicals | Ethanol | Rats | Radiation therapy | Malondialdehyde | Radiation dosage | Studies | Mutagenesis | Lungs | Laboratory animals | Radiation damage | Cancer | Original
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3. Full Text Cancer stem cells (CSCs) in cancer progression and therapy
by Najafi, Masoud and Farhood, Bagher and Mortezaee, Keywan
Journal of cellular physiology, ISSN 0021-9541, 06/2019, Volume 234, Issue 6, pp. 8381 - 8395
hypoxia | epithelial–mesenchymal transition (EMT) | tumor microenvironment (TME) | Notch | dedifferentiation | cancer stem cell (CSC) | therapy resistance | cancer cell | β‐catenin | cancer‐associated fibroblast (CAF) | WNT | CD44 | CSC niche | CD133 | transforming growth factor‐β (TGF‐β) | transforming growth factor-β (TGF-β) | cancer-associated fibroblast (CAF) | β-catenin | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Tumor Microenvironment - drug effects | Humans | Tumor Hypoxia - drug effects | Cancer-Associated Fibroblasts - drug effects | Antineoplastic Agents - therapeutic use | Epithelial-Mesenchymal Transition - genetics | Neoplasms - drug therapy | Cell Differentiation - genetics | Cancer-Associated Fibroblasts - pathology | Tumor Microenvironment - genetics | Neoplasms - genetics | Signal Transduction - drug effects | Neoplastic Stem Cells - pathology | Neoplasms - pathology | Stem Cell Niche - genetics | Care and treatment | Stem cells | Development and progression | Bone morphogenetic proteins | Transplantation | Transforming growth factors | Health aspects | Cancer | Antigens | Therapy | Phenotypes | Wnt protein | Transforming growth factor | siRNA | Progenitor cells | Cell surface | Metastases | Immunotherapy | Cells (biology) | Fibroblasts | Biomarkers | Hypoxia | Differentiation | Surface markers | Growth factors | Tumors | Index Medicus
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4. Full Text Immune system in cancer radiotherapy: Resistance mechanisms and therapy perspectives
by Mortezaee, Keywan and Najafi, Masoud
Critical reviews in oncology/hematology, ISSN 1040-8428, 01/2021, Volume 157, pp. 103180 - 103180
Tumor microenvironment (TME) | Immunotherapy | Regulatory T cells (Tregs) | Programmed death ligand 1 (PD-L1) | Radiotherapy | CD8+ T lymphocytes | Programmed cell Death-1 (PD-1) | Tumor Microenvironment | Immunologic Factors | Neoplasms - radiotherapy | Immune System | Humans | Index Medicus
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5. Full Text Transforming growth factor‐β signaling: Tumorigenesis and targeting for cancer therapy
by Ahmadi, Amirhossein and Najafi, Masoud and Farhood, Bagher and Mortezaee, Keywan
Journal of cellular physiology, ISSN 0021-9541, 08/2019, Volume 234, Issue 8, pp. 12173 - 12187
cancer therapy | cancer cell | epithelial–mesenchymal transition (EMT) | Smad4 | tumor microenvironment (TME) | metastasis | transforming growth factor (TGF)‐β | TβRII | cancer stem cell (CSC) | transforming growth factor (TGF)-β | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Signal Transduction - physiology | Tumor Microenvironment - physiology | Carcinogenesis - metabolism | Transforming Growth Factor beta - metabolism | Smad protein | Mesenchyme | Transforming growth factor-a | Multitasking | AKT protein | Metastasis | Kinases | Metastases | Proteins | Signaling | Signal transduction | Receptors | Clonal deletion | Tumorigenesis | Growth factors | Cancer | Tumors | Index Medicus
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6. Full Text Contribution of regulatory T cells to cancer: A review
by Najafi, Masoud and Farhood, Bagher and Mortezaee, Keywan
Journal of cellular physiology, ISSN 0021-9541, 06/2019, Volume 234, Issue 6, pp. 7983 - 7993
cancer cell | cyclooxygenase‐2 | immunosuppression | transforming growth factor‐β | regulatory T cell | tumor microenvironment | PD‐1 | forkhead box P3 | signal transducer and activator of transcription | transforming growth factor-β | cyclooxygenase-2 | PD-1 | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | T-Lymphocytes, Cytotoxic - immunology | Myeloid-Derived Suppressor Cells - immunology | Humans | Neoplasm Proteins - immunology | Cancer-Associated Fibroblasts - immunology | Forkhead Transcription Factors - genetics | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Neoplasm Metastasis | Neoplasms - genetics | Neoplasms - immunology | Killer Cells, Natural - immunology | Neoplasm Proteins - genetics | Neoplasms - pathology | Macrophages - immunology | COX-2 inhibitors | Bone morphogenetic proteins | Killer cells | T cells | Immunotherapy | Cancer | Transcription | Homeostasis | Cytotoxicity | Lymphocytes T | Macrophages | Suppressor cells | Metastases | Receptors | Lymphocytes | Fibroblasts | Foxp3 protein | Forkhead protein | Natural killer cells | Growth factors | Immune system | Phenotypes | Immunostimulation | Immunoregulation | Inflammation | Mucin | Cell death | Tumors | Apoptosis | Index Medicus
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7. Full Text Cancer stem cell (CSC) resistance drivers
by Najafi, Masoud and Mortezaee, Keywan and Majidpoor, Jamal
Life sciences (1973), ISSN 0024-3205, 10/2019, Volume 234, pp. 116781 - 116781
Tumor microenvironment (TME) | Resistance | Stemness | Epithelial-mesenchymal transition (EMT) | Chemotherapy | Cancer stem cell (CSC) | Dedifferentiation | Reactive oxygen species (ROS) | Plasticity | Radiotherapy | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Deprivation | Regulators | Reactive oxygen species | Mesenchyme | Crosstalk | Maintenance | Autophagy | Stem cells | Deoxyribonucleic acid--DNA | Phagocytosis | Tumors | Apoptosis | Cancer | Index Medicus
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8. Full Text Cyclooxygenase‐2 in cancer: A review
by Hashemi Goradel, Nasser and Najafi, Masoud and Salehi, Eniseh and Farhood, Bagher and Mortezaee, Keywan
Journal of cellular physiology, ISSN 0021-9541, 05/2019, Volume 234, Issue 5, pp. 5683 - 5699
cancer cell | NF‐κβ | prostaglandin | COX‐2 | NF-κβ | COX-2 | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Macrophages - pathology | Signal Transduction | Neoplastic Stem Cells - drug effects | Humans | Cancer-Associated Fibroblasts - drug effects | Neoplasms - enzymology | Tumor Microenvironment | Antineoplastic Agents - therapeutic use | Macrophages - enzymology | Neoplasms - drug therapy | Cancer-Associated Fibroblasts - pathology | Animals | Cyclooxygenase 2 - metabolism | Cyclooxygenase 2 Inhibitors - therapeutic use | Neoplastic Stem Cells - pathology | Macrophages - drug effects | Neoplasms - pathology | Cancer-Associated Fibroblasts - enzymology | Neoplastic Stem Cells - enzymology | Cell proliferation | COX-2 inhibitors | Prostaglandin E2 | Hormones | Metastasis | Drug resistance | Kinases | Macrophages | Cancer therapies | Carcinogenesis | Modulators | Metastases | Prostaglandin endoperoxide synthase | Proteins | Angiogenesis | Carcinogens | Epidermal growth factor | Fibroblasts | Inhibition | Growth factors | Epidermal growth factor receptors | Health risks | Risk reduction | MAP kinase | Radiation therapy | Yes-associated protein | Chemotherapy | Protein kinase | Stem cells | Hypoxia | Cancer | Apoptosis | Index Medicus
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