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International Journal of Radiation Oncology, Biology, Physics, ISSN 0360-3016, 2013, Volume 85, Issue 4, pp. 1110 - 1118
Purpose To explore the activity of a potent Chk1 inhibitor (SAR-020106) in combination with radiation. Methods and Materials Colony and mechanistic in vitro... 
Radiology | Hematology, Oncology and Palliative Medicine | CELLS | REPAIR | ONCOLOGY | PATHWAY | ABROGATION | KINASE | CHECKPOINT | TUMORIGENESIS | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | P53 | Microscopy - methods | Cell Cycle - genetics | G2 Phase - genetics | Tumor Stem Cell Assay - methods | Humans | DNA Repair - radiation effects | DNA Repair - genetics | Cyclin-Dependent Kinase Inhibitor p21 - deficiency | G2 Phase - drug effects | Radiation Tolerance - radiation effects | Isoquinolines - pharmacology | DNA Damage - genetics | Time-Lapse Imaging - methods | DNA Damage - drug effects | DNA Repair - drug effects | Protein Kinases - drug effects | Mitosis - radiation effects | Cell Cycle - radiation effects | Radiation-Sensitizing Agents - pharmacology | Radiation Tolerance - genetics | Tumor Suppressor Protein p53 - deficiency | Immunohistochemistry - methods | Radiation Tolerance - drug effects | Animals | Mitosis - drug effects | Histones - analysis | Mice, Nude | Papillomaviridae - classification | Cell Line, Tumor | Checkpoint Kinase 1 | Mice | HeLa Cells | Cell Cycle - drug effects | Pyrazines - pharmacology | Apoptosis | Papillomavirus infections | APOPTOSIS | HEAD | IRRADIATION | IN VITRO | CARCINOMAS | RADIOLOGY AND NUCLEAR MEDICINE | TUMOR CELLS | GENE RECOMBINATION | IN VIVO | NECK | RADIOSENSITIVITY | DNA DAMAGES
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