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Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 134 - 134
Introduction: The PALB2 gene encodes the partner and localizer of BRCA2 protein, which interacts with BRCA12 and is involved in homologous recombination DNA... 
Journal Article
Cancer Research, ISSN 0008-5472, 05/2015, Volume 75, Issue 9 Supplement, pp. P2 - P2-03-09
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 05/2015, Volume 107, Issue 5, p. 1
  Rapid advancements in massively parallel sequencing methods have enabled the analysis of breast cancer genomes at an unprecedented resolution, which have... 
Genomics | Estrogen | Genomes | Breast cancer | Mutation | Tumors
Journal Article
Breast Cancer Research, ISSN 1465-5411, 05/2014, Volume 16, Issue 3, pp. 210 - 210
In recent years it has become clear that cancer cells within a single tumor can display striking morphological, genetic and behavioral variability. Burgeoning... 
STEM-CELLS | ONCOLOGY | RARE MUTATIONS | QUANTIFICATION | RESISTANCE | GENETIC-HETEROGENEITY | CLONAL EVOLUTION | IN-SITU | MUTATIONAL EVOLUTION | DUCTAL CARCINOMA | REVEALS | Breast Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Breast Neoplasms - pathology | Humans | Neoplastic Stem Cells - pathology | Female | Models, Genetic | Mutation | Genetic Heterogeneity | Clonal Evolution | Review
Journal Article
Journal Article
The Journal of Pathology, ISSN 0022-3417, 03/2015, Volume 235, Issue 4, pp. 571 - 580
Mutations in genes encoding proteins involved in RNA splicing have been found to occur at relatively high frequencies in several tumour types including... 
breast cancer | spliceostatin A | alternative splicing | drivers | SF3B1 | next‐generation sequencing | Alternative splicing | Breast cancer | Next-generation sequencing | Drivers | Spliceostatin A | DUCTAL CARCINOMAS | MICROPAPILLARY CARCINOMAS | next-generation sequencing | MYELODYSPLASIA | PATHOLOGY | TUMORS | PRE-MESSENGER-RNA | UVEAL MELANOMA | ONCOLOGY | PATHWAY | GENES | CHRONIC LYMPHOCYTIC-LEUKEMIA | SPLICING FACTOR | Carcinoma, Papillary - genetics | Receptors, Estrogen - metabolism | Adenocarcinoma, Mucinous - pathology | Humans | Gene Expression Regulation, Neoplastic | Phosphoproteins - antagonists & inhibitors | Molecular Targeted Therapy | Phosphoproteins - metabolism | Carcinoma, Papillary - metabolism | Breast Neoplasms - metabolism | Dose-Response Relationship, Drug | Ribonucleoprotein, U2 Small Nuclear - metabolism | Transfection | RNA Interference | Ribonucleoprotein, U2 Small Nuclear - genetics | Female | Antineoplastic Agents - pharmacology | Adenocarcinoma, Mucinous - drug therapy | Pyrans - pharmacology | Cell Survival - drug effects | Genetic Predisposition to Disease | Alternative Splicing - genetics | RNA Splicing Factors | Carcinoma, Papillary - drug therapy | Phosphoproteins - genetics | Breast Neoplasms - drug therapy | Carcinoma, Papillary - pathology | Phenotype | Alternative Splicing - drug effects | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Adenocarcinoma, Mucinous - metabolism | Cell Line, Tumor | Ribonucleoprotein, U2 Small Nuclear - antagonists & inhibitors | Adenocarcinoma, Mucinous - genetics | Mutation | Spiro Compounds - pharmacology | RNA | Leukemia | Genomics | Melanoma | Proteins | Analysis | Pancreatic cancer | Genetic research | Genetic aspects | Nucleotide sequencing | Health aspects | DNA sequencing | Original Papers
Journal Article
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 07/2017, Volume 23, Issue 14, pp. 3859 - 3870
Purpose: Metaplastic breast carcinoma (MBC) is a rare and aggressive histologic type of breast cancer, predominantly of triple-negative phenotype, and... 
HETEROGENEITY | INACTIVATION | MUTATIONAL PROCESSES | ONCOLOGY | CLINICAL ONCOLOGY/COLLEGE | PATHWAY | RECOMMENDATIONS | CANCER AMERICAN SOCIETY | GROWTH | PRACTICE GUIDELINE UPDATE | TUMORS | Carcinoma, Ductal, Breast - genetics | PTEN Phosphohydrolase - genetics | Class I Phosphatidylinositol 3-Kinases - genetics | Humans | Gene Expression Regulation, Neoplastic | Signal Transduction - genetics | Transcription Factors - genetics | Proto-Oncogene Proteins c-akt - genetics | Tumor Suppressor Protein p53 - genetics | Metaplasia - pathology | Whole Exome Sequencing | Phosphatidylinositol 3-Kinases - genetics | TOR Serine-Threonine Kinases - genetics | Triple Negative Breast Neoplasms - genetics | Wnt Signaling Pathway - genetics | Triple Negative Breast Neoplasms - pathology | Carcinoma, Ductal, Breast - pathology | Female | Biomarkers, Tumor - genetics | Metaplasia - genetics | Mutation | Cadherins - genetics | Nuclear Proteins - genetics | TOR protein | Wnt protein | Mesenchyme | Invasiveness | p53 Protein | Genes | AKT protein | Breast cancer | Genomes | 1-Phosphatidylinositol 3-kinase | Gene sequencing | Metaplasia | Breast carcinoma | Alterations | Experimental design | Breast | PTEN protein | Tumors | Cancer | somatic mutations | metaplastic breast cancer | Massively parallel sequencing | Wnt pathway | copy number alterations
Journal Article
The Journal of Pathology, ISSN 0022-3417, 03/2016, Volume 238, Issue 4, pp. 508 - 518
Journal Article
The Journal of Pathology, ISSN 0022-3417, 10/2015, Volume 237, Issue 2, pp. 179 - 189
Adenoid cystic carcinoma (AdCC) is a rare type of triple‐negative breast cancer (TNBC) characterized by the presence of the MYB–NFIB fusion gene. The molecular... 
genomics | MYB–NFIB | adenoid cystic carcinoma | triple‐negative breast cancer | massively parallel sequencing | triple-negative breast cancer | MYB-NFIB | HEAD | HISTOLOGICAL SPECIAL TYPES | MYB EXPRESSION | PATHOLOGY | SALIVARY-GLANDS | MISSENSE MUTATIONS | SOMATIC MUTATIONS | ONCOLOGY | MUTATIONAL LANDSCAPE | COPY-NUMBER ALTERATION | NFIB GENE FUSION | HUMAN CANCER | Genetic Predisposition to Disease | Carcinoma, Adenoid Cystic - pathology | Biomarkers, Tumor - analysis | Gene Frequency | Humans | Gene Expression Regulation, Neoplastic | Carcinoma, Adenoid Cystic - chemistry | In Situ Hybridization, Fluorescence | Gene Dosage | Genes, myb | Reverse Transcriptase Polymerase Chain Reaction | DNA Copy Number Variations | Phenotype | Triple Negative Breast Neoplasms - chemistry | Triple Negative Breast Neoplasms - genetics | Carcinoma, Adenoid Cystic - genetics | DNA Mutational Analysis | Oncogene Proteins, Fusion - genetics | Triple Negative Breast Neoplasms - pathology | Female | Biomarkers, Tumor - genetics | Mutation | Genomics - methods | Real-Time Polymerase Chain Reaction | Ubiquitin | Carcinoma | Multiprocessing | Genomics | Genetic aspects | Breast cancer | Tumor proteins | Chromosomes | Cancer | Adenoid cystic carcinoma | massively parallel | Oncology | SOMATIC | Pathology | MUTATIONS | Cancer and Oncology | Cancer och onkologi
Journal Article
Nature Reviews Cancer, ISSN 1474-175X, 03/2014, Volume 14, Issue 3, pp. 147 - 148
Metaplastic breast carcinoma (MBC) is a special histological type of invasive breast cancer. MBC is a descriptive and operational term for a heterogeneous... 
ONCOLOGY | PATHWAY | GENES | BRCA1 | CANCER | TUMORS | P53 | Metaplasia | Phenotype | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Neoplasm Invasiveness | Humans | Female | Mutation | Care and treatment | Gene mutations | Analysis | Physiological aspects | Breast cancer | Genetic aspects | Research
Journal Article
The Journal of Pathology, ISSN 0022-3417, 04/2014, Volume 232, Issue 5, pp. 553 - 565
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2017, Volume 23, Issue 3, pp. 376 - 385
Journal Article