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1. Full Text Front Cover: Phenanthroline—A Versatile Ligand for Advanced Functional Polymeric Materials (Chem. Eur. J. 66/2018)
by Rothfuss, Hannah and Knöfel, Nicolai D and Tzvetkova, Pavleta and Michenfelder, Nadine C and Baraban, Sergej and Unterreiner, Andreas‐Neil and Roesky, Peter W and Barner‐Kowollik, Christopher
Chemistry – A European Journal, ISSN 0947-6539, 11/2018, Volume 24, Issue 66, pp. 17367 - 17367
The synthesis of phenanthroline‐containing polymer chains enables the subsequent incorporation of metal‐ions to generate functional macromolecular... 
copolymerization | N ligands | nanoparticles | supramolecular chemistry | lanthanides
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2. Full Text Base J represses genes at the end of polycistronic gene clusters inLeishmania majorby promoting RNAP II termination
: Base J promotes termination and represses gene expression
by Reynolds, David L and Hofmeister, Brigitte T and Cliffe, Laura and Siegel, T. Nicolai and Anderson, Britta A and Beverley, Stephen M and Schmitz, Robert J and Sabatini, Robert
Molecular Microbiology, ISSN 0950-382X, 08/2016, Volume 101, Issue 4, pp. 559 - 574
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3. Full Text Regulation of transcription termination by glucosylated hydroxymethyluracil, base J, in Leishmania major and Trypanosoma brucei
by Reynolds, David and Reynolds, David and Cliffe, Laura and Cliffe, Laura and Förstner, Konrad U and Förstner, Konrad U and Hon, Chung-Chau and Hon, Chung-Chau and Siegel, T. Nicolai and Siegel, T Nicolai and Sabatini, Robert and Sabatini, Robert
Nucleic acids research, ISSN 0305-1048, 2014, Volume 42, Issue 15, pp. 9717 - 9729
Base J, beta-d-glucosyl-hydroxymethyluracil, is an epigenetic modification of thymine in the nuclear DNA of flagellated protozoa of the order Kinetoplastida. J... 
VARIANT SURFACE GLYCOPROTEIN | RNA-POLYMERASE-II | DNA GLYCOSYLATION | MESSENGER-RNA | HELA-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SWI2/SNF2-LIKE PROTEIN | GENE-EXPRESSION | 5-BROMODEOXYURIDINE | POLYCISTRONIC TRANSCRIPTION | GENOME | Cell Line | Leishmania major - enzymology | Leishmania major - genetics | Transcription Termination, Genetic | Gene Expression Regulation | RNA Polymerase II - metabolism | Trypanosoma brucei brucei - genetics | Uracil - physiology | Trypanosoma brucei brucei - enzymology | RNA, Protozoan - analysis | Glucosides | Uracil - analogs & derivatives | Gene regulation, Chromatin and Epigenetics
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4. Full Text Corrigendum to "Hyperbolic Weyl groups and the four normed division algebras" [J. Algebra 322 1295-1339]
by Feingold, Alex J and Kleinschmidt, Axel and Nicolai, Hermann
Journal of Algebra, ISSN 0021-8693, 11/2017, Volume 489, p. 586
The following note contains corrections to our 2009 publication . 
Algebra
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5. Full Text Base J represses genes at the end of polycistronic gene clusters in Leishmania major by promoting RNAP II termination
by Reynolds, David L and Hofmeister, Brigitte T and Cliffe, Laura and Siegel, T. Nicolai and Anderson, Britta A and Beverley, Stephen M and Schmitz, Robert J and Sabatini, Robert
Molecular Microbiology, ISSN 0950-382X, 08/2016, Volume 101, Issue 4, pp. 559 - 574
Summary The genomes of kinetoplastids are organized into polycistronic gene clusters that are flanked by the modified DNA base J. Previous work has established... 
TRYPANOSOMA-BRUCEI | CRUZI | MESSENGER-RNA | HYDROXYMETHYLURACIL | DNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROBIOLOGY | GLUCOSYLTRANSFERASE | TRANSCRIPTION INITIATION | PARASITE | EXPRESSION | GENOME | Multigene Family | Leishmania major - genetics | RNA, Messenger - genetics | Gene Expression Regulation | RNA Polymerase II - metabolism | RNA, Messenger - metabolism | Glucosides - metabolism | Glucosides - genetics | Leishmania major - enzymology | Leishmania major - metabolism | RNA Polymerase II - genetics | Uracil - metabolism | Transcription, Genetic - genetics | Uracil - analogs & derivatives | Genetic transcription | RNA | Genes | RNA polymerases | Gene expression | Bacteriology | RNA polymerase
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6. Full Text Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
by Ridker, Paul M and Everett, Brendan M and Thuren, Tom and MacFadyen, Jean G and Chang, William H and Ballantyne, Christie and Fonseca, Francisco and Nicolau, Jose and Koenig, Wolfgang and Anker, Stefan D and Kastelein, John J.P and Cornel, Jan H and Pais, Prem and Pella, Daniel and Genest, Jacques and Cifkova, Renata and Lorenzatti, Alberto and Forster, Tamas and Kobalava, Zhanna and Vida-Simiti, Luminita and Flather, Marcus and Shimokawa, Hiroaki and Ogawa, Hisao and Dellborg, Mikael and Rossi, Paulo R.F and Troquay, Roland P.T and Libby, Peter and Glynn, Robert J and CANTOS Trial Grp and CANTOS Trial Group and Sahlgrenska akademin and Institute of Medicine and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Institutionen för medicin
The New England Journal of Medicine, ISSN 0028-4793, 09/2017, Volume 377, Issue 12, pp. 1119 - 1131
Patients with myocardial infarction and a high-sensitivity CRP level of 2 mg or more per liter were assigned to one of three canakinumab doses or placebo. The... 
C-REACTIVE PROTEIN | MEDICINE, GENERAL & INTERNAL | HEART-DISEASE | MYOCARDIAL-INFARCTION | STATIN THERAPY | CLONAL HEMATOPOIESIS | INFLAMMATION | CARDIOVASCULAR-DISEASE | INTERLEUKIN-1-BETA INHIBITION | RISK | CORONARY-ARTERY-DISEASE | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Male | Secondary Prevention | Infection - etiology | Incidence | Dose-Response Relationship, Drug | Anti-Inflammatory Agents - adverse effects | C-Reactive Protein - metabolism | Lipids - blood | Cardiovascular Diseases - epidemiology | Cardiovascular Diseases - mortality | Anti-Inflammatory Agents - administration & dosage | Female | Neutropenia - chemically induced | Interleukin-1beta - antagonists & inhibitors | Stroke - prevention & control | Atherosclerosis - drug therapy | Double-Blind Method | Interleukin-1beta - immunology | Atherosclerosis - blood | Myocardial Infarction - drug therapy | Antibodies, Monoclonal - administration & dosage | Aged | Myocardial Infarction - prevention & control | Myocardial infarction | Cerebral infarction | Stroke | Heart attacks | C-reactive protein | Cytokines | Clinical trials | Cardiovascular disease | Innate immunity | Inflammation | Angina | Angina pectoris | Thrombosis | Cholesterol | Disease prevention | Proteins | Arteriosclerosis | Atherosclerosis | Cardiovascular diseases | Health risk assessment | Drug therapy | Drug dosages | Klinisk medicin | Clinical Medicine
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