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Diabetes, ISSN 0012-1797, 08/2009, Volume 58, Issue 8, p. 1929
  Inactivating mutations in glucokinase (GCK) cause mild fasting hyperglycemia. Identification of a GCK mutation has implications for treatment and prognosis;... 
Journal Article
Diabetes, ISSN 0012-1797, 08/2009, Volume 58, Issue 8, pp. 1929 - 1935
OBJECTIVE--Inactivating mutations in glucokinase (GCK) cause mild fasting hyperglycemia. Identification of a GCK mutation has implications for treatment and... 
Hyperglycemia | Promoters (Genetics) | Gene mutations | Glucokinase | Causes of | Physiological aspects | Genetic aspects | Research | Health aspects
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0120295 - e0120295
Journal Article
Journal of the American College of Surgeons, ISSN 1072-7515, 10/2017, Volume 225, Issue 4, pp. S175 - S176
Journal Article
EPJ Web of Conferences, ISSN 2101-6275, 06/2018, Volume 184, p. 1008
The Trojan Horse Method (THM) is an indirect method that allows to get information about a two body reaction cross-section even at very low energy, avoiding... 
Malware | Divergence | Reaction mechanisms | Experiments | Kinematics | Ion beams
Conference Proceeding
by Lundqvist, Andreas and van Hoef, Vincent and Zhang, Xiaonan and Wennerberg, Erik and Lorent, Julie and Witt, Kristina and Sanz, Laia Masvidal and Liang, Shuo and Murray, Shannon and Larsson, Ola and Kiessling, Rolf and Mao, Yumeng and Sidhom, John-William and Bessell, Catherine A and Havel, Jonathan and Schneck, Jonathan and Chan, Timothy A and Sachsenmeier, Eliot and Woods, David and Berglund, Anders and Ramakrishnan, Rupal and Sodre, Andressa and Weber, Jeffrey and Zappasodi, Roberta and Li, Yanyun and Qi, Jingjing and Wong, Philip and Sirard, Cynthia and Postow, Michael and Newman, Walter and Koon, Henry and Velcheti, Vamsidhar and Callahan, Margaret K and Wolchok, Jedd D and Merghoub, Taha and Lum, Lawrence G and Choi, Minsig and Thakur, Archana and Deol, Abhinav and Dyson, Gregory and Shields, Anthony and Haymaker, Cara and Uemura, Marc and Murthy, Ravi and James, Marihella and Wang, Daqing and Brevard, Julie and Monaghan, Catherine and Swann, Suzanne and Geib, James and Cornfeld, Mark and Chunduru, Srinivas and Agrawal, Sudhir and Yee, Cassian and Wargo, Jennifer and Patel, Sapna P and Amaria, Rodabe and Tawbi, Hussein and Glitza, Isabella and Woodman, Scott and Hwu, Wen-Jen and Davies, Michael A and Hwu, Patrick and Overwijk, Willem W and Bernatchez, Chantale and Diab, Adi and Massarelli, Erminia and Segal, Neil H and Ribrag, Vincent and Melero, Ignacio and Gangadhar, Tara C and Urba, Walter and Schadendorf, Dirk and Ferris, Robert L and Houot, Roch and Morschhauser, Franck and Logan, Theodore and Luke, Jason J and Sharfman, William and Barlesi, Fabrice and Ott, Patrick A and Mansi, Laura and Kummar, Shivaani and Salles, Gilles and Carpio, Cecilia and Meier, Roland and Krishnan, Suba and McDonald, Dan and Maurer, Matthew and Gu, Xuemin and Neely, Jaclyn and Suryawanshi, Satyendra and Levy, Ronald and Khushalani, Nikhil and Wu, Jennifer and Zhang, Jinyu and Basher, Fahmin and Rubinstein, Mark and Bucsek, Mark and Qiao, Guanxi and ...
Journal for ImmunoTherapy of Cancer, ISSN 2051-1426, 11/2016, Volume 4, Issue S1
Journal Article
Journal of Thoracic and Cardiovascular Surgery, The, ISSN 0022-5223, 2016, Volume 151, Issue 2, pp. 538 - 546
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0121765 - e0121765
Smyd1/Bop is an evolutionary conserved histone methyltransferase previously shown by conventional knockout to be critical for embryonic heart development. To... 
PROGENITOR CELLS | SMOOTH-MUSCLE | PROTEIN | GENE | FIELD | MYND | MULTIDISCIPLINARY SCIENCES | IN-VIVO | ENDOPLASMIC-RETICULUM STRESS | DIRECT TRANSCRIPTIONAL TARGET | AUTOPHAGY | Up-Regulation | Cell Proliferation | Oxidative Stress | Humans | Transcription Factors - deficiency | Cercopithecus aethiops | Molecular Sequence Data | Muscle Proteins - deficiency | DNA-Binding Proteins - deficiency | Cell Cycle Proteins - chemistry | DNA-Binding Proteins - metabolism | Gene Expression Regulation, Developmental | Myocardium - metabolism | Muscle Proteins - metabolism | Transcription, Genetic | Amino Acid Sequence | Heart - growth & development | Cell Cycle Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Gene Knockout Techniques | Myocardium - cytology | Muscle Proteins - genetics | Transcription Factors - metabolism | Animals | Embryo, Mammalian - embryology | Endoplasmic Reticulum Stress | Mice | Methylation | COS Cells | Heart | Histones | Embryonic development | Genetic engineering | Developmental biology | Pediatrics | Oxidative stress | Laboratories | Impairment | Transgenic | Cardiovascular disease | Biochemistry | Lethality | Autophagy | Defects | Morphogenesis | Proteins | Embryogenesis | Developmental stages | Clonal deletion | Evolutionary conservation | Rodents | Deletion | Histone methyltransferase | Outflow | Nkx2.5 protein | Heart failure | Stresses | Phenotypes | Departments | Superhigh frequencies | Zebrafish | Cardiomyocytes | Biophysics | Gene expression | Embryos | Ventricle | Molecular biology | Endoplasmic reticulum | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2016, Volume 11, Issue 4, pp. e0154590 - e0154590
Objective Clinical studies demonstrated a potential link between atherosclerosis and periodontitis. Porphyromonas gingivalis (Pg), one of the main periodontal... 
IMMUNE-RESPONSE | APOPTOSIS | ACTIVATION | EPITHELIAL-CELLS | PERIODONTAL PATHOGENS | MULTIDISCIPLINARY SCIENCES | DISEASE | LOW-DENSITY-LIPOPROTEIN | ATHEROSCLEROSIS | EXPRESSION | CATHEPSIN-B | Cell Survival - drug effects | Atherosclerosis - pathology | Caspase 9 - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Apoptosis - drug effects | Porphyromonas gingivalis - pathogenicity | Humans | Caspase 3 - metabolism | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Lipoproteins, LDL - pharmacology | Periodontitis - pathology | Caspase 1 - metabolism | Human Umbilical Vein Endothelial Cells - microbiology | Apoptotic Protease-Activating Factor 1 - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Bacteroidaceae Infections - microbiology | Lipopolysaccharides - pharmacology | Porphyromonas gingivalis - metabolism | Bacteroidaceae Infections - pathology | Lipoproteins (low density) | Bax protein | Bcl-2 protein | Smooth muscle | Propidium iodide | Stimulation | Infections | Synergism | Caspase-1 | Lipopolysaccharides | Pathways | Enzymatic activity | Atherosclerosis | Gangrene | Hemoglobin | Pathogens | Bacterial infections | Iodides | Cytokines | Mortality | Caspase | Inflammation | Tumor necrosis factor-α | Gene expression | Endothelial cells | Periodontitis | Apaf-1 protein | Cell death | Arteriosclerosis | Annexin V | Umbilical vein | Viability | Apoptosis | Index Medicus
Journal Article
Annals of Thoracic Surgery, ISSN 0003-4975, 2008, Volume 86, Issue 4, pp. 1123 - 1129
Journal Article