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Cancer Science, ISSN 1347-9032, 07/2013, Volume 104, Issue 7, pp. 810 - 816
The transcription factor SOX2 is essential for the maintenance of embryonic stem cells and normal development of the esophagus. Our previous study revealed... 
PLURIPOTENT STEM-CELLS | LUNG | IN-VITRO | AMPLIFICATION | ONCOLOGY | AKT | CANCER | EXPRESSION | LINES | TOR Serine-Threonine Kinases - metabolism | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Male | Multiprotein Complexes - genetics | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - genetics | Esophageal Neoplasms - pathology | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - antagonists & inhibitors | SOXB1 Transcription Factors - metabolism | TOR Serine-Threonine Kinases - antagonists & inhibitors | Multiprotein Complexes - metabolism | Heterografts | TOR Serine-Threonine Kinases - genetics | SOXB1 Transcription Factors - genetics | Esophageal Neoplasms - metabolism | Phosphorylation - drug effects | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Esophageal Squamous Cell Carcinoma | Cell Survival - drug effects | Signal Transduction | Morpholines - pharmacology | Sirolimus - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Animals | MAP Kinase Signaling System - drug effects | Esophageal Neoplasms - genetics | Mice, Nude | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Mice, Inbred BALB C | Squamous cell carcinoma | DNA microarrays | Growth | Analysis | Genetic research | Embryonic stem cells | Esophageal cancer | Tumors
Journal Article
Journal of Gastroenterology, ISSN 0944-1174, 3/2018, Volume 53, Issue 3, pp. 438 - 448
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2017, Volume 12, Issue 3, p. e0173661
Erythropoietin (EPO), clinically used as a hematopoietic drug, has received much attention due to its nonhematopoietic effects. EPO reportedly has beneficial... 
FAT OXIDATION | ADIPOCYTES | HEPATIC INSULIN-RESISTANCE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | INCREASES ENERGY-EXPENDITURE | METABOLIC-ACTIVITY | SENSITIVITY | PPAR-GAMMA | DIFFERENTIATION | ADIPOGENESIS | Erythropoietin - pharmacology | Glucose Tolerance Test | Obesity - drug therapy | Mice, Inbred C57BL | Insulin Resistance | Adipose Tissue, White - metabolism | Diet, High-Fat - adverse effects | Male | Adipocytes - drug effects | DNA-Binding Proteins - metabolism | Thermogenesis - physiology | Transcription Factors - metabolism | Fibroblast Growth Factors - metabolism | Glucose Intolerance - drug therapy | Animals | Adipocytes - metabolism | Obesity - etiology | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Erythropoietin - metabolism | Thermogenesis - drug effects | Anti-Obesity Agents - pharmacology | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | Adipose tissues | Obesity | Usage | Care and treatment | Erythropoietin | Homeostasis | Research | Glucose tolerance tests | Metabolic rate | Pediatrics | Fibroblast growth factor | Adipose tissue | Peptides | Body fat | Liver | Body weight | Science | Homology | Adipocytes | Glucose | Kinases | High fat diet | Uncoupling protein 1 | Rodents | Skin temperature | Adipose tissue (brown) | Growth factors | Locomotor activity | Gluconeogenesis | Recombinant | Carbohydrates | Excretion | Anemia | Diabetes mellitus | Stat3 protein | MiRNA | Oxygen consumption | Metabolism | Gene expression | Insulin | Hemopoiesis | Medicine | Glucose tolerance | Musculoskeletal system | Thermogenesis | Diet | Ribonucleic acids | Weight reduction | Adrenergic receptors | Skin | Mice | Diabetes
Journal Article
Journal Article
Journal Article
Human Pathology, ISSN 0046-8177, 2017, Volume 62, pp. 134 - 140
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1533 - 1544
The cause of organ failure is enigmatic for many degenerative diseases, including end-stage liver disease. Here, using a CCl4-induced rat model of irreversible... 
LONG-TERM | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | NUCLEAR | MECHANISM | CHRONIC LIVER-DISEASE | CIRRHOSIS | RATS | HEPATOCYTE | EXPRESSION | TRANSPLANTATION | Liver Cirrhosis, Experimental - therapy | Liver Failure - etiology | Dependovirus - genetics | Genetic Therapy | Hepatocyte Nuclear Factor 1-alpha - genetics | Hepatocyte Nuclear Factor 3-beta - genetics | Rats, Inbred Lew | Transcriptome | Hepatocyte Nuclear Factor 1-alpha - biosynthesis | Liver Failure - pathology | Hepatocytes - pathology | Male | Gene Expression Profiling | Genetic Vectors - therapeutic use | Hepatocytes - metabolism | Gene Regulatory Networks | Hepatocyte Nuclear Factor 4 - physiology | Recombinant Fusion Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Hepatocyte Nuclear Factor 4 - biosynthesis | Liver Cirrhosis, Experimental - pathology | Liver Cirrhosis, Experimental - complications | Hepatocyte Nuclear Factor 3-beta - biosynthesis | Transcription Factors - physiology | Transduction, Genetic | PPAR alpha - biosynthesis | Down-Regulation | Cells, Cultured | Gene Expression Regulation | Rats | PPAR alpha - genetics | Hepatocyte Nuclear Factor 4 - genetics | Liver Cirrhosis, Experimental - genetics | Liver Failure - genetics | Disease Progression | Animals | Carbon Tetrachloride Poisoning - therapy | Liver Failure - therapy | CCAAT-Enhancer-Binding Protein-alpha - genetics | Carbon Tetrachloride Poisoning - genetics | Cell Dedifferentiation - genetics | Transcription factors | Liver diseases | Genetic research | Development and progression | Genetic aspects | Genetic transcription | Research | Properties | Hypertension | Cytochrome | Transplants & implants | Rodents | Stem cells | Metabolism | Laboratory animals | Liver cirrhosis | Hepatology | Gastroenterology
Journal Article
Journal Article
Journal of Gastroenterology, ISSN 0944-1174, 6/2011, Volume 46, Issue 6, pp. 809 - 821
Journal Article