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Nature Communications, ISSN 2041-1723, 04/2017, Volume 8, p. 14727
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2018, Volume 13, Issue 7, p. e0201566
Background and aims Nonalcoholic fatty liver disease is one of the major complications of obesity, occurring already in pediatric age. Insulin like growth... 
FIBROSIS | ADULT PATIENTS | IGF-I | HEPATIC STELLATE CELLS | CIRRHOSIS | MULTIDISCIPLINARY SCIENCES | LIFE-STYLE INTERVENTIONS | DISEASE | REPLACEMENT THERAPY | RATS | NONALCOHOLIC STEATOHEPATITIS | Non-alcoholic Fatty Liver Disease - pathology | Receptor, IGF Type 1 - metabolism | Liver - pathology | Humans | Male | Insulin-Like Growth Factor I - genetics | Non-alcoholic Fatty Liver Disease - complications | Liver Cirrhosis - metabolism | Female | Child | Liver Cirrhosis - genetics | Pediatric Obesity - pathology | Gene Expression | Non-alcoholic Fatty Liver Disease - genetics | Liver Cirrhosis - complications | Liver - metabolism | Non-alcoholic Fatty Liver Disease - metabolism | Pediatric Obesity - genetics | Pediatric Obesity - metabolism | Receptor, IGF Type 1 - genetics | Disease Progression | Biopsy | Adolescent | Liver Cirrhosis - pathology | Pediatric Obesity - complications | Insulin-Like Growth Factor I - metabolism | Cohort Studies | Pediatrics | Medical research | Liver diseases | Medicine, Experimental | Development and progression | Obesity in children | Children | Obesity in adolescence | Health aspects | Adolescence | Obesity | Stellate cells | Complications | Insulin-like growth factor I | Liver | Pharmacology | mRNA | Insulin-like growth factors | Males | Insulin | Chemical compounds | Studies | Fatty liver | Rodents | Fibrosis | Adolescents | Immunofluorescence | Females
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 2045 - 14
Two recent randomized controlled trials demonstrated improved radiographic, histological and hepatometabolic cues of non-alcoholic steatohepatitis (NASH) in... 
ADULT PATIENTS | VITAMIN-E | METABOLISM | CHOLINE | FATTY LIVER-DISEASE | MULTIDISCIPLINARY SCIENCES | NONALCOHOLIC STEATOHEPATITIS | MECHANISMS | SERUM | TRANSPLANTATION | CHILDREN | Pediatrics | Clinical trials | Arachidonic acid | Docosahexaenoic acid | Fatty acids | Patients | Linolenic acid | Metabolites | Vitamin D | Biopsy | Vitamin E | Choline | Biomarkers | Lipid metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2018, Volume 13, Issue 6, p. e0198490
Early life events are associated with the susceptibility to chronic diseases in adult life. Perturbations of endoplasmic reticulum (ER) homeostasis activate... 
INTRAUTERINE GROWTH-RETARDATION | INHIBITION | METABOLISM | FATTY LIVER-DISEASE | ER STRESS | MULTIDISCIPLINARY SCIENCES | CONSEQUENCES | ISCHEMIC-HEART-DISEASE | ENDOPLASMIC-RETICULUM STRESS | MODEL | THRIFTY PHENOTYPE HYPOTHESIS | Liver - pathology | Endoplasmic Reticulum - metabolism | Male | RNA, Messenger - metabolism | Heat-Shock Proteins - genetics | Leptin - blood | Fatty Acids, Nonesterified - blood | Female | Aspartate-Ammonia Ligase - metabolism | Disease Models, Animal | Glucose Tolerance Test | Unfolded Protein Response - genetics | Heat-Shock Proteins - metabolism | Liver - metabolism | Gene Expression Regulation | Aspartate-Ammonia Ligase - genetics | Metabolome | Rats | Fetal Growth Retardation - metabolism | Rats, Sprague-Dawley | Fetal Growth Retardation - pathology | Pregnancy | Animals | X-Box Binding Protein 1 - metabolism | X-Box Binding Protein 1 - genetics | Fetus | Cellular signal transduction | Models | Research | Gene expression | Growth retardation | Phosphorylation | Transcription factors | Disease | Liver | Genes | Homeostasis | Homology | mRNA | Activation | Glucose | Kinases | Proteins | Aspartate-ammonia ligase | Fatty liver | Protein folding | Rodents | Hepatology | Physiology | Lipogenesis | Gluconeogenesis | Medical research | Fetuses | Double-stranded RNA | Exposure | Metabolism | Steatosis | Glucose tolerance | Hypotheses | Hospitals | Ribonucleic acids | Protein kinase | Fibrosis | Asparagine | Insulin resistance | Diabetes | Gene therapy | Endoplasmic reticulum
Journal Article
Pediatric Research, ISSN 0031-3998, 2018, Volume 84, Issue 5, pp. 696 - 703
BACKGROUND: FADS1 gene encodes delta 5 desaturase, a rate-limiting enzyme in the metabolism of n-3 and n-6 polyunsaturated fatty acids (PUFAs). Minor alleles... 
GENETIC-VARIATION | LONG-CHAIN OMEGA-3 | NAFLD | POLYMORPHISMS | N-3 | OMEGA-3-FATTY-ACIDS | ADULTS | PEDIATRICS | FISH-OIL | PARAMETERS | NASH | Pediatrics | Liver diseases
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 05/2017, Volume 24, Issue 5, pp. 889 - 902
Hepatocellular carcinoma (HCC) is the most common type of liver cancer in humans. The focal adhesion tyrosine kinase (FAK) is often over-expressed in human HCC... 
OVEREXPRESSION | METHYLATION | PROTEIN | INHIBITION | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | NUCLEAR FAK | MECHANISMS | HISTONE METHYLTRANSFERASE EZH2 | CANCER | EXPRESSION | CELL BIOLOGY | Focal Adhesion Kinase 1 - genetics | Neoplasm Transplantation | RNA, Small Interfering - genetics | E2F2 Transcription Factor - genetics | Apoptosis - drug effects | Humans | Gene Expression Regulation, Neoplastic | Apoptosis - genetics | Male | Receptor, Notch2 - genetics | E2F2 Transcription Factor - metabolism | Tumor Suppressor Protein p53 - genetics | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Focal Adhesion Kinase 1 - metabolism | Promoter Regions, Genetic | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Liver Neoplasms - genetics | Signal Transduction | Receptor, Notch2 - metabolism | Tumor Suppressor Protein p53 - metabolism | E2F3 Transcription Factor - genetics | G2 Phase Cell Cycle Checkpoints | Hep G2 Cells | Animals | Histones - genetics | Mice, Nude | Aminopyridines - pharmacology | Carcinoma, Hepatocellular - pathology | E2F3 Transcription Factor - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Histones - metabolism | Focal Adhesion Kinase 1 - antagonists & inhibitors | Carcinoma, Hepatocellular - metabolism | RNA, Small Interfering - metabolism | Original Paper
Journal Article