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Nature, ISSN 0028-0836, 2013, Volume 498, Issue 7455, pp. 492 - 496
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 07/2015, Volume 112, Issue 27, pp. 8421 - 8426
Cerebral cavernous malformation (CCM) is a disease of the central nervous system causing hemorrhage-prone multiple lumen vascular malformations and very severe... 
Cerebral cavernous malformation | Sulindac metabolites | catenin | Endothelial cells | Vascular pathology | cerebral cavernous malformation | beta-catenin | endothelial cells | SIGNALING CONTROLS | MULTIDISCIPLINARY SCIENCES | BLOOD-BRAIN-BARRIER | EFFECTOR | CEREBRAL CAVERNOUS MALFORMATIONS | vascular pathology | PATHWAY | RAP1 | DISEASE | JUNCTIONS | GROWTH | sulindac metabolites | Immunohistochemistry | Central Nervous System Neoplasms - metabolism | Endothelial Cells - metabolism | Central Nervous System Neoplasms - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Hemangioma, Cavernous, Central Nervous System - drug therapy | beta Catenin - metabolism | Hemangioma, Cavernous, Central Nervous System - genetics | Hemangioma, Cavernous, Central Nervous System - metabolism | beta Catenin - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Mice, Knockout | Intracellular Signaling Peptides and Proteins - deficiency | Sulindac - analogs & derivatives | Animals | Signal Transduction - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Central Nervous System Neoplasms - drug therapy | Intracellular Signaling Peptides and Proteins - genetics | Sulindac - pharmacology | Transforming Growth Factor beta - metabolism | Disease Models, Animal | Usage | Sulindac | Genetic aspects | Research | Drug therapy | Health aspects | Cerebrovascular disease | Biological Sciences | β-catenin | Other Medical Sciences not elsewhere specified | Other Medical Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Annan medicin och hälsovetenskap | Övrig annan medicin och hälsovetenskap
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 8/2004, Volume 166, Issue 3, pp. 359 - 367
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70233
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2014, Volume 124, Issue 10, pp. 4335 - 4350
Journal Article
Journal of Cell Science, ISSN 0021-9533, 04/2010, Volume 123, Issue 7, pp. 1073 - 1080
Journal Article
Journal Article
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 01/2016, Volume 8, Issue 1, pp. 6 - 24
Cerebral cavernous malformations (CCMs) are vascular malformations located within the central nervous system often resulting in cerebral hemorrhage.... 
endothelial cells | TGFβ‐BMP | CCM | KLF4 | EndMT | Endothelial cells | TGFβ-BMP | MEDICINE, RESEARCH & EXPERIMENTAL | RHO KINASE | BLOOD-BRAIN-BARRIER | KRUPPEL-LIKE FACTORS | ENDOTHELIAL-MESENCHYMAL TRANSITION | TGF beta-BMP | SIGNALING PATHWAY | VASCULAR INTEGRITY | SHEAR-STRESS | VE-CADHERIN | GROWTH-FACTOR | TRANSCRIPTION FACTOR | Cell Proliferation | KRIT1 Protein | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Hemangioma, Cavernous, Central Nervous System - genetics | Hemangioma, Cavernous, Central Nervous System - metabolism | Bone Morphogenetic Protein 6 - genetics | RNA Interference | Kruppel-Like Transcription Factors - metabolism | HEK293 Cells | Disease Models, Animal | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Bone Morphogenetic Protein 6 - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Microtubule-Associated Proteins - antagonists & inhibitors | Mitogen-Activated Protein Kinase 7 - metabolism | Disease Progression | Mice, Knockout | Animals | Bone Morphogenetic Protein 6 - metabolism | Hemangioma, Cavernous, Central Nervous System - pathology | Endothelial Cells - cytology | Smad1 Protein - metabolism | Mice | Mutation | Kruppel-Like Transcription Factors - antagonists & inhibitors | Kruppel-Like Transcription Factors - genetics | Transforming Growth Factor beta - metabolism | Brain | Bone morphogenetic proteins | Transforming growth factors | Mortality | Stem cells | Vascular Biology & Angiogenesis | Genetics, Gene Therapy & Genetic Disease | Cardiovascular System | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2011, Volume 208, Issue 9, pp. 1835 - 1847
Journal Article
The EMBO Journal, ISSN 0261-4189, 04/2008, Volume 27, Issue 7, pp. 993 - 1004
VE‐cadherin is an endothelial‐specific transmembrane protein concentrated at cell‐to‐cell adherens junctions. Besides promoting cell adhesion and controlling... 
VE‐cadherin | TGF‐β | endothelial cells | Smad | signal transduction | Signal transduction | TGF-β | Endothelial cells | VE-cadherin | HEREDITARY HEMORRHAGIC TELANGIECTASIA | ANGIOGENESIS | MICE LACKING ENDOGLIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CATENIN | CELL BIOLOGY | RECEPTOR-LIKE KINASE-1 | TGF-beta | GROWTH-FACTOR-BETA | GENE | CELL-MIGRATION | BLOOD-VESSELS | EXPRESSION | Embryo, Mammalian - drug effects | Activin Receptors, Type II - metabolism | Transcription, Genetic - drug effects | Allantois - cytology | Cadherins - metabolism | Humans | Smad3 Protein - metabolism | Embryo, Mammalian - metabolism | Antigens, CD - metabolism | Cell Nucleus - metabolism | Protein Binding - drug effects | Allantois - drug effects | Phosphorylation - drug effects | Dimerization | Protein-Serine-Threonine Kinases - metabolism | Endothelial Cells - metabolism | Smad2 Protein - metabolism | Cell Movement - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Allantois - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Embryo, Mammalian - cytology | Endothelial Cells - cytology | Models, Biological | Cell Proliferation - drug effects | Mice | Kinetics | Cell Nucleus - drug effects | Cadherins - deficiency | Endothelial Cells - drug effects
Journal Article