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PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, p. e37505
Background: Heart failure (HF) involves alterations in metabolism, but little is known about cardiomyopathy-(CM)-specific or diabetes-independent alterations... 
OXIDATIVE CAPACITY | SKELETAL-MUSCLE | PROTEIN | MITOCHONDRIAL | TRANSPORTERS | MULTIDISCIPLINARY SCIENCES | ACTIVATED RECEPTOR-ALPHA | ENDOPLASMIC-RETICULUM STRESS | PROMOTER | FAILURE | DEFICIENCY | Heart Ventricles - cytology | Calcium Channels - metabolism | Calcium - metabolism | Humans | Middle Aged | Heart Failure - physiopathology | Immunoblotting | Male | DNA Primers - genetics | RNA, Messenger - metabolism | Case-Control Studies | CD36 Antigens - metabolism | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Statistics, Nonparametric | Female | Fatty Acids - metabolism | Real-Time Polymerase Chain Reaction | Gene Expression Regulation - genetics | Heat-Shock Proteins - metabolism | Gene Expression Regulation - physiology | Heart Failure - genetics | Heart Failure - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Transcription Factors - metabolism | Metabolic Networks and Pathways - genetics | PPAR alpha - metabolism | Proteins | Messenger RNA | Genes | Physiological aspects | Genetic aspects | Diabetes | Gene expression | T cells | Protein binding | Heart | Plasma | Correlation | Biomedical research | Transcription | Calcium | Laboratories | Cardiomyopathy | Calcium metabolism | Cytotoxicity | Lipids | Lymphocytes T | Alterations | Transgenic animals | Rodents | Oxidation | Protein transport | Cardiology | Heart diseases | Inositol 1,4,5-trisphosphate | Inositol 1,4,5-trisphosphate receptors | Heart failure | Diabetes mellitus | CD36 antigen | Metabolism | Patients | Fatty acids | Musculoskeletal system | Acids | Myocardium | Ligands | Peroxisome proliferator-activated receptors | Ventricle | Handling | Calci en l'organisme | Malalties del cor | Calcium in the body | Cardiologia | Expressió gènica
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