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Journal of Clinical Investigation, ISSN 0021-9738, 06/2014, Volume 124, Issue 6, pp. 2294 - 2294
Kidney disease is one of the most prevalent chronic conditions and is a frequent complication of diabetes, cardiovascular disease, and obesity. Recent advances... 
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 11/2011, Volume 22, Issue 11, pp. 2119 - 2128
Journal Article
2016, Fourth edition., STAT!Ref electronic medical library, ISBN 1555816029, 1 volume (various pagings)
Provides a comprehensive analysis of general methodologies, environmental public health microbiology, microbial ecology, and biodegradation and... 
Écologie microbienne | Microbial ecology | Sanitary microbiology | Microbiologie sanitaire | Laboratory manuals
Book
American Journal of Kidney Diseases, ISSN 0272-6386, 08/2018, Volume 72, Issue 2, pp. 164 - 167
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 12/2016, Volume 27, Issue 12, pp. 3600 - 3610
APOL1 risk variants are associated with kidney disease in blacks, but the mechanisms of renal injury associated with APOL1 risk variants are unknown. Because... 
Pre-Eclampsia - genetics | Pregnancy | Animals | Lipoproteins, HDL - genetics | Podocytes - physiology | Apolipoproteins - genetics | Apolipoprotein L1 | Female | Mice, Transgenic | Mice | Kidney Diseases - genetics | Disease Models, Animal | chronic kidney disease | focal segmental glomerulosclerosis | transgenic mouse | podocyte | Basic Research | genetic renal disease
Journal Article
Cell, ISSN 0092-8674, 2011, Volume 145, Issue 4, pp. 513 - 528
Journal Article
American Journal of Kidney Diseases, ISSN 0272-6386, 10/2017, Volume 70, Issue 4, pp. 460 - 463
Journal Article
American journal of physiology. Renal physiology, ISSN 0363-6127, 01/2019, Volume 316, Issue 1, pp. F1 - F8
The mechanism that explains the association of APOL1 variants with nondiabetic kidney diseases in African Americans remains unclear. Kidney disease risk is... 
podocytes | chronic kidney disease | genetics | LOCALIZATION | PHYSIOLOGY | PROTEIN | GENE VARIANTS | ASSOCIATE | HIV-ASSOCIATED NEPHROPATHY | UROLOGY & NEPHROLOGY | AFRICAN-AMERICANS | RISK VARIANTS | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | STAGE RENAL-DISEASE | INNATE IMMUNITY
Journal Article
International Journal of Nephrology and Renovascular Disease, ISSN 1178-7058, 01/2014, Volume 7, pp. 57 - 67
Mitochondrial diseases can be related to mutations in either the nuclear or mitochondrial genome. Childhood presentations are commonly associated with renal... 
Genetics | Mitochondria | Kidney | Renal manifestations of general diseases | Mitochondrial diseases | Diagnosis | Cytochrome | Phosphorylation | Disease | Genes | Cell division | Genomes | Mitochondrial DNA | Metabolism | Proteins | Genotype & phenotype | Population | Mutation | Deoxyribonucleic acid--DNA | Transfer RNA
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 03/2018, Volume 29, Issue 3, pp. 869 - 879
Coding variants in the APOL1 gene are associated with kidney diseases in African ancestral populations; yet, the underlying biologic mechanisms remain... 
TRYPANOSOME LYTIC FACTOR | TRYPANOLYTIC FACTOR APOL1 | HIV-ASSOCIATED NEPHROPATHY | APOLIPOPROTEIN L1 | ALLOGRAFT SURVIVAL | UROLOGY & NEPHROLOGY | RENAL-DISEASE | AFRICAN-AMERICANS | CHRONIC KIDNEY-DISEASE | RISK VARIANTS | CELL-DEATH | kidney | autophagy | cell death | Basic Research | genetic renal disease
Journal Article
by Chaki, Moumita and Airik, Rannar and Ghosh, Amiya K and Giles, Rachel H and Chen, Rui and Slaats, Gisela G and Wang, Hui and Wang, Shaohui and Hurd, Toby W and Zhou, Weibin and Cluckey, Andrew and Gee, Heon Yung and Ramaswami, Gokul and Hong, Chen-Jei and Hamilton, Bruce A and Červenka, Igor and Ganji, Ranjani Sri and Bryja, Vitezslav and Arts, Heleen H and van Reeuwijk, Jeroen and Oud, Machteld M and Letteboer, Stef J.F and Roepman, Ronald and Husson, Hervé and Ibraghimov-Beskrovnaya, Oxana and Yasunaga, Takayuki and Walz, Gerd and Eley, Lorraine and Sayer, John A and Schermer, Bernhard and Liebau, Max C and Benzing, Thomas and Le Corre, Stephanie and Drummond, Iain and Janssen, Sabine and Allen, Susan J and Natarajan, Sivakumar and O’Toole, John F and Attanasio, Massimo and Saunier, Sophie and Antignac, Corinne and Koenekoop, Robert K and Ren, Huanan and Lopez, Irma and Nayir, Ahmet and Stoetzel, Corinne and Dollfus, Helene and Massoudi, Rustin and Gleeson, Joseph G and Andreoli, Sharon P and Doherty, Dan G and Lindstrad, Anna and Golzio, Christelle and Katsanis, Nicholas and Pape, Lars and Abboud, Emad B and Al-Rajhi, Ali A and Lewis, Richard A and Omran, Heymut and Lee, Eva Y.-H.P and Sekiguchi, JoAnn M and Saunders, Rudel and Johnson, Colin A and Garner, Elizabeth and Vanselow, Katja and Andersen, Jens S and Shlomai, Joseph and Nurnberg, Peter and Nurnberg, Gudrun and Levy, Shawn and Smogorzewska, Agata and Otto, Edgar A and Hildebrandt, Friedhelm
Cell, ISSN 0092-8674, 08/2012, Volume 150, Issue 3, pp. 533 - 548
Journal Article