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Nature Medicine, ISSN 1078-8956, 09/2015, Volume 21, Issue 9, pp. 1038 - 1047
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 05/2004, Volume 350, Issue 21, pp. 2129 - 2139
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2016, Volume 113, Issue 47, pp. 13456 - 13461
Oncogenic activation of protein kinase drives tumor growth by promoting mitogen-activated protein kinase (MAPK) pathway signaling. Because oncogenic mutations... 
BRAF | Targeted therapy | Lung | Cancer | DABRAFENIB | targeted therapy | MULTICENTER | MULTIDISCIPLINARY SCIENCES | SENSITIVITY | OPEN-LABEL | PROLIFERATION | MELANOMAS | DEPENDENCE | lung | RESISTANCE | cancer | MUTATIONS | PROGRESSION | Lung Neoplasms - drug therapy | Humans | Antineoplastic Agents - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Gene Knockdown Techniques | Heterocyclic Compounds, 2-Ring - adverse effects | Heterocyclic Compounds, 2-Ring - therapeutic use | Female | Antineoplastic Agents - pharmacology | Lung Neoplasms - genetics | Lung Neoplasms - enzymology | Treatment Outcome | Heterocyclic Compounds, 2-Ring - pharmacokinetics | Enzyme Activation - drug effects | Mice, SCID | Mutation - genetics | Sulfonamides - pharmacology | Mechanistic Target of Rapamycin Complex 1 - metabolism | Sulfonamides - pharmacokinetics | Heterocyclic Compounds, 2-Ring - pharmacology | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Animals | MAP Kinase Signaling System - drug effects | Sulfonamides - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Proto-Oncogene Proteins B-raf - genetics | Cell Line, Tumor | Sulfonamides - adverse effects | Protein Kinase Inhibitors - pharmacology | Drug Resistance, Neoplasm - drug effects | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2006, Volume 103, Issue 7, pp. 2316 - 2321
Journal Article
Nature Genetics, ISSN 1061-4036, 01/2017, Volume 49, Issue 1, pp. 87 - 96
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2005, Volume 102, Issue 21, pp. 7665 - 7670
Non-small cell lung cancers (NSCLCs) with activating mutations in the kinase domain of the epidermal growth factor receptor (EGFR) demonstrate dramatic, but... 
Biological Sciences | Receptors | Drug trafficking | Internalization | Liver | Cell lines | Lesions | Codons | Genetic mutation | Tumors | Resistance mechanisms | Molecular targeted therapy | Drug resistance | Tyrosine kinase inhibitor | Non-small cell lung cancer | non-small cell lung cancer | CELL LUNG-CANCER | APOPTOSIS | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | SENSITIVITY | molecular targeted therapy | drug resistance | tyrosine kinase inhibitor | MUTATIONS | IMATINIB RESISTANCE | Erlotinib Hydrochloride | Receptor, Epidermal Growth Factor - genetics | Aniline Compounds | Humans | Lung Neoplasms - metabolism | Molecular Sequence Data | Immunoblotting | Quinolines - pharmacology | Receptor, ErbB-2 - physiology | Quinazolines - metabolism | Base Sequence | Organic Chemicals - pharmacology | Phosphorylation - drug effects | Tumor Cells, Cultured | Receptor, Epidermal Growth Factor - physiology | Lung Neoplasms - genetics | Neoplasm Recurrence, Local - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Mutation - genetics | Sequence Analysis, DNA | Drug Resistance - genetics | Signal Transduction - drug effects | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Neoplasm Recurrence, Local - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Aminoquinolines | Lung cancer | Research
Journal Article
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 08/2019, Volume 129, Issue 8, pp. 3401 - 3406
Journal Article
Nature Genetics, ISSN 1061-4036, 12/2005, Volume 37, Issue 12, pp. 1315 - 1316
Journal Article
Lung Cancer: Targets and Therapy, ISSN 1179-2728, 04/2015, Volume 6, Issue default, pp. 27 - 34
Journal Article
Journal Article
Brain Pathology, ISSN 1015-6305, 01/2015, Volume 25, Issue 1, pp. 111 - 112
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 380, Issue 1, pp. 144 - 152
Highlights • A two-step CTC enrichment process to improve the purity of CTCs is reported. • Three different tumor types- prostate, lung and pancreas cancer... 
Hematology, Oncology and Palliative Medicine | Fluorescence-activated cell sorting | Circulating tumor cells | Copy number | High-purity | Sequencing | Cell adhesion matrix | MUTATION-SPECIFIC ANTIBODIES | ADENOCARCINOMA | PHENOTYPE | EGFR MUTATIONS | BIOMARKERS | GROWTH-FACTOR RECEPTOR | CIRCULATING TUMOR-CELLS | ONCOLOGY | RESISTANCE | INHIBITORS | Receptor, Epidermal Growth Factor - genetics | Pancreatic Neoplasms - metabolism | Humans | Lung Neoplasms - metabolism | Carcinoma, Pancreatic Ductal - metabolism | Lung Neoplasms - pathology | Male | Prostatic Neoplasms, Castration-Resistant - genetics | Prostatic Neoplasms, Castration-Resistant - pathology | Carcinoma, Pancreatic Ductal - genetics | Flow Cytometry | Neoplastic Stem Cells - metabolism | DNA Mutational Analysis | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Genomics - methods | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Genetic Predisposition to Disease | Carcinoma, Non-Small-Cell Lung - genetics | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Carcinoma, Non-Small-Cell Lung - metabolism | Pancreatic Neoplasms - genetics | Cell Separation - methods | Carcinoma, Pancreatic Ductal - pathology | Cell Adhesion | Prostatic Neoplasms, Castration-Resistant - metabolism | Collagen - metabolism | Phenotype | Cell Line, Tumor | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | Mutation | Cell Movement | Genetic aspects | Metastasis | Pancreatic cancer | Genomics | Cancer
Journal Article