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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2006, Volume 103, Issue 50, pp. 19093 - 19098
Regional alveolar hypoxia causes local vasoconstriction in the lung, shifting blood flow from hypoxic to normoxic areas, thereby maintaining gas exchange. This... 
Pulmonary alveoli | Lungs | Vasoconstriction | Fluorescence | Hypoxia | Cell membranes | Pulmonary hypertension | Pulmonary artery | Pulmonary gas exchange | Vasoconstrictors | Arterial hypoxemia | Precapillary pulmonary arterial smooth-muscle cells | Transient receptor potential channel 6-deficient mouse model | Hypoxia-induced diacylglycerol accumulation | pulmonary hypertension | MULTIDISCIPLINARY SCIENCES | hypoxia-incluced diacylglycerol accumulation | CAPACITATIVE CA2+ ENTRY | OPERATED CA2 | RECEPTOR POTENTIAL CHANNELS | INHIBITION | SMOOTH-MUSCLE-CELLS | ENDOTHELIN-1 | precapillary pulmonary arterial smooth-muscle cells | HYPERTENSION | ARTERIAL MYOCYTES | transient receptor potential channel 6-deficient mouse model arterial hypoxemia | NONSELECTIVE CATION CHANNELS | OXYGEN-SENSING MECHANISMS | Acute Disease | Gene Expression | Cations - chemistry | Lung Diseases - metabolism | Cations - metabolism | TRPC Cation Channels - deficiency | Aorta - metabolism | Hypoxia - metabolism | Mice, Knockout | Hypoxia - genetics | Lung Diseases - genetics | Animals | Diglycerides - metabolism | Membrane Potentials | TRPC Cation Channels - metabolism | Pulmonary Gas Exchange | Mice | TRPC Cation Channels - genetics | Smooth muscle | Research | Diacylglycerol | hypoxia-induced diacylglycerol accumulation | transient receptor potential channel 6-deficient mouse model | Biological Sciences | arterial hypoxemia
Journal Article
Immunity, ISSN 1074-7613, 04/2017, Volume 46, Issue 4, pp. 675 - 689
Activated T cells produce reactive oxygen species (ROS), which trigger the antioxidative glutathione (GSH) response necessary to buffer rising ROS and prevent... 
Gclc | glutathione | Myc | glycolysis | reactive oxygen species | ROS | mTOR | metabolism | T cells | GSH | metabolic reprogramming | NFAT | MAMMALIAN TARGET | CANCER-CELLS | GLUTAMINE REQUIREMENT | RECEPTOR STIMULATION | SIGNALING PATHWAY | LYMPHOCYTE ACTIVATION | C-MYC | COMPLEX 1 | INDUCED DEATH | IMMUNOLOGY | KINASE ACTIVATION | Reactive Oxygen Species - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Glutamate-Cysteine Ligase - deficiency | TOR Serine-Threonine Kinases - metabolism | Glutathione - metabolism | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Glutamine - metabolism | Immunoblotting | Signal Transduction - genetics | Proto-Oncogene Proteins c-myc - metabolism | Mice, Knockout | Inflammation - metabolism | Animals | T-Lymphocytes - metabolism | Glycolysis | Inflammation - genetics | Glutamate-Cysteine Ligase - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Energy Metabolism - genetics | Antiviral agents | Glucose metabolism | Cysteine | Thiols | Ligases | Physiological aspects | Glutamate | Cells | Inflammation | TOR protein | Transcription factors | Phosphorylation | Homeostasis | Lymphocytes T | Myc protein | NF-AT protein | Antiinfectives and antibacterials | Cell activation | Energy utilization | Cell growth | Gene targeting | Lymphocytes | Cell cycle | Physiology | Glutathione | Antigens | Integration | Rapamycin | T cell receptors | Metabolism | Ablation | Apoptosis
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 08/2005, Volume 25, Issue 16, pp. 6980 - 6989
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 12/2007, Volume 455, Issue 3, pp. 465 - 477
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2016, Volume 11, Issue 6, p. e0156929
In addition to their well-known antibacterial activity some antimicrobial peptides and proteins (AMPs) display also antiviral effects. A 27 aa peptide from the... 
GRAM-NEGATIVE BACTERIA | REPLICATION | LL-37 | SUBTYPES | MULTIDISCIPLINARY SCIENCES | MECHANISMS | HUMAN ALPHA-DEFENSINS | PEPTIDE | Virus Replication - drug effects | Antiviral Agents - pharmacology | Cricetinae | Influenza A Virus, H3N2 Subtype - drug effects | Humans | Influenza A Virus, H3N2 Subtype - pathogenicity | Antimicrobial Cationic Peptides - pharmacology | Cells, Cultured | Influenza A Virus, H1N1 Subtype - pathogenicity | Peptide Fragments - pharmacology | Influenza A Virus, H5N1 Subtype - drug effects | Antiviral Agents - metabolism | Antimicrobial Cationic Peptides - metabolism | Blood Proteins - metabolism | Influenza A Virus, H1N1 Subtype - drug effects | Blood Proteins - pharmacology | Animals | Dogs | Madin Darby Canine Kidney Cells | Influenza A virus - pathogenicity | Influenza A Virus, H5N1 Subtype - pathogenicity | Influenza A virus - drug effects | Neutrophils - metabolism | Avian influenza | Physiological aspects | Care and treatment | Research | Antimicrobial peptides | Peptides | Viruses | Homology | Genomes | Proteins | Immunology | Measles | Infectivity | Human immunodeficiency virus--HIV | Antiviral activity | Antibacterial activity | Bacteria | Gram-negative bacteria | Immune system | Pathogens | Neutrophils | Antimicrobial agents | Permeability | Electron microscopy | Pathogenicity | Stomatitis | Influenza | BPI protein | Cytoplasm | HIV | Human immunodeficiency virus
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2019, Volume 49, Issue 5, pp. 812 - 815
We found that deletion of the final 30 amino acids of transcription factor IRF4's (interferon‐regulatory factor) C‐terminus creates hyperactive IRF4. When... 
Mutation | IRF4 | Th17 | Transcriptional activity | T cell differentiation | CROSSROADS | IMMUNOLOGY | Interferon | Biological response modifiers | T cells | Cell differentiation | Clonal deletion | CD8 antigen | Interferon regulatory factor 4 | Lymphocytes | Deletion | Amino acids | Lymphocytes T | Differentiation | CD4 antigen
Journal Article
Archives of Virology, ISSN 0304-8608, 10/2013, Volume 158, Issue 10, pp. 2049 - 2058
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e68812
Journal Article