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The Lancet, ISSN 0140-6736, 2012, Volume 380, Issue 9842, pp. 680 - 688
Idiopathic pulmonary fi brosis is a serious and progressive chronic lung disease that is characterised by altered cellular composition and homoeostasis in the... 
Journal Article
AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, ISSN 1073-449X, 06/2019, Volume 199, Issue 12, pp. 1454 - 1456
The report by Reyfman and colleagues (pp. 1517–1536) in this issue of the Journal provides important insight into pathogenic cell types in lung fibrosis on an... 
RESPIRATORY SYSTEM | CRITICAL CARE MEDICINE | Datasets | Proteins | Algorithms | Pulmonary fibrosis | Lungs | Lung diseases | Fibroblasts | Ligands | Surfactants | Gene expression | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2019, Volume 199, Issue 4, pp. 408 - 410
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 04/2018, Volume 197, Issue 8, pp. 981 - 983
In human idiopathic pulmonary fibrosis or in influenza H1N1-challenged murine lungs (e.g., Krt5+), subpopulations proliferate and coexpress various bronchial... 
OBSTRUCTIVE PULMONARY-DISEASE | RESPIRATORY SYSTEM | AIRWAY STEM-CELLS | REGENERATION | SQUAMOUS METAPLASIA | BASAL-CELLS | CRITICAL CARE MEDICINE | Airway management | Pulmonary fibrosis | Transplants & implants | Disease | Lungs | Swine flu | Pathogenesis | Stem cells | Homeostasis | Chronic obstructive pulmonary disease | Cancer
Journal Article
American journal of respiratory cell and molecular biology, 08/2019, Volume 61, Issue 2, p. 141
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2016, Volume 139, Issue 5, pp. 1525 - 1535
Abstract Background Chronic immune diseases (CIDs), such as asthma, are highly prevalent. Currently available pharmaceuticals improve symptoms, but cannot cure... 
Allergy and Immunology | allergic airway disease | D-tryptophan | screening | bacterial substance | immune modulation | probiotic bacteria | gut microbiota | D-AMINO ACIDS | DENDRITIC CELLS | MOLECULE N-(3-OXODODECANOYL)-L-HOMOSERINE LACTONE | RANDOMIZED CONTROLLED-TRIAL | IMMUNOLOGY | ATOPIC-DERMATITIS | ALLERGY | IN-VIVO | DOUBLE-BLIND | PLACEBO-CONTROLLED TRIAL | 1ST 6 MONTHS | PRIMARY PREVENTION | Bacteria - metabolism | Dendritic Cells | Humans | Cells, Cultured | Lipopolysaccharides | Probiotics | Tryptophan - biosynthesis | Animals | Asthma - immunology | Cell Line, Tumor | Female | Mice, Inbred BALB C | Gastrointestinal Microbiome - immunology | Cytokines - immunology | Prevention | Tryptophan | Bacteria | Biogeochemistry | Microbiota (Symbiotic organisms) | Asthma | Dendritic cells | Drugs | Nuclear magnetic resonance--NMR | Crosstalk | Lung | Clinical trials | Amino acids | Lymphocytes T | Respiratory tract | Microorganisms | Metabolites | Lymphocytes | L-Tryptophan | Polarity | Biocompatibility | Respiratory tract diseases | CCL17 protein | Magnetic induction | Medical research | Fractionation | Immunoregulation | Secretion | Bioassays | Hypersensitivity | Mass spectroscopy | Medical screening | Lymphoma | Allergies | Biological activity | D-Amino acids | Feeding | Costimulator | Disease prevention | Immune systems | Respiratory diseases | Mass spectrometry | Hodgkin's disease | Pharmaceuticals | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2009, Volume 119, Issue 4, pp. 772 - 787
Idiopathic pulmonary fibrosis (IPF) is characterized by distorted lung architecture and loss of respiratory function. Enhanced (myo)fibroblast activation, ECM... 
EPITHELIAL-MESENCHYMAL TRANSITION | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG FIBROSIS | CELLS | APOPTOSIS | TISSUE GROWTH-FACTOR | CCN FAMILY | MECHANISMS | EXPRESSION | INTERSTITIAL PNEUMONIAS | Oncogene Proteins - genetics | Up-Regulation | Idiopathic Pulmonary Fibrosis - genetics | Epithelial Cells - metabolism | Humans | Middle Aged | Male | Pulmonary Fibrosis - genetics | Wnt Proteins - metabolism | Oncogene Proteins - physiology | CCN Intercellular Signaling Proteins | Idiopathic Pulmonary Fibrosis - etiology | Pulmonary Alveoli - metabolism | Adult | Female | Intracellular Signaling Peptides and Proteins - genetics | Pulmonary Fibrosis - etiology | Bleomycin - toxicity | Disease Models, Animal | Pulmonary Alveoli - pathology | Mice, Inbred C57BL | Oncogene Proteins - pharmacology | Epithelial Cells - pathology | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Recombinant Proteins - pharmacology | beta Catenin - metabolism | Animals | Models, Biological | Proto-Oncogene Proteins - physiology | Pulmonary Fibrosis - physiopathology | Cell Proliferation - drug effects | Idiopathic Pulmonary Fibrosis - physiopathology | Mice | Intracellular Signaling Peptides and Proteins - physiology | Cellular proteins | Pulmonary fibrosis | Genes | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e63226 - e63226
Non-Small-Cell-Lung-Cancer (NSCLC) represents approximately 85% of all lung cancers and remains poorly understood. While signaling pathways operative during... 
BREAST-CANCER | GROWTH-FACTOR-BETA | CARCINOGENESIS | ANGIOGENESIS | SIGNALING PATHWAY | IDIOPATHIC-PULMONARY-FIBROSIS | MULTIDISCIPLINARY SCIENCES | MITOCHONDRIAL METABOLISM | IN-VIVO | EXPRESSION | TUMOR MICROENVIRONMENT | Humans | Lung Neoplasms - metabolism | Hedgehog Proteins - metabolism | Lung Neoplasms - pathology | Veratrum Alkaloids - pharmacology | Fibroblasts - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Cell Survival - drug effects | Neoplasm Invasiveness | Carcinoma, Non-Small-Cell Lung - metabolism | Hedgehog Proteins - antagonists & inhibitors | Fibroblasts - pathology | Transcription Factors - metabolism | Cell Movement - drug effects | Collagen - metabolism | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Cell Line, Tumor | Ligands | Cell Proliferation - drug effects | Mice | Zinc Finger Protein GLI1 | Mesoderm - pathology | Cyclin D - metabolism | Development and progression | Squamous cell carcinoma | DNA binding proteins | Lung cancer, Non-small cell | Stem cells | Cell proliferation | Adenocarcinoma | Thoracic surgery | Transcription factors | Mesenchyme | Leukemia | Lung cancer | Crosstalk | Lung | Activation | Metastasis | Cyclin D1 | Cancer therapies | Metastases | Cyclin D2 | Morphogenesis | Proteins | Angiogenesis | Cell activation | Cell growth | Fibroblasts | Lung diseases | Breast cancer | Lung carcinoma | Gene expression | Studies | Signaling | Pulmonary fibrosis | Hedgehog protein | Stromal cells | Medical prognosis | Collagen | Cancer | Index Medicus
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 07/2010, Volume 182, Issue 2, pp. 220 - 229
Journal Article