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Molecular Cell, ISSN 1097-2765, 03/2012, Volume 45, Issue 6, pp. 764 - 776
Aberrant ErbB2 receptor tyrosine kinase activation in breast cancer is strongly linked to an invasive disease. The molecular basis of ErbB2-driven invasion is... 
PATHWAYS | ONCOGENE | METASTASIS | EPITHELIAL-CELLS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | CYSTEINE CATHEPSINS | FIBROBLASTS | MULTISTAGE TUMORIGENESIS | ERBB RECEPTORS | CELL BIOLOGY | Cathepsin L - genetics | Protein Kinase C-alpha - metabolism | Proto-Oncogene Protein c-ets-1 - metabolism | Receptor, ErbB-2 - genetics | Protein-Tyrosine Kinases - metabolism | Cathepsin B - genetics | Humans | Protein Kinase C-alpha - genetics | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Cathepsin L - metabolism | Breast Neoplasms - metabolism | Protein-Tyrosine Kinases - genetics | Kruppel-Like Transcription Factors - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Female | Promoter Regions, Genetic | p21-Activated Kinases - genetics | Myotonin-Protein Kinase | Response Elements | Signal Transduction | Neoplasm Invasiveness | Cathepsin B - metabolism | p21-Activated Kinases - metabolism | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Proto-Oncogene Protein c-ets-1 - genetics | Kruppel-Like Transcription Factors - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Tyrosine | Cysteine | Oncology, Experimental | Genes | Cathepsins | Breast cancer | DNA binding proteins | Genetic transcription | Research | Cystine | Analysis | Universities and colleges | Protein kinases | Phosphotransferases | Protein binding | Cancer | Protein kinase C | Transcription factors | Introns | Invasiveness | cathepsins | AKT protein | ErbB-2 protein | Zinc | Enhancers | Cathepsin B | Protein-tyrosine kinase receptors | Protein-serine/threonine kinase
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