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PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e75483 - e75483
Ras association domain family protein 1A (RASSF1A) is a tumor suppressor gene silenced in cancer. Here we report that RASSF1A is a novel regulator of... 
YES-ASSOCIATED PROTEIN | APOPTOSIS | C-ABL | DNA METHYLTRANSFERASE-1 | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | YAP | STEM-CELL PROLIFERATION | NF-KAPPA-B | PROMOTES | P53 | Inflammation - pathology | Apoptosis - drug effects | Colitis, Ulcerative - genetics | Colon - drug effects | NF-kappa B - metabolism | Intestinal Mucosa - drug effects | Tumor Suppressor Proteins - deficiency | Colitis, Ulcerative - drug therapy | Toll-Like Receptors - metabolism | Tumor Suppressor Proteins - metabolism | Signal Transduction | Colon - pathology | bcl-2-Associated X Protein - metabolism | Epithelial Cells - pathology | Colitis, Ulcerative - pathology | Pyrimidines - pharmacology | Piperazines - pharmacology | Colon - metabolism | Mice, Knockout | Tumor Protein p73 | Toll-Like Receptors - genetics | Mice | Intestinal Mucosa - pathology | Inflammation - chemically induced | Dextran Sulfate | Intestinal Mucosa - metabolism | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Phosphoproteins - metabolism | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Inflammation - drug therapy | Tumor Suppressor Proteins - genetics | Benzamides - pharmacology | Nuclear Proteins - genetics | bcl-2-Associated X Protein - genetics | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Phosphoproteins - genetics | DNA-Binding Proteins - genetics | Imatinib Mesylate | Proto-Oncogene Proteins c-abl - pharmacology | Animals | NF-kappa B - genetics | Colitis, Ulcerative - chemically induced | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Cell Proliferation - drug effects | Adaptor Proteins, Signal Transducing - metabolism | Tyrosine | Genes | Inflammation | Permeability | Dextran | Cell death | Gastrointestinal diseases | Genetic aspects | Genetic engineering | Colitis | Tumor proteins | Tumors | Cancer | Pediatrics | p53 Protein | Biochemistry | Proteins | Physiology | Tumorigenesis | Inhibition | Damage accumulation | Protein-tyrosine kinase | Deoxyribonucleic acid--DNA | Imatinib | Mortality | Gene expression | Survival | Yes-associated protein | Inflammatory bowel disease | Cell injury | Sodium | Pharmacy | TNF inhibitors | Oxidative stress | Phosphorylation | Inflammatory bowel diseases | Bax protein | Epithelial cells | Gene regulation | DNA damage | Homeostasis | Kinases | Accumulation | Intestine | Rodents | Cell cycle | DNA methylation | Toll-like receptors | Injuries | Pharmaceutical sciences | Cell survival | Dentistry | Immune systems | Medicine | Lymphocytes B | Epigenetics | Tumor suppressor genes | Kidney diseases | Diabetes | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 2007, Volume 363, Issue 3, pp. 506 - 512
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