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British Journal of Dermatology, ISSN 0007-0963, 12/2016, Volume 175, Issue 6, pp. 1142 - 1144
Linked Article: Pinto Gouveia et al. Br J Dermatol 2016; 175:1274–1283. 
TOXIC EPIDERMAL NECROLYSIS | SYSTEMIC SYMPTOMS | T-CELLS | HYPERSENSITIVITY | DERMATOLOGY | Drug Hypersensitivity Syndrome | Eosinophilia | Exanthema | Humans | Drug Eruptions
Journal Article
JEADV : Journal of the European Academy of Dermatology and Venereology, ISSN 0926-9959, 2009, Volume 23, Issue 2, pp. 1 - 70
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 01/2010, Volume 29, Issue 2, pp. 227 - 236
Many studies have highlighted the critical role of c-Kit in normal melanocyte development but its role in melanoma development remains unclear. Although c-Kit... 
MAPK pathway | C-Kit | Imatinib | PI3K/Akt pathway | Melanoma | FACTOR-RECEPTOR | SURVIVAL | ACTIVATION | c-Kit | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | imatinib | melanoma | CELL BIOLOGY | ONCOGENE | INHIBITION | ONCOLOGY | MUTATION | GENETICS & HEREDITY | MELANOMA-CELLS | EXPRESSION
Journal Article
Oncogene, ISSN 0950-9232, 12/2011, Volume 30, Issue 49, pp. 4887 - 4900
Aberrant expression of Secreted Protein Acidic and Rich in Cysteine (SPARC)/osteonectin has been associated with an invasive tumor cell phenotype and poor... 
melanoma | tumor biology | signaling | p53 | CYSTEINE SPARC | APOPTOSIS | INTEGRIN-LINKED KINASE | CANCER CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MALIGNANT-MELANOMA | E-CADHERIN | TUMOR-SUPPRESSOR P53 | CELL BIOLOGY | ONCOLOGY | THERAPEUTIC TARGET | GENETICS & HEREDITY | UP-REGULATION | EXPRESSION | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Gene Knockdown Techniques | Osteonectin - deficiency | Osteonectin - genetics | RNA Interference | Time Factors | Melanoma - genetics | Protein Binding - drug effects | Antineoplastic Agents - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Melanoma - metabolism | Cell Survival - drug effects | Tumor Suppressor Protein p53 - metabolism | bcl-2-Associated X Protein - metabolism | Osteonectin - metabolism | Melanoma - pathology | Up-Regulation - drug effects | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Mice | Oxidative Stress - drug effects | Dactinomycin - pharmacology | Care and treatment | Cysteine | Melanoma | Development and progression | Tumor suppressor genes | Cellular signal transduction | Research | Dactinomycin | Apoptosis | Proteins | Signal transduction | Phosphorylation | Gene expression | Skin cancer | Drugs | MDM2 protein | Cell survival | Bax protein | Tumor cells | RNA-mediated interference | AKT protein | Cytotoxicity | Caspase-3 | p53 protein | Mitochondria | Osteonectin | Tumorigenesis
Journal Article
British Journal of Dermatology, ISSN 0007-0963, 02/2007, Volume 156, Issue 2, pp. 258 - 262
Summary For nearly 200 years it has been appreciated that plaque psoriasis consists of a number of distinct clinical phenotypes. However, a reliable and simple... 
clinical study | phenotype | International Psoriasis Council | genetics | psoriasis | Genetics | Phenotype | Psoriasis | Clinical study | PALMOPLANTAR PUSTULOSIS | LATE-ONSET | PSORS1 | DERMATOLOGY | Psoriasis - classification | Psoriasis - pathology | Humans | Analysis | Genetic aspects
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 514, Issue 7521, pp. 247 - 251
The polycomb repressive complex 2 (PRC2) exerts oncogenic effects in many tumour types(1). However, loss-of-function mutations in PRC2 components occur in a... 
SUPPRESSOR | GENE | MULTIDISCIPLINARY SCIENCES | MICE | NF1 MICRODELETION PATIENTS | MUTATIONS | BET BROMODOMAIN INHIBITION | CANCER | TUMORS | NEUROFIBROMATOSIS TYPE-1 | EZH2 | Chromatin - metabolism | Transcription, Genetic - drug effects | Polycomb Repressive Complex 2 - genetics | Humans | Transcription Factors - deficiency | ras Proteins - metabolism | Neurofibromin 1 - deficiency | Glioma - genetics | Neoplasms - genetics | Melanoma - genetics | Tumor Suppressor Proteins - deficiency | Glioma - pathology | Nerve Sheath Neoplasms - pathology | Nuclear Proteins - deficiency | Tumor Suppressor Proteins - genetics | Epigenesis, Genetic - drug effects | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Nuclear Proteins - genetics | Chromatin - drug effects | Nerve Sheath Neoplasms - drug therapy | Disease Models, Animal | Triazoles - therapeutic use | Nerve Sheath Neoplasms - genetics | Tumor Suppressor Proteins - metabolism | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | ras Proteins - antagonists & inhibitors | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Melanoma - pathology | Transcription Factors - genetics | Azepines - therapeutic use | Neoplasms - drug therapy | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Animals | Polycomb Repressive Complex 2 - deficiency | Melanoma - drug therapy | Nuclear Proteins - antagonists & inhibitors | Mice | Neoplasms - pathology | Polycomb Repressive Complex 2 - metabolism | Chromatin - genetics | Neurofibromin 1 - genetics | Glioma - drug therapy | Epigenetic inheritance | Ras genes | Genetic transcription | Research | Gliomas | Studies | Mutation | Genes | Tumors | Defects
Journal Article