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PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, p. e0108247
Journal Article
Journal Article
Blood, ISSN 0006-4971, 12/2014, Volume 124, Issue 21, pp. 4540 - 4540
Abstract Objectives and background: Clinical studies are currently initiated where treatment of CML patients with tyrosine kinase inhibitors is stopped. One of... 
Journal Article
Journal Article
Annals of Hematology, ISSN 0939-5555, 12/2018, Volume 97, Issue 12, pp. 2299 - 2308
Philadelphia-negative myeloproliferative neoplasms (MPNs) are a diverse group of diseases whose common feature is the presence of V617F mutation of the JAK2... 
ddPCR | Limit of detection | JAK-inhibitor | Medicine & Public Health | Hematology | Myeloproliferative neoplasms | Minimal residual disease | Oncology | qPCR | JAK2(V617F) | POLYCYTHEMIA-VERA | DISORDERS | PERIPHERAL-BLOOD | ESSENTIAL THROMBOCYTHEMIA | PRIMARY MYELOFIBROSIS | JAK2V617F | MUTATION | STEM-CELL TRANSPLANTATION | HEMATOLOGY | Original
Journal Article
BMC Cancer, ISSN 1471-2407, 03/2011, Volume 11, Issue 1, pp. 107 - 107
Background: As supplement to KRAS mutational analysis, BRAF and PIK3CA mutations as well as expression of PTEN may account for additional non-responders to... 
Cetuximab and Irinotecan | metastatic colorectal cancerKRASBRAFPIK3CA/PTEN mutations | KRAS MUTATIONS | COLON-CANCER | PANITUMUMAB | SURVIVAL | GROWTH-FACTOR RECEPTOR | PREDICTS | THERAPY | ONCOLOGY | INHIBITOR CETUXIMAB | RESISTANCE | EXPRESSION | Receptor, Epidermal Growth Factor - genetics | Prognosis | Colorectal Neoplasms - genetics | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Salvage Therapy | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Colorectal Neoplasms - diagnosis | Antibodies, Monoclonal, Humanized | Neoplasm Metastasis | DNA Mutational Analysis | Colorectal Neoplasms - drug therapy | Aged, 80 and over | Adult | Camptothecin - administration & dosage | Female | Carcinoma - pathology | Cetuximab | Receptor, Epidermal Growth Factor - physiology | Camptothecin - analogs & derivatives | Genetic Linkage | Carcinoma - drug therapy | Camptothecin - adverse effects | Biomarkers, Pharmacological - analysis | Biomarkers, Tumor - analysis | Carcinoma - diagnosis | Treatment Outcome | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Carcinoma - genetics | Aged | Biomarkers, Tumor - genetics | Colorectal Neoplasms - pathology | Immunohistochemistry | Care and treatment | Usage | Epidermal growth factor | Patient outcomes | Colorectal cancer | Research | Health aspects | Risk factors | metastatic colorectal cancer | KRAS | BRAF | PIK3CA/PTEN mutations
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, p. e0183620
The chronic Philadelphia-negative myeloproliferative neoplasms (MPNs) are acquired stem cell neoplasms which ultimately may transform to acute myelogenous... 
C-REACTIVE PROTEIN | POLYCYTHEMIA-VERA | CIRCULATING YKL-40 | MULTIDISCIPLINARY SCIENCES | TYROSINE KINASE JAK2 | CLONAL EVOLUTION | ACUTE MYELOID-LEUKEMIA | ESSENTIAL THROMBOCYTHEMIA | NF-KAPPA-B | SOMATIC MUTATIONS | CHRONIC MYELOPROLIFERATIVE NEOPLASMS | Models, Theoretical | Inflammation - pathology | Humans | Carcinogenesis - genetics | Janus Kinase 2 - genetics | Polycythemia Vera - genetics | Thrombocythemia, Essential - pathology | Myeloproliferative Disorders - pathology | Clonal Evolution - genetics | Thrombocythemia, Essential - genetics | Myeloproliferative Disorders - genetics | Disease Progression | Neoplasms - genetics | Inflammation - genetics | Neoplastic Stem Cells - pathology | Polycythemia Vera - pathology | Neoplasms - pathology | Stem cells | Physiological aspects | Development and progression | Genetic aspects | Inflammation | Research | Properties | Myeloproliferative disorders | Risk factors | Cell proliferation | Intervention | Haplotypes | Pathogenesis | Leukemia | Metastasis | Epidemiology | Inflammatory diseases | Neoplasms | Immunology | Developmental stages | Mathematical analysis | Feedback | Evolution | Mathematical models | Myelofibrosis | Hematology | Myeloid leukemia | Cloning | Crohns disease | Studies | Hospitals | Cell death | Interferon | Mutation | Acute myeloid leukemia | Cancer | Apoptosis | Tumors
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0165336
Journal Article
Translational Oncology, ISSN 1936-5233, 10/2018, Volume 11, Issue 5, pp. 1220 - 1224
Measuring total cell-free DNA (cfDNA) or cancer-specific mutations herein has presented as new tools in aiding the treatment of cancer patients. Studies show... 
CELL LUNG-CANCER | MUTANT-DNA | PLASMA | THERAPY | EVOLUTION | ONCOLOGY | QUANTIFICATION | RESISTANCE | SAMPLES | METASTATIC COLORECTAL-CANCER | EGFR
Journal Article
BLOOD, ISSN 0006-4971, 08/2019, Volume 134, Issue 5, pp. 469 - 479
The JAK2 V617F and calreticulin mutations (CALR) are frequent within myeloproliferative neoplasms (MPNs). JAK2 V617F has been detected in the general... 
C-REACTIVE PROTEIN | CALR MUTATIONS | POLYCYTHEMIA-VERA | RISK-FACTORS | VENOUS THROMBOEMBOLISM | CLONAL HEMATOPOIESIS | MYELOPROLIFERATIVE NEOPLASMS | ESSENTIAL THROMBOCYTHEMIA | CHRONIC INFLAMMATION | ALCOHOL-DRINKING | HEMATOLOGY
Journal Article