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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2010, Volume 107, Issue 4, pp. 1431 - 1436
Control of organ size by cell proliferation and survival is a fundamental developmental process, and its deregulation leads to cancer. However, the molecular... 
Cell growth | Phosphorylation | Hepatocytes | Cell death | Liver | Drosophila | Stem cells | Cell lines | Tumors | Apoptosis | Tumor suppressor | Hippo signaling | Yap phosphorylation | CELL CONTACT INHIBITION | PROGENITOR CELLS | GROWTH-CONTROL | HIPPO SIGNALING PATHWAY | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | tumor suppressor | TEAD/TEF FAMILY | FEEDBACK LOOP | LIVER STEM-CELLS | TRANSCRIPTION FACTOR | PROMOTES APOPTOSIS | Proto-Oncogene Proteins - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Cell Proliferation | Liver - enzymology | Liver Neoplasms - genetics | Cell Survival | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Organ Size | Proto-Oncogene Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Liver - growth & development | Hepatocyte Growth Factor - metabolism | Animals | Cell Transformation, Neoplastic - genetics | Gene Expression Regulation, Developmental | Hepatocyte Growth Factor - genetics | Liver Neoplasms - pathology | Liver - cytology | Mice | Mutation | Liver Neoplasms - enzymology | Protein-Serine-Threonine Kinases - metabolism | Cell proliferation | Growth | Physiological aspects | Cellular signal transduction | Genetic aspects | Research | Signal transduction | Insects | Cytokines | Rodents | Kinases | Index Medicus | Biological Sciences
Journal Article
Gut, ISSN 0017-5749, 09/2018, Volume 67, Issue 9, pp. 1692 - 1703
ObjectiveHippo signalling is a recently identified major oncosuppressive pathway that plays critical roles in inhibiting hepatocyte proliferation, survival and... 
Inflammation | Macrophages | Liver | Cancer | Hepatocellular Carcinoma | ORGAN SIZE CONTROL | PATHWAY | INFLAMMATION | LIVER | GROWTH | TISSUE | MONOCYTE RECRUITMENT | TUMOR-ASSOCIATED MACROPHAGES | GASTROENTEROLOGY & HEPATOLOGY | CANCER | T-CELLS | Cell Proliferation - genetics | Protein-Serine-Threonine Kinases - genetics | Proto-Oncogene Proteins - genetics | Signal Transduction - genetics | Carcinoma, Hepatocellular - enzymology | Hepatocytes - metabolism | Liver Neoplasms, Experimental - therapy | Phosphoproteins - genetics | Transcription Factors - genetics | Animals | Cell Transformation, Neoplastic | Mice, Mutant Strains | Carcinoma, Hepatocellular - genetics | Hepatocyte Growth Factor - genetics | Liver Neoplasms, Experimental - enzymology | Carcinoma, Hepatocellular - therapy | Liver Neoplasms, Experimental - genetics | Mice | Protein-Serine-Threonine Kinases - metabolism | Immunohistochemistry | Flow cytometry | Phenotypes | Liver diseases | Statistical analysis | Transcription | Alcohol | Hepatocellular carcinoma | Metastasis | Mammals | Gene expression | Kinases | Yes-associated protein | Liver cancer | Signal transduction | Monocytes | Clonal deletion | Hepatocytes | Monocyte chemoattractant protein | Rodents | Genetic analysis | Monocyte chemoattractant protein 1 | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Hepatology, ISSN 0270-9139, 07/2014, Volume 60, Issue 1, pp. 146 - 157
Journal Article
Hepatology (Baltimore, Md.), ISSN 0270-9139, 11/2013, Volume 58, Issue 5, pp. 1814 - 1823
We have previously demonstrated that chronic plus binge ethanol feeding acts synergistically to induce liver injury in mice; however, the mechanisms underlying... 
alcoholic liver injury | inflammation | Adhesion molecules
Journal Article
Hepatology, ISSN 0270-9139, 11/2013, Volume 58, Issue 5, pp. 1814 - 1823
Journal Article